Clara M Cheng

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi request reprint Tau is hyperphosphorylated in the insulin-like growth factor-I null brain
    Clara M Cheng
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA
    Endocrinology 146:5086-91. 2005
  2. ncbi request reprint Caloric restriction augments brain glutamic acid decarboxylase-65 and -67 expression
    Clara M Cheng
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892
    J Neurosci Res 77:270-6. 2004
  3. ncbi request reprint Perturbation of the transforming growth factor β system in Turner syndrome
    Jian Zhou
    National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD 20892, USA
    Beijing Da Xue Xue Bao 44:720-4. 2012
  4. ncbi request reprint Insulin-like growth factor 1 is essential for normal dendritic growth
    Clara M Cheng
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland
    J Neurosci Res 73:1-9. 2003
  5. ncbi request reprint Genomic imprinting and Turner syndrome
    Carolyn A Bondy
    Section on Epigenetics and Development, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD, USA
    Pediatr Endocrinol Rev 9:728-32. 2012
  6. ncbi request reprint A ketogenic diet increases brain insulin-like growth factor receptor and glucose transporter gene expression
    Clara M Cheng
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA
    Endocrinology 144:2676-82. 2003
  7. ncbi request reprint Signaling by insulin-like growth factor 1 in brain
    Carolyn A Bondy
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development NIH, Bldg 10 10N262, 10 Center Drive, Bethesda, MD 20892, USA
    Eur J Pharmacol 490:25-31. 2004
  8. ncbi request reprint Insulin-like growth factor-1 promotes neuronal glucose utilization during brain development and repair processes
    Carolyn A Bondy
    Developmental Endocrinology Branch, NICHD, NIH Bethesda, Maryland 20892, USA
    Int Rev Neurobiol 51:189-217. 2002
  9. pmc Autoimmune disorders in women with turner syndrome and women with karyotypically normal primary ovarian insufficiency
    Vladimir K Bakalov
    Section on Epigenetics and Development, Program on Developmental Endocrinology and Genetics, National Institute of Child Health and Human Development, 10 Center Dr CRC 1 3330 Bethesda, MD 20892 1103, USA
    J Autoimmun 38:315-21. 2012

Detail Information

Publications9

  1. ncbi request reprint Tau is hyperphosphorylated in the insulin-like growth factor-I null brain
    Clara M Cheng
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA
    Endocrinology 146:5086-91. 2005
    ..These observations indicate that IGF-I has a major role in regulating tau phosphorylation in the aging brain, whereas its role in promoting or preventing oxidative stress remains uncertain...
  2. ncbi request reprint Caloric restriction augments brain glutamic acid decarboxylase-65 and -67 expression
    Clara M Cheng
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892
    J Neurosci Res 77:270-6. 2004
    ....
  3. ncbi request reprint Perturbation of the transforming growth factor β system in Turner syndrome
    Jian Zhou
    National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD 20892, USA
    Beijing Da Xue Xue Bao 44:720-4. 2012
    ..To measure components of the circulating transforming growth factor β (TGFβ) system in patients with Turner syndrome (TS) compared to relevant controls and to ascertain correlation with endocrine and cardiovascular parameters...
  4. ncbi request reprint Insulin-like growth factor 1 is essential for normal dendritic growth
    Clara M Cheng
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland
    J Neurosci Res 73:1-9. 2003
    ..01) in Igf1 null mice. Evidence is presented that Igf1 deletion causes disruptions in lipid and microtubule metabolism, leading to impaired neuronal somatic and dendritic growth. Published 2003 Wiley-Liss, Inc...
  5. ncbi request reprint Genomic imprinting and Turner syndrome
    Carolyn A Bondy
    Section on Epigenetics and Development, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD, USA
    Pediatr Endocrinol Rev 9:728-32. 2012
    ..This may contribute to a high rate of fetal loss in human monosomy via impaired placentation in the most severe cases, and to loss of paternal contribution to growth in the mildest manifestation...
  6. ncbi request reprint A ketogenic diet increases brain insulin-like growth factor receptor and glucose transporter gene expression
    Clara M Cheng
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA
    Endocrinology 144:2676-82. 2003
    ..In addition, the data may be consistent with a role for enhanced IGF1R and GLUT expression in ketogenic diet-induced seizure suppression...
  7. ncbi request reprint Signaling by insulin-like growth factor 1 in brain
    Carolyn A Bondy
    Developmental Endocrinology Branch, National Institute of Child Health and Human Development NIH, Bldg 10 10N262, 10 Center Drive, Bethesda, MD 20892, USA
    Eur J Pharmacol 490:25-31. 2004
    ....
  8. ncbi request reprint Insulin-like growth factor-1 promotes neuronal glucose utilization during brain development and repair processes
    Carolyn A Bondy
    Developmental Endocrinology Branch, NICHD, NIH Bethesda, Maryland 20892, USA
    Int Rev Neurobiol 51:189-217. 2002
  9. pmc Autoimmune disorders in women with turner syndrome and women with karyotypically normal primary ovarian insufficiency
    Vladimir K Bakalov
    Section on Epigenetics and Development, Program on Developmental Endocrinology and Genetics, National Institute of Child Health and Human Development, 10 Center Dr CRC 1 3330 Bethesda, MD 20892 1103, USA
    J Autoimmun 38:315-21. 2012
    ..The absence of a normal second X-chromosome further contributes to increased autoimmunity in TS...