Wenying Shou

Summary

Affiliation: Memorial Sloan-Kettering Cancer Center
Country: USA

Publications

  1. pmc Multiple telophase arrest bypassed (tab) mutants alleviate the essential requirement for Cdc15 in exit from mitosis in S. cerevisiae
    Wenying Shou
    Division of Biology, 156 29 Howard Hughes Medical Institute, California Institute of Technology, Pasadena, CA 91125, USA
    BMC Genet 3:4. 2002
  2. ncbi request reprint Mass spectrometry-based methods for phosphorylation site mapping of hyperphosphorylated proteins applied to Net1, a regulator of exit from mitosis in yeast
    Susan Loughrey Chen
    Proteomics and Biological Mass Spectrometry, GlaxoSmithKline, King of Prussia, Pennsylvania 19406, USA
    Mol Cell Proteomics 1:186-96. 2002
  3. pmc Loss of CDC5 function in Saccharomyces cerevisiae leads to defects in Swe1p regulation and Bfa1p/Bub2p-independent cytokinesis
    Chong Jin Park
    Laboratory of Metabolism, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
    Genetics 163:21-33. 2003
  4. pmc Synthetic cooperation in engineered yeast populations
    Wenying Shou
    Computational Biology Program, Memorial Sloan Kettering Cancer Center, New York, NY 10021, USA
    Proc Natl Acad Sci U S A 104:1877-82. 2007
  5. ncbi request reprint Phosphorylation by cyclin B-Cdk underlies release of mitotic exit activator Cdc14 from the nucleolus
    Ramzi Azzam
    Division of Biology, California Institute of Technology, Pasadena, CA 91125, USA
    Science 305:516-9. 2004

Collaborators

  • Raymond J Deshaies
  • Ramzi Azzam
  • Roland S Annan
  • Steven A Carr
  • Chong Jin Park
  • Susan Loughrey Chen
  • Angie S Mah
  • Kim Nasmyth
  • Gabriela Alexandru
  • Susan L Chen
  • Philip R Lee
  • Sukgil Song
  • Kyung S Lee
  • Michael J Huddleston

Detail Information

Publications5

  1. pmc Multiple telophase arrest bypassed (tab) mutants alleviate the essential requirement for Cdc15 in exit from mitosis in S. cerevisiae
    Wenying Shou
    Division of Biology, 156 29 Howard Hughes Medical Institute, California Institute of Technology, Pasadena, CA 91125, USA
    BMC Genet 3:4. 2002
    ..However, the remaining tab mutants were not characterized...
  2. ncbi request reprint Mass spectrometry-based methods for phosphorylation site mapping of hyperphosphorylated proteins applied to Net1, a regulator of exit from mitosis in yeast
    Susan Loughrey Chen
    Proteomics and Biological Mass Spectrometry, GlaxoSmithKline, King of Prussia, Pennsylvania 19406, USA
    Mol Cell Proteomics 1:186-96. 2002
    ..The failure of any single method to identify all sites in highly phosphorylated Net1N, however, raises significant concerns about how feasible it is to map phosphorylation sites throughout the proteome using existing technologies...
  3. pmc Loss of CDC5 function in Saccharomyces cerevisiae leads to defects in Swe1p regulation and Bfa1p/Bub2p-independent cytokinesis
    Chong Jin Park
    Laboratory of Metabolism, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
    Genetics 163:21-33. 2003
    ..Thus, Cdc5p contributes to the activation of the Swe1p-dependent Cdc28p/Clb pathway, normal septin function, and cytokinesis...
  4. pmc Synthetic cooperation in engineered yeast populations
    Wenying Shou
    Computational Biology Program, Memorial Sloan Kettering Cancer Center, New York, NY 10021, USA
    Proc Natl Acad Sci U S A 104:1877-82. 2007
    ....
  5. ncbi request reprint Phosphorylation by cyclin B-Cdk underlies release of mitotic exit activator Cdc14 from the nucleolus
    Ramzi Azzam
    Division of Biology, California Institute of Technology, Pasadena, CA 91125, USA
    Science 305:516-9. 2004
    ..Thus, a regulatory circuit exists to ensure that the arbiter of the mitotic state, Cdk, sets in motion events that culminate in exit from mitosis...