Haowei Shen

Summary

Affiliation: Medical University of South Carolina
Country: USA

Publications

  1. ncbi Heroin relapse requires long-term potentiation-like plasticity mediated by NMDA2b-containing receptors
    Haowei Shen
    Department of Neurosciences, Medical University of South Carolina, Charleston, SC 29425, USA
    Proc Natl Acad Sci U S A 108:19407-12. 2011
  2. ncbi Automated quantification of dendritic spine density and spine head diameter in medium spiny neurons of the nucleus accumbens
    Haowei Shen
    Department of Neurosciences, Medical University of South Carolina, 173 Ashley Avenue, BSB 403, Charleston, SC 29425, USA
    Brain Struct Funct 213:149-57. 2008
  3. ncbi Relapse induced by cues predicting cocaine depends on rapid, transient synaptic potentiation
    Cassandra D Gipson
    Department of Neurosciences, Medical University of South Carolina, Charleston, SC 29425, USA
    Neuron 77:867-72. 2013
  4. ncbi Reversing cocaine-induced synaptic potentiation provides enduring protection from relapse
    Khaled Moussawi
    Department of Neurosciences, Medical University of South Carolina, Charleston, SC 29425, USA
    Proc Natl Acad Sci U S A 108:385-90. 2011
  5. ncbi Inhibition of actin polymerization prevents cocaine-induced changes in spine morphology in the nucleus accumbens
    Shigenobu Toda
    Department of Neurosciences, Medical University of South Carolina, 173 Ashley Ave, BSB410, Charleston, SC 29425, USA
    Neurotox Res 18:410-5. 2010
  6. ncbi Role of mGluR5 neurotransmission in reinstated cocaine-seeking
    Xiusong Wang
    Department of Neurosciences, Medical University of South Carolina, Charleston, USA
    Addict Biol 18:40-9. 2013
  7. ncbi NAC1 regulates the recruitment of the proteasome complex into dendritic spines
    Haowei Shen
    Department of Neurosciences, Medical University of South Carolina, Charleston, South Carolina 29425, USA
    J Neurosci 27:8903-13. 2007
  8. ncbi Glutamate transmission in addiction
    Peter W Kalivas
    Department of Neurosciences, 173 Ashley Ave, BSB410, Medical University of South Carolina, Charleston, SC 29464, USA
    Neuropharmacology 56:169-73. 2009

Detail Information

Publications8

  1. ncbi Heroin relapse requires long-term potentiation-like plasticity mediated by NMDA2b-containing receptors
    Haowei Shen
    Department of Neurosciences, Medical University of South Carolina, Charleston, SC 29425, USA
    Proc Natl Acad Sci U S A 108:19407-12. 2011
    ..Moreover, the data reveal NR2B-containing NMDA receptors as a previously unexplored therapeutic target for treating heroin addiction...
  2. ncbi Automated quantification of dendritic spine density and spine head diameter in medium spiny neurons of the nucleus accumbens
    Haowei Shen
    Department of Neurosciences, Medical University of South Carolina, 173 Ashley Avenue, BSB 403, Charleston, SC 29425, USA
    Brain Struct Funct 213:149-57. 2008
    ..The capacity to rapidly assess spine density and spine head diameter will facilitate quantifying spine plasticity induced by pharmacological and environmental manipulations...
  3. ncbi Relapse induced by cues predicting cocaine depends on rapid, transient synaptic potentiation
    Cassandra D Gipson
    Department of Neurosciences, Medical University of South Carolina, Charleston, SC 29425, USA
    Neuron 77:867-72. 2013
    ..The marked cue-induced synaptic changes in the accumbens were correlated with the intensity of cocaine, but not sucrose seeking, and may explain the difficulty addicts experience in managing relapse to cocaine use...
  4. ncbi Reversing cocaine-induced synaptic potentiation provides enduring protection from relapse
    Khaled Moussawi
    Department of Neurosciences, Medical University of South Carolina, Charleston, SC 29425, USA
    Proc Natl Acad Sci U S A 108:385-90. 2011
    ....
  5. ncbi Inhibition of actin polymerization prevents cocaine-induced changes in spine morphology in the nucleus accumbens
    Shigenobu Toda
    Department of Neurosciences, Medical University of South Carolina, 173 Ashley Ave, BSB410, Charleston, SC 29425, USA
    Neurotox Res 18:410-5. 2010
    ....
  6. ncbi Role of mGluR5 neurotransmission in reinstated cocaine-seeking
    Xiusong Wang
    Department of Neurosciences, Medical University of South Carolina, Charleston, USA
    Addict Biol 18:40-9. 2013
    ..Taken together, these data show that mGluR5 inhibition and stimulation in the NAcore can regulate cocaine-seeking, and demonstrate that one mechanism for this effect is via interactions with Homer proteins...
  7. ncbi NAC1 regulates the recruitment of the proteasome complex into dendritic spines
    Haowei Shen
    Department of Neurosciences, Medical University of South Carolina, Charleston, South Carolina 29425, USA
    J Neurosci 27:8903-13. 2007
    ..These experiments show that NAC1 modulates the translocation of the UPS from the nucleus into dendritic spines, thereby suggesting a potential missing link in the recruitment of necessary proteolysis machinery for synaptic remodeling...
  8. ncbi Glutamate transmission in addiction
    Peter W Kalivas
    Department of Neurosciences, 173 Ashley Ave, BSB410, Medical University of South Carolina, Charleston, SC 29464, USA
    Neuropharmacology 56:169-73. 2009
    ..This information points to the value of exploring pharmacotherapeutic manipulation of glutamate plasticity in treating drug addiction...