P T Ram

Summary

Affiliation: Mount Sinai School of Medicine
Country: USA

Publications

  1. ncbi request reprint Stat3-mediated transformation of NIH-3T3 cells by the constitutively active Q205L Galphao protein
    P T Ram
    Department of Pharmacology, Immunobiology Center, Mount Sinai School of Medicine, One Gustave L Levy Place, New York, NY 10029, USA
    Science 287:142-4. 2000
  2. ncbi request reprint G protein coupled receptor signaling through the Src and Stat3 pathway: role in proliferation and transformation
    P T Ram
    Department of Pharmacology, Mount Sinai School of Medicine, New York, NY 10029, USA
    Oncogene 20:1601-6. 2001
  3. pmc Expression of Q227L-galphas in MCF-7 human breast cancer cells inhibits tumorigenesis
    J Chen
    Department of Pharmacology, Mount Sinai School of Medicine, New York, NY 10029, USA
    Proc Natl Acad Sci U S A 95:2648-52. 1998

Detail Information

Publications3

  1. ncbi request reprint Stat3-mediated transformation of NIH-3T3 cells by the constitutively active Q205L Galphao protein
    P T Ram
    Department of Pharmacology, Immunobiology Center, Mount Sinai School of Medicine, One Gustave L Levy Place, New York, NY 10029, USA
    Science 287:142-4. 2000
    ..The results indicate that Stat3 can function as a downstream effector for Galphao* and mediate its biological effects...
  2. ncbi request reprint G protein coupled receptor signaling through the Src and Stat3 pathway: role in proliferation and transformation
    P T Ram
    Department of Pharmacology, Mount Sinai School of Medicine, New York, NY 10029, USA
    Oncogene 20:1601-6. 2001
    ..So increasingly it is becoming clear that the Src and Stat3 pathways are potential effectors for G proteins and that they may play a role in G protein function...
  3. pmc Expression of Q227L-galphas in MCF-7 human breast cancer cells inhibits tumorigenesis
    J Chen
    Department of Pharmacology, Mount Sinai School of Medicine, New York, NY 10029, USA
    Proc Natl Acad Sci U S A 95:2648-52. 1998
    ..Interactions between the Gs/adenylyl cyclase and MAPK-1,2 signaling pathways could be one mechanism by which expression of the transformed phenotype in mammary epithelial cells are regulated...