Research Topics
| Tomoyo OkadaSummaryAffiliation: Memorial Sloan-Kettering Cancer Center Country: USA Publications
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Detail Information
Publications
Merlin/NF-2 mediates contact inhibition of growth by suppressing recruitment of Rac to the plasma membraneTomoyo Okada
Cell Biology Program, Memorial Sloan Kettering Cancer Center, New York, NY 10021, USA
J Cell Biol 171:361-71. 2005..These results provide a framework for understanding the tumor suppressor function of Merlin and indicate that Merlin mediates contact inhibition of growth by suppressing recruitment of Rac to matrix adhesions...
Shedding light on Merlin's wizardryTomoyo Okada
Cell Biology Program, Sloan Kettering Institute for Cancer Research, Memorial Sloan Kettering Cancer Center, New York, NY 10021, USA
Trends Cell Biol 17:222-9. 2007..Here, we review these recent findings and their relevance to the tumor suppressor function of Merlin...
Loss of the tumor suppressor gene NF2, encoding merlin, constitutively activates integrin-dependent mTORC1 signalingMiguel A López-Lago
Cell Biology Program, Sloan Kettering Institute for Cancer Research, Memorial Sloan Kettering Cancer Center, 1275 York Ave, New York, NY 10065, USA
Mol Cell Biol 29:4235-49. 2009....
Restoration of C/EBPα in dedifferentiated liposarcoma induces G2/M cell cycle arrest and apoptosisYuhsin V Wu
Sarcoma Biology Laboratory, Sarcoma Disease Management Program, Department of Surgery, Memorial Sloan Kettering Cancer Center, New York, NY 10021, USA
Genes Chromosomes Cancer 51:313-27. 2012..These results demonstrate that loss of C/EBPα is an important factor in suppressing apoptosis and maintaining the dedifferentiated state in DDLS. Restoring C/EBPα may be a useful therapeutic approach for DDLS...
Single- and multiple-molecule dynamics of the signaling from H-Ras to cRaf-1 visualized on the plasma membrane of living cellsKayo Hibino
Department of Systems and Human Science, Graduate School of Engineering Science, Osaka University, 1-3 Machikaneyama-cho, Toyonaka 560-8531, Japan
Chemphyschem 4:748-53. 2003
