J E Baker

Summary

Affiliation: Medical College of Wisconsin
Country: USA

Publications

  1. pmc Intestinal microbiota determine severity of myocardial infarction in rats
    Vy Lam
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    FASEB J 26:1727-35. 2012
  2. pmc Gadolinium decreases inflammation related to myocardial ischemia and reperfusion injury
    Jennifer L Strande
    Division of Cardiovascular Medicine, Medical College of Wisconsin, Milwaukee, Wisconsin, USA
    J Inflamm (Lond) 6:34. 2009
  3. ncbi request reprint Resistance to myocardial ischemia in five rat strains: is there a genetic component of cardioprotection?
    J E Baker
    Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee, Wisconsin 53226, USA
    Am J Physiol Heart Circ Physiol 278:H1395-400. 2000
  4. ncbi request reprint Darbepoetin alfa protects the rat heart against infarction: dose-response, phase of action, and mechanisms
    John E Baker
    Division of Cardiothoracic Surgery Department of Pharmacology and Toxicology, Medical College of Wisconsin, Milwaukee, Wisconsin 53226, USA
    J Cardiovasc Pharmacol 49:337-45. 2007
  5. pmc Nitrite confers protection against myocardial infarction: role of xanthine oxidoreductase, NADPH oxidase and K(ATP) channels
    John E Baker
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, Milwaukee, WI 53226, USA
    J Mol Cell Cardiol 43:437-44. 2007
  6. pmc Human thrombopoietin reduces myocardial infarct size, apoptosis, and stunning following ischaemia/reperfusion in rats
    John E Baker
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    Cardiovasc Res 77:44-53. 2008
  7. ncbi request reprint Oxidative stress and adaptation of the infant heart to hypoxia and ischemia
    John E Baker
    Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee, WI, USA
    Antioxid Redox Signal 6:423-9. 2004
  8. pmc 10 Gy total body irradiation increases risk of coronary sclerosis, degeneration of heart structure and function in a rat model
    John E Baker
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    Int J Radiat Biol 85:1089-100. 2009
  9. ncbi request reprint Erythropoietin mimics ischemic preconditioning
    John E Baker
    Pharmacology and Toxicology, Biochemistry and Pediatric Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    Vascul Pharmacol 42:233-41. 2005
  10. pmc Radiation as a risk factor for cardiovascular disease
    John E Baker
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    Antioxid Redox Signal 15:1945-56. 2011

