Yingli Wang

Summary

Affiliation: Mount Sinai School of Medicine
Country: USA

Publications

  1. pmc Activation of p38 MAPK pathway in the skull abnormalities of Apert syndrome Fgfr2(+P253R) mice
    Yingli Wang
    Department of Genetics and Genomic Sciences, Mount Sinai School of Medicine, New York, New York, USA
    BMC Dev Biol 10:22. 2010
  2. pmc p38 Inhibition ameliorates skin and skull abnormalities in Fgfr2 Beare-Stevenson mice
    Yingli Wang
    Department of Genetics and Genomic Sciences, Mount Sinai School of Medicine, New York, New York 10029, USA
    J Clin Invest 122:2153-64. 2012

Detail Information

Publications2

  1. pmc Activation of p38 MAPK pathway in the skull abnormalities of Apert syndrome Fgfr2(+P253R) mice
    Yingli Wang
    Department of Genetics and Genomic Sciences, Mount Sinai School of Medicine, New York, New York, USA
    BMC Dev Biol 10:22. 2010
    ..We previously reported an inbred transgenic mouse model with the Fgfr2 +/S252W mutation on the C57BL/6J background for Apert syndrome. Here we present a mouse model for the Fgfr2+/P253R mutation...
  2. pmc p38 Inhibition ameliorates skin and skull abnormalities in Fgfr2 Beare-Stevenson mice
    Yingli Wang
    Department of Genetics and Genomic Sciences, Mount Sinai School of Medicine, New York, New York 10029, USA
    J Clin Invest 122:2153-64. 2012
    ..The demonstration of a pathogenic role for p38 activation may lead to the development of therapeutic strategies for BSS and related conditions, such as acanthosis nigricans or craniosynostosis...