Research Topics
| Sonja M BestSummaryAffiliation: National Institutes of Health Country: USA Publications
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Detail Information
Publications
Caspase activation during virus infection: more than just the kiss of death?Sonja M Best
Laboratory of Persistent Viral Diseases, NIAID, NIH, Rocky Mountain Labs, Hamilton, MT 59840, USA
Virology 320:191-4. 2004
Viral subversion of apoptotic enzymes: escape from death rowSonja M Best
Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, Montana 59840, USA
Annu Rev Microbiol 62:171-92. 2008..Examination of these interactions contributes to our understanding of both virus pathogenesis and the regulation of apoptotic enzymes in normal cellular functions...
Inhibition of interferon-stimulated JAK-STAT signaling by a tick-borne flavivirus and identification of NS5 as an interferon antagonistSonja M Best
Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, NIAID, NIH, Hamilton, MT 59840, USA
J Virol 79:12828-39. 2005..Because NS5 may interfere with both innate and acquired immune responses to virus infection, this protein may have a significant role in viral pathogenesis...
Pathogenesis of aleutian mink disease parvovirus and similarities to b19 infectionS M Best
Laboratory of Persistent Viral Diseases, NIAID, NIH Rocky Mountain Laboratories, 903 S Fourth St, Hamilton, MT 59840, USA
J Vet Med B Infect Dis Vet Public Health 52:331-4. 2005..Thus, a study of the regulation of NS1 expression and its interactions with cell signalling pathways may lead to increased understanding of the restricted replication of these two viruses, and perhaps of persistent infection...
Identification of genetic determinants of a tick-borne flavivirus associated with host-specific adaptation and pathogenicityDana N Mitzel
Laboratory of Virology, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, National Institutes of Health, 903 South Fourth Street, Hamilton, MT 59840, USA
Virology 381:268-76. 2008..Thus, our data implicate a role for E, M, NS3, NS4A and NS4B in host adaptation and pathogenicity of tick-borne flaviviruses...
Identification of residues critical for the interferon antagonist function of Langat virus NS5 reveals a role for the RNA-dependent RNA polymerase domainGregory S Park
Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, Hamilton, MT 59840, USA
J Virol 81:6936-46. 2007..This insight into the mechanism underlying flavivirus IFN evasion strategies will facilitate the design of antiviral therapeutics that potentiate the action of IFN during infection...
Tick-borne flaviviruses: dissecting host immune responses and virus countermeasuresShelly J Robertson
Laboratory of Virology, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, National Institutes of Health, 903 S 4th Street, Hamilton, MT 59840, USA
Immunol Res 43:172-86. 2009..This work will provide a more detailed understanding of early events in virus infection and their impact on flavivirus pathogenesis...
Tick-borne flavivirus infection in Ixodes scapularis larvae: development of a novel method for synchronous viral infection of ticksDana N Mitzel
Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, NIAID, NIH, Hamilton, MT 59840, USA
Virology 365:410-8. 2007....
Two mink parvoviruses use different cellular receptors for entry into CRFK cellsGregory S Park
Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, NIAID, NIH, Hamilton, MT 59840, USA
Virology 340:1-9. 2005..The use of two different receptors by MEV and ADV to infect the same cell line is yet another difference between these two parvoviruses that may contribute to their unique pathogenesis in mink...
Caspase cleavage of the nonstructural protein NS1 mediates replication of Aleutian mink disease parvovirusSonja M Best
Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, NIAID, NIH, Hamilton, Montana 59840, USA
J Virol 77:5305-12. 2003..The requirement for caspase activity during permissive replication suggests that limitation of caspase activation and apoptosis in vivo may be a novel approach to restricting virus replication...
TRIM79α, an interferon-stimulated gene product, restricts tick-borne encephalitis virus replication by degrading the viral RNA polymeraseR Travis Taylor
Innate Immunity and Pathogenesis Unit, Laboratory of Virology, Rocky Mountain Laboratories, Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, MT 59840, USA
Cell Host Microbe 10:185-96. 2011..The specificity of TRIM79α for TBEV reveals a remarkable ability of the innate IFN response to discriminate between closely related flaviviruses...
Caspase activation is required for permissive replication of Aleutian mink disease parvovirus in vitroSonja M Best
Laboratory of Persistent Viral Diseases, NIAID, NIH, Rocky Mountain Laboratories, 903 South Fourth Street, Hamilton, Montana 59840, USA
Virology 292:224-34. 2002..If ADV infection of cells in vivo fails to initiate caspase activation, the requirement of caspase activity for replication may not be met, thus providing a possible mechanism for persistent, restricted infection...
Antagonism of type I interferon responses by new world hantavirusesJessica R Levine
Rocky Mountain Laboratories, Hamilton, MT 59840, USA
J Virol 84:11790-801. 2010..These data suggest that IFN antagonism strategies by hantaviruses are quite variable, even between species with similar disease phenotypes, and may help to better elucidate species-specific pathogenesis...
Assessing ubiquitination of viral proteins: Lessons from flavivirus NS5R Travis Taylor
Innate Immunity and Pathogenesis Unit, Laboratory of Virology, Rocky Mountain Laboratories, DIR, NIAID, NIH, 903 S 4th St, Hamilton, MT 59840, USA
Methods 55:166-71. 2011....
The NS5 protein of the virulent West Nile virus NY99 strain is a potent antagonist of type I interferon-mediated JAK-STAT signalingMaudry Laurent-Rolle
Laboratory of Virology, Rocky Mountain Laboratories, 903 S Fourth St, Hamilton, MT 59840, USA
J Virol 84:3503-15. 2010..Thus, a naturally occurring mutation is associated with the function of NS5 in IFN antagonism and may influence virulence of WNV field isolates...
Identification of aleutian mink disease parvovirus capsid sequences mediating antibody-dependent enhancement of infection, virus neutralization, and immune complex formationM E Bloom
Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, Hamilton, Montana 59840, USA
J Virol 75:11116-27. 2001..The observation that antibodies against a single target peptide in the ADV capsid can mediate both neutralization and ADE may explain the failure of capsid-based vaccines...
Differential salivary gland transcript expression profile in Ixodes scapularis nymphs upon feeding or flavivirus infectionKristin L McNally
Laboratory of Virology, Rocky Mountain Laboratories, NIAID, NIH, 903 S Fourth St, Hamilton, MT 59840, USA
Ticks Tick Borne Dis 3:18-26. 2012..Further analysis of these transcripts may identify salivary factors that affect the transmission or replication of tick-borne flaviviruses...