Collaborators

Detail Information

Publications41

  1. pmc Intestinal microbiota determine severity of myocardial infarction in rats
    Vy Lam
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    FASEB J 26:1727-35. 2012
    ..This proof-of-concept study is the first to identify a mechanistic link between changes in intestinal microbiota and myocardial infarction and demonstrates that a probiotic supplement can reduce myocardial infarct size...
  2. pmc Gadolinium decreases inflammation related to myocardial ischemia and reperfusion injury
    Jennifer L Strande
    Division of Cardiovascular Medicine, Medical College of Wisconsin, Milwaukee, Wisconsin, USA
    J Inflamm (Lond) 6:34. 2009
    ..We determined the impact of GdCl3 treatment for reperfusion injury on 1) circulating monoctye and neutrophil counts, 2) secretion of inflammatory cytokines, and 3) influx of monocytes and neutrophils into the myocardium...
  3. ncbi request reprint Resistance to myocardial ischemia in five rat strains: is there a genetic component of cardioprotection?
    J E Baker
    Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee, Wisconsin 53226, USA
    Am J Physiol Heart Circ Physiol 278:H1395-400. 2000
    ....
  4. ncbi request reprint Darbepoetin alfa protects the rat heart against infarction: dose-response, phase of action, and mechanisms
    John E Baker
    Division of Cardiothoracic Surgery Department of Pharmacology and Toxicology, Medical College of Wisconsin, Milwaukee, Wisconsin 53226, USA
    J Cardiovasc Pharmacol 49:337-45. 2007
    ..Dpa confers immediate and sustained cardioprotection in rats, suggesting a potential therapeutic role of this long-acting erythropoietin analog for the treatment of acute myocardial infarction...
  5. pmc Nitrite confers protection against myocardial infarction: role of xanthine oxidoreductase, NADPH oxidase and K(ATP) channels
    John E Baker
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, Milwaukee, WI 53226, USA
    J Mol Cell Cardiol 43:437-44. 2007
    ..Inhibition of NADPH oxidase and K(ATP) channels abolished nitrite-induced cardioprotection. Nitrite but not nitrate protects against infarction by a mechanism involving xanthine oxidoreductase, NADPH oxidase and K(ATP) channels...
  6. pmc Human thrombopoietin reduces myocardial infarct size, apoptosis, and stunning following ischaemia/reperfusion in rats
    John E Baker
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    Cardiovasc Res 77:44-53. 2008
    ..Thrombopoietin (Tpo) is known for its ability to stimulate platelet production. However, it is currently unknown whether Tpo plays a physiological function in the heart...
  7. ncbi request reprint Oxidative stress and adaptation of the infant heart to hypoxia and ischemia
    John E Baker
    Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee, WI, USA
    Antioxid Redox Signal 6:423-9. 2004
    ..Thus, NOS3 appears to be critically important in adaptation of infant hearts to chronic hypoxia and in resistance to subsequent ischemia by regulating the production of reactive oxygen and nitrogen species...
  8. pmc 10 Gy total body irradiation increases risk of coronary sclerosis, degeneration of heart structure and function in a rat model
    John E Baker
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    Int J Radiat Biol 85:1089-100. 2009
    ..To determine the impact of 10 Gy total body irradiation (TBI) or local thorax irradiation, a dose relevant to a radiological terrorist threat, on lipid and liver profile, coronary microvasculature and ventricular function...
  9. ncbi request reprint Erythropoietin mimics ischemic preconditioning
    John E Baker
    Pharmacology and Toxicology, Biochemistry and Pediatric Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    Vascul Pharmacol 42:233-41. 2005
    ..Cardioprotection conferred by Epo has been shown to be equal in magnitude to that conferred by ischemic preconditioning. However, the underlying mechanisms by which Epo protects the heart against injury caused by ischemia remain unknown...
  10. pmc Radiation as a risk factor for cardiovascular disease
    John E Baker
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    Antioxid Redox Signal 15:1945-56. 2011
    ....
  11. ncbi request reprint Adaptation to chronic hypoxia confers tolerance to subsequent myocardial ischemia by increased nitric oxide production
    J E Baker
    Department of Cardiothoracic Surgery, Medical College of Wisconsin, Milwaukee 53226, USA
    Ann N Y Acad Sci 874:236-53. 1999
    ..Increased tolerance to ischemia in rabbit hearts adapted to chronic hypoxia is associated with increased expression of constitutive nitric oxide synthase...
  12. ncbi request reprint Nitric oxide activates the sarcolemmal K(ATP) channel in normoxic and chronically hypoxic hearts by a cyclic GMP-dependent mechanism
    J E Baker
    Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee, WI 53226, USA
    J Mol Cell Cardiol 33:331-41. 2001
    ..The effect of ODQ could not be reversed by GSNO. We conclude that NO activates the sarcolemmal K(ATP) channel in normoxic and chronically hypoxic hearts by a cyclic GMP-dependent mechanism...
