Research Topics
Genomes and GenesSpecies | Ping Ying PanSummaryAffiliation: Mount Sinai School of Medicine Country: USA Publications
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Detail Information
Publications
Advancements in immune tolerancePing Ying Pan
Department of Gene and Cell Medicine, Mount Sinai School od Medicine, New York, NY 10029, USA
Adv Drug Deliv Rev 60:91-105. 2008..This review briefly summarizes the major tolerogenic cell populations and their mechanisms of action, while focusing mainly on potential exploitation of their tolerogenic mechanisms for clinical applications...
Immune stimulatory receptor CD40 is required for T-cell suppression and T regulatory cell activation mediated by myeloid-derived suppressor cells in cancerPing Ying Pan
Department of Gene and Cell Medicine, Mount Sinai School of Medicine, New York, New York 10029, USA
Cancer Res 70:99-108. 2010..Blockade of CD40-CD40L interaction between MDSC and Treg may provide a new strategy to ablate tumoral immune suppression and thereby heighten responses to immunotherapy...
Myeloid-derived suppressor cells prevent type 1 diabetes in murine modelsBingjiao Yin
Department of Gene and Cell Medicine, Mount Sinai School of Medicine, New York, NY 10029, USA
J Immunol 185:5828-34. 2010....
Development and function of myeloid-derived suppressor cells generated from mouse embryonic and hematopoietic stem cellsZuping Zhou
Department of Gene and Cell Medicine, Mount Sinai School of Medicine, New York, New York 10029, USA
Stem Cells 28:620-32. 2010..The successful in vitro generation of MDSCs may represent a critical step toward potential clinical application of MDSCs...
Reversion of immune tolerance in advanced malignancy: modulation of myeloid-derived suppressor cell development by blockade of stem-cell factor functionPing Ying Pan
Department of Gene and Cell Medicine, Mount Sinai School of Medicine, New York, NY 10029, USA
Blood 111:219-28. 2008..This information supports the notion that modulation of MDSC development may be required to achieve effective immune-enhancing therapy for the treatment of advanced tumors...
Gr-1+CD115+ immature myeloid suppressor cells mediate the development of tumor-induced T regulatory cells and T-cell anergy in tumor-bearing hostBo Huang
Departments of Gene and Cell Medicine and General Surgery, Mount Sinai School of Medicine, One Gustave L Levy Place, New York, NY 10029, USA
Cancer Res 66:1123-31. 2006..Our data provide evidence that Gr-1(+)CD115(+) MSC can mediate the development of Treg in tumor-bearing mice and show a novel immune suppressive mechanism by which MSCs can suppress antitumor responses...
The novel role of tyrosine kinase inhibitor in the reversal of immune suppression and modulation of tumor microenvironment for immune-based cancer therapiesJunko Ozao-Choy
Departments of Gene and Cell Medicine, Mount Sinai School of Medicine, New York, New York, USA
Cancer Res 69:2514-22. 2009..These data suggest that sunitinib can be used to reverse immune suppression and as a potentially useful adjunct for enhancing the efficacy of immune-based cancer therapy for advanced malignancies...
Paired immunoglobin-like receptor-B regulates the suppressive function and fate of myeloid-derived suppressor cellsGe Ma
Department of Gene and Cell Medicine, Mount Sinai School of Medicine, 1425 Madison Avenue, Room 13 02, New York, NY 10029 6574, USA
Immunity 34:385-95. 2011..Inhibition of the PIR-B signaling pathway promoted MDSC differentiation into M1 macrophages...
Regulation of dendritic cell function by NK cells: mechanisms underlying the synergism in the combination therapy of IL-12 and 4-1BB activationPing Ying Pan
Carl C Icahn Institute for Gene Therapy and Molecular Medicine, Mount Sinai School of Medicine, New York, NY 10029, USA
J Immunol 172:4779-89. 2004....
NK and CD8+ T cell-mediated eradication of poorly immunogenic B16-F10 melanoma by the combined action of IL-12 gene therapy and 4-1BB costimulationDongping Xu
Carl C. Icahn Institute for Gene Therapy and Molecular Medicine, Mount Sinai School of Medicine, New York, NY 10029, USA
Int J Cancer 109:499-506. 2004....
OX40 ligation enhances primary and memory cytotoxic T lymphocyte responses in an immunotherapy for hepatic colon metastasesPing-Ying Pan
Carl C. Icahn Institute for Gene Therapy and Molecular Medicine, Mount Sinai School of Medicine, New York, New York 10029, USA
Mol Ther 6:528-36. 2002..mIL-12) + anti-4-1BB + anti-OX40 antibodies may provide a better treatment modality for patients with advanced cancers, often associated with a state of immune suppression or tolerance...
