Katherine R Calvo

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. pmc Myelodysplasia in autosomal dominant and sporadic monocytopenia immunodeficiency syndrome: diagnostic features and clinical implications
    Katherine R Calvo
    Hematology Section, Department of Laboratory Medicine, NIH Clinical Center, 10 Center Dr, Bethesda, MD 20892 1508, USA
    Haematologica 96:1221-5. 2011
  2. ncbi request reprint Clinical proteomics: from biomarker discovery and cell signaling profiles to individualized personal therapy
    Katherine R Calvo
    Laboratory of Pathology, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
    Biosci Rep 25:107-25. 2005
  3. pmc IL-4 protein expression and basal activation of Erk in vivo in follicular lymphoma
    Katherine R Calvo
    Hematopathology Section, Laboratory of Pathology LP, NIH Clinical Center, Department of Laboratory Medicine, 10 Center Drive, Bldg 10 2C306, Bethesda, MD 20892 1500, USA
    Blood 112:3818-26. 2008
  4. ncbi request reprint Modeling progression risk for smoldering multiple myeloma: results from a prospective clinical study
    Benjamin M Cherry
    Multiple Myeloma Section, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
    Leuk Lymphoma 54:2215-8. 2013
  5. pmc MicroRNA profiling of follicular lymphoma identifies microRNAs related to cell proliferation and tumor response
    Weixin Wang
    Department of Laboratory Medicine, Hematology Section NIH Clinical Center, 10 Center Dr, Bldg 10 2C306 Bethesda, 20892 1508, USA
    Haematologica 97:586-94. 2012
  6. doi request reprint Proteomic signatures of epidermal growth factor receptor and survival signal pathways correspond to gefitinib sensitivity in head and neck cancer
    Francisco G Pernas
    Tumor Biology Section, Head and Neck Surgery Branch, National Institute on Deafness and Other Communication Disorders, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
    Clin Cancer Res 15:2361-72. 2009
  7. pmc Both variant and IGHV4-34-expressing hairy cell leukemia lack the BRAF V600E mutation
    Liqiang Xi
    Laboratory of Pathology, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
    Blood 119:3330-2. 2012
  8. pmc Mutations in GATA2 are associated with the autosomal dominant and sporadic monocytopenia and mycobacterial infection (MonoMAC) syndrome
    Amy P Hsu
    Laboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases NIAID, National Institutes of Health NIH, Bethesda, MD 20892 1684, USA
    Blood 118:2653-5. 2011
  9. pmc Eltrombopag restores trilineage hematopoiesis in refractory severe aplastic anemia that can be sustained on discontinuation of drug
    Ronan Desmond
    Hematology Branch, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD
    Blood 123:1818-25. 2014
  10. pmc Measurement of the absolute immature platelet number reflects marrow production and is not impacted by platelet transfusion
    Taha Bat
    Hematology Branch, National Heart, Lung, and BloodInstitute, National Institutes of Health, Bethesda, MD 20892, USA
    Transfusion 53:1201-4. 2013