  13. ncbi request reprint Preconditioning in immature rabbit hearts: role of KATP channels
    J E Baker
    Division of Cardiothoracic Surgery, Department of Pharmacology and Medical College of Wisconsin, Milwaukee, USA
    Circulation 99:1249-54. 1999
    ..We determined whether immature hearts normoxic or chronically hypoxic from birth could be preconditioned, the time window or memory of the cardioprotective effect, and the involvement of the KATP channel...
  14. ncbi request reprint Increased tolerance of the chronically hypoxic immature heart to ischemia. Contribution of the KATP channel
    J E Baker
    Department of Pharmacology, Medical College of Wisconsin, Milwaukee 53226, USA
    Circulation 95:1278-85. 1997
    ..We determined whether this increased tolerance to ischemia was due to an alteration in the ATP-sensitive potassium (KATP) channel and whether increased KATP channel activation was associated with increases in intracellular lactate...
  15. ncbi request reprint Increased mitochondrial K(ATP) channel activity during chronic myocardial hypoxia: is cardioprotection mediated by improved bioenergetics?
    J T Eells
    Department of Pharmacology, Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee, Wisconsin, USA
    Circ Res 87:915-21. 2000
    ..We conclude that mitochondrial K(ATP) channels are activated in chronically hypoxic rabbit hearts and implicate activation of this channel in the improved mitochondrial bioenergetics and cardioprotection observed...
  16. ncbi request reprint Sarcolemmal and mitochondrial K(atp)channels mediate cardioprotection in chronically hypoxic hearts
    X Kong
    Division of Pediatric Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    J Mol Cell Cardiol 33:1041-5. 2001
    ..The combination of 5-HD and HMR 1098 abolished the cardioprotective effect of chronic hypoxia. We conclude that both sarcolemmal and mitochondrial K(ATP)channels contribute to cardioprotection in the chronically hypoxic heart...
  17. ncbi request reprint Vasodilatory and toxic effects of spin traps on aerobic cardiac function
    E A Konorev
    Biophysics Research Institute, Medical College of Wisconsin, Milwaukee 53226
    Free Radic Biol Med 14:127-37. 1993
    ..We conclude that all spin traps examined acted as coronary vasodilators. Their negative chronotropic and inotropic effects were minimal in comparison and only manifest at the higher concentrations studied...
  18. ncbi request reprint Chronic myocardial hypoxia increases nitric oxide synthase and decreases caveolin-3
    Y Shi
    Division of Pediatric Surgery, Milwaukee, WI 53226, USA
    Free Radic Biol Med 29:695-703. 2000
    ..2 +/- 0.5-fold. These data suggest that a decrease in caveolin-3 plays a role in the mechanisms by which chronic hypoxia increases NOS3 activity in the myocardium...
  19. ncbi request reprint Cardioprotection in chronically hypoxic rabbits persists on exposure to normoxia: role of NOS and KATP channels
    Colleen M Fitzpatrick
    Division of Pediatric Surgery, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    Am J Physiol Heart Circ Physiol 288:H62-8. 2005
    ..We conclude that cardioprotection conferred by adaptation to hypoxia from birth persists on subsequent exposure to normoxia and is associated with enhanced NOS activity and activation of K(ATP) channels...
  20. ncbi request reprint Activation of protein kinases in chronically hypoxic infant human and rabbit hearts: role in cardioprotection
    Parvaneh Rafiee
    Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee 53226, USA
    Circulation 106:239-45. 2002
    ..However, the signaling pathways by which infant hearts adapt to chronic hypoxia and resist subsequent surgical ischemia is unknown...
  21. ncbi request reprint Delayed cardioprotection by isoflurane: role of K(ATP) channels
    Marija Tonkovic-Capin
    Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee, 53226, USA
    Am J Physiol Heart Circ Physiol 283:H61-8. 2002
    ..We conclude that both mitochondrial and sarcolemmal K(ATP) channels contribute to isoflurane-induced delayed cardioprotection...
  22. ncbi request reprint Spin label oximetry to assess extracellular oxygen during myocardial ischemia
    J E Baker
    Department of Cardiothoracic Surgery, Medical College of Wisconsin, Milwaukee 53226, USA
    Free Radic Biol Med 22:109-15. 1997
    ..05) and 61 +/- 5 mumol/liter (p < .05), respectively. We conclude that spin-label oximetry can directly and precisely measure myocardial oxygen consumption at constant temperature during ischemia in the intact heart...
  23. ncbi request reprint Acute cardioprotective effects of erythropoietin in infant rabbits are mediated by activation of protein kinases and potassium channels
    Yang Shi
    Pediatric Surgery, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    Basic Res Cardiol 99:173-82. 2004
    ..We conclude the rapid activation of potassium channels and protein kinases by erythropoietin represents an important new mechanism for increasing cardioprotection...
  24. ncbi request reprint Erythropoietin protects the infant heart against ischemia-reperfusion injury by triggering multiple signaling pathways
    Parvaneh Rafiee
