J L Cmarik

Summary

Affiliation: National Cancer Institute
Country: USA

Publications

  1. pmc Differentially expressed protein Pdcd4 inhibits tumor promoter-induced neoplastic transformation
    J L Cmarik
    Basic Research Laboratory, Frederick Cancer Research and Development Center, National Cancer Institute, Frederick, MD 21702, USA
    Proc Natl Acad Sci U S A 96:14037-42. 1999
  2. ncbi request reprint A novel transformation suppressor, Pdcd4, inhibits AP-1 transactivation but not NF-kappaB or ODC transactivation
    H S Yang
    Gene Regulation Section, Basic Research Laboratory, National Cancer Institute, Frederick, MD 21702, USA
    Oncogene 20:669-76. 2001
  3. ncbi request reprint cDNA cloning and mapping of mouse pleckstrin (Plek), a gene upregulated in transformation-resistant cells
    J L Cmarik
    Basic Research Laboratory, National Cancer Institute, Frederick, Maryland, 21702, USA
    Genomics 66:204-12. 2000

Collaborators

  • M Dean
  • H S Yang
  • A K Verma
  • A P Jansen
  • N H Colburn
  • R Nair
  • K Shibahara

Detail Information

Publications3

  1. pmc Differentially expressed protein Pdcd4 inhibits tumor promoter-induced neoplastic transformation
    J L Cmarik
    Basic Research Laboratory, Frederick Cancer Research and Development Center, National Cancer Institute, Frederick, MD 21702, USA
    Proc Natl Acad Sci U S A 96:14037-42. 1999
    ..The antisense-transfected cells were reverted to their initial P- phenotype by overexpression of a Pdcd4 sense fragment. These observations demonstrate that the Pdcd4 protein inhibits neoplastic transformation...
  2. ncbi request reprint A novel transformation suppressor, Pdcd4, inhibits AP-1 transactivation but not NF-kappaB or ODC transactivation
    H S Yang
    Gene Regulation Section, Basic Research Laboratory, National Cancer Institute, Frederick, MD 21702, USA
    Oncogene 20:669-76. 2001
    ..Pdcd4 may thus constitute a useful molecular target for cancer prevention...
  3. ncbi request reprint cDNA cloning and mapping of mouse pleckstrin (Plek), a gene upregulated in transformation-resistant cells
    J L Cmarik
    Basic Research Laboratory, National Cancer Institute, Frederick, Maryland, 21702, USA
    Genomics 66:204-12. 2000
    ..We report the complete mouse cDNA sequence of pleckstrin and the localization of the gene to chromosome 11, its expression in a nonhematopoetic cell line, and its potential role in blocking neoplastic transformation...