Collaborators

Detail Information

Publications15

  1. pmc Myelodysplasia in autosomal dominant and sporadic monocytopenia immunodeficiency syndrome: diagnostic features and clinical implications
    Katherine R Calvo
    Hematology Section, Department of Laboratory Medicine, NIH Clinical Center, 10 Center Dr, Bethesda, MD 20892 1508, USA
    Haematologica 96:1221-5. 2011
    ..MonoMAC appears to be a unique, inherited syndrome of bone marrow failure. We describe distinctive bone marrow features to help in its recognition and diagnosis. (Clinicaltrials.gov identifiers: NCT00018044, NCT00923364, NCT01212055)...
  2. ncbi request reprint Clinical proteomics: from biomarker discovery and cell signaling profiles to individualized personal therapy
    Katherine R Calvo
    Laboratory of Pathology, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
    Biosci Rep 25:107-25. 2005
    ....
  3. pmc IL-4 protein expression and basal activation of Erk in vivo in follicular lymphoma
    Katherine R Calvo
    Hematopathology Section, Laboratory of Pathology LP, NIH Clinical Center, Department of Laboratory Medicine, 10 Center Drive, Bldg 10 2C306, Bethesda, MD 20892 1500, USA
    Blood 112:3818-26. 2008
    ..These findings suggest IL-4, Erk, and possibly Stat-6 may play a role in the biology of FL and may serve as targets for future therapies...
  4. ncbi request reprint Modeling progression risk for smoldering multiple myeloma: results from a prospective clinical study
    Benjamin M Cherry
    Multiple Myeloma Section, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
    Leuk Lymphoma 54:2215-8. 2013
    ..0001), low versus non-low (p = 0.0007) and high versus non-high (p < 0.0001) risk. There is a need for prospectively validated models to characterize individual patient risk of transformation to MM...
  5. pmc MicroRNA profiling of follicular lymphoma identifies microRNAs related to cell proliferation and tumor response
    Weixin Wang
    Department of Laboratory Medicine, Hematology Section NIH Clinical Center, 10 Center Dr, Bldg 10 2C306 Bethesda, 20892 1508, USA
    Haematologica 97:586-94. 2012
    ..MicroRNAs can play an important role in tumorigenesis through post-transcriptional regulation of gene expression, and are not well characterized in follicular lymphoma...
  6. doi request reprint Proteomic signatures of epidermal growth factor receptor and survival signal pathways correspond to gefitinib sensitivity in head and neck cancer
    Francisco G Pernas
    Tumor Biology Section, Head and Neck Surgery Branch, National Institute on Deafness and Other Communication Disorders, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
    Clin Cancer Res 15:2361-72. 2009
    ..To investigate the underlying molecular mechanism, the proteomic signatures and responses of EGFR and downstream signals have been studied in a panel of HNSCC cell lines and tumor specimens pre- and post-gefitinib treatment...
  7. pmc Both variant and IGHV4-34-expressing hairy cell leukemia lack the BRAF V600E mutation
    Liqiang Xi
    Laboratory of Pathology, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
    Blood 119:3330-2. 2012
    ..Our results suggest that HCLv and IGHV4-34(+) HCLs have a different pathogenesis than HCLc and that a significant minority of other HCLc are also wild-type for BRAF V600...
  8. pmc Mutations in GATA2 are associated with the autosomal dominant and sporadic monocytopenia and mycobacterial infection (MonoMAC) syndrome
    Amy P Hsu
    Laboratory of Clinical Infectious Diseases, National Institute of Allergy and Infectious Diseases NIAID, National Institutes of Health NIH, Bethesda, MD 20892 1684, USA
    Blood 118:2653-5. 2011
    ..Thus, GATA2 joins RUNX1 and CEBPA not only as a familial leukemia gene but also as a cause of a complex congenital immunodeficiency that evolves over decades and combines predisposition to infection and myeloid malignancy...
  9. pmc Eltrombopag restores trilineage hematopoiesis in refractory severe aplastic anemia that can be sustained on discontinuation of drug
    Ronan Desmond
    Hematology Branch, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD
    Blood 123:1818-25. 2014
    ..This study is registered at www.clinicaltrials.gov as #NCT00922883. ..
  10. pmc Measurement of the absolute immature platelet number reflects marrow production and is not impacted by platelet transfusion
    Taha Bat
    Hematology Branch, National Heart, Lung, and BloodInstitute, National Institutes of Health, Bethesda, MD 20892, USA
    Transfusion 53:1201-4. 2013
    ..The measurement of immature circulating PLTs based on RNA content using an automated counter is now feasible. This study evaluated the impact of recent PLT transfusion on measurement of immature PLT variables...
  11. pmc Feasibility and safety of sequential research-related tumor core biopsies in clinical trials
    Jung Min Lee
    Medical Oncology Branch, Center for Cancer Research, National Cancer Institute, Bethesda, MD 20892, USA
    Cancer 119:1357-64. 2013
    ..Definition of patient characteristics and optimal target tissue for safe research tumor biopsy in the era of antiangiogenic and targeted agents is needed...
  12. doi request reprint Role of microRNAs from monoclonal gammopathy of undetermined significance to multiple myeloma
    Katherine R Calvo
    Hematology Section, Department of Laboratory Medicine, National Institutes of Health Clinical Center, Bethesda, MD
    Semin Hematol 48:39-45. 2011
    ....
  13. doi request reprint Flow cytometry immunophenotyping of hematolymphoid neoplasia
    Katherine R Calvo
    Department of Laboratory Medicine, Hematology Section, National Institutes of Health Clinical Center, Bethesda, MD, USA
    Methods Mol Biol 699:295-316. 2011
    ..In this chapter, we present a general approach to analyze hematolymphoid populations. Specimen handling, gating strategies, and appropriate antibody panels for flow cytometric detection of abnormal hematologic populations are presented...
  14. ncbi request reprint Quantitative production of macrophages or neutrophils ex vivo using conditional Hoxb8
    Gang G Wang
    Department of Pathology and Molecular Pathology Graduate Program, School of Medicine, University of California at San Diego, 9500 Gilman Drive, La Jolla, CA 92093, USA
    Nat Methods 3:287-93. 2006
    ..Using knockout or small interfering RNA (siRNA) technologies, this ER-Hoxb8 phagocyte maturation system represents a rapid analytical tool for studying macrophage and neutrophil biology...
  15. ncbi request reprint Nup98-HoxA9 immortalizes myeloid progenitors, enforces expression of Hoxa9, Hoxa7 and Meis1, and alters cytokine-specific responses in a manner similar to that induced by retroviral co-expression of Hoxa9 and Meis1
    Katherine R Calvo
    University of California School of Medicine, Department of Pathology 9500 Gilman Drive, La Jolla, California, CA 92093 0612, USA
    Oncogene 21:4247-56. 2002
    ..Rapid leukaemogenesis by Nup98-HoxA9 may therefore result from both the intrinsic functions of Nup98-HoxA9, as well as of those of coexpressed HOX and MEIS1 genes...