    Medical College of Wisconsin, Division of Pediatric Surgery, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    Basic Res Cardiol 100:187-97. 2005
    ..SB203580 inhibited only p38 MAPK activation by EPO. We can conclude EPO increases immediate cardioprotection through the activation of multiple signal transduction pathways...
  25. ncbi request reprint Hyperoxic and hyperbaric-induced cardioprotection: role of nitric oxide synthase 3
    Bernadette P Cabigas
    Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee, WI 53226, USA
    Cardiovasc Res 72:143-51. 2006
    ..We determined whether the product of FIO2 x ATM (oxygen partial pressure) controls the extent of hyperoxic+hyperbaric-induced cardioprotection and involves activation of nitric oxide synthase (NOS)...
  26. ncbi request reprint Cellular redistribution of inducible Hsp70 protein in the human and rabbit heart in response to the stress of chronic hypoxia: role of protein kinases,
    Parvaneh Rafiee
    Division of Pediatric Surgery, Medical College of Wisconsin and Children s Hospital of Wisconsin, Milwaukee, Wisconsin 53226, USA
    J Biol Chem 278:43636-44. 2003
    ..We conclude the Hsp70 signal transduction pathway plays an important role in adaptation of infant human and rabbit hearts to chronic hypoxemia...
  27. ncbi request reprint Heat shock protein 90 mediates the balance of nitric oxide and superoxide anion from endothelial nitric-oxide synthase
    K A Pritchard
    Division of Pediatric Surgery, Department of Pharmacology and Toxicology, Medical College of Wisconsin, Milwaukee, Wisconsin 53226, USA
    J Biol Chem 276:17621-4. 2001
    ..These data show that Hsp90 is essential for eNOS-dependent.NO production and that inhibition of ATP-dependent conformational changes in Hsp90 uncouples eNOS activity and increases eNOS-dependent O(2) production...
  28. pmc Parstatin: a cryptic peptide involved in cardioprotection after ischaemia and reperfusion injury
    Jennifer L Strande
    Division of Cardiovascular Medicine, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI, USA
    Cardiovasc Res 83:325-34. 2009
    ..Although much research has focused on the activated receptor, little is known about the 41-amino acid N-terminal peptide (parstatin). We hypothesized that parstatin would protect the heart against ischaemia-reperfusion injury...
  29. ncbi request reprint Mitochondrial big conductance KCa channel and cardioprotection in infant rabbit heart
    Yang Shi
    Surgery Division of Pediatric Surgery, Medical College of Wisconsin, Mikwaukee, WI 53226, USA
    J Cardiovasc Pharmacol 50:497-502. 2007
    ..Paxilline did not affect normoxic or hypoxic hearts. Activation of mitoBKCa protects normoxic infant rabbit hearts; however, cardioprotection by chronic hypoxia in infant rabbits does not appear involve mitoBKCa...
  30. ncbi request reprint BN phenome: detailed characterization of the cardiovascular, renal, and pulmonary systems of the sequenced rat
    Anne E Kwitek
    Department of Physiology, Medical College of Wisconsin, Milwaukee, Wisconsin 53226, USA
    Physiol Genomics 25:303-13. 2006
    ..These data provide a baseline for physiological comparison of traits related to cardiovascular, lung, blood, and renal function in the sequenced BN rats relative to the major strains of rats studied in biomedical research...
  31. ncbi request reprint Chronic hypoxia increases endothelial nitric oxide synthase generation of nitric oxide by increasing heat shock protein 90 association and serine phosphorylation
    Yang Shi
    Division of Pediatric Surgery, Medical College of Wisconsin, Milwaukee, WI 53226, USA
    Circ Res 91:300-6. 2002
    ..Such changes in eNOS function appear to play a critical role in protecting the myocardium against ischemic injury...
  32. pmc Evidence for a role of opioids in epoxyeicosatrienoic acid-induced cardioprotection in rat hearts
    Garrett J Gross
    Dept of Pharmacology and Toxicology, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    Am J Physiol Heart Circ Physiol 298:H2201-7. 2010
    ..Taken together, these results suggest that the major cardioprotective effects of the EETs depend on activation of a G(i/o) protein-coupled delta- and/or kappa-opioid receptor...
  33. pmc Chronic hypoxia enhances 15-lipoxygenase-mediated vasorelaxation in rabbit arteries
    Nitin T Aggarwal
    Dept of Pharmacology and Toxicology, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    Am J Physiol Heart Circ Physiol 296:H678-88. 2009
    ..Therefore, hypoxia increased the relaxations to agonists in the rabbit mesenteric arteries by enhancing endothelial 15-LO-1 expression and synthesis of the hyperpolarizing factors THETA and HEETA...
  34. pmc Gadolinium limits myocardial infarction in the rat: dose-response, temporal relations and mechanisms
    Alfred C Nicolosi
    Division of Cardiothoracic Surgery, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    J Mol Cell Cardiol 44:345-51. 2008
    ..The mechanism underlying Gd-induced preconditioning appears to be multi-factorial, involving JAK-2, STAT-3 and p44 MAPK pathways, as well as K(ATP) channels...
  35. pmc Inhibiting protease-activated receptor 4 limits myocardial ischemia/reperfusion injury in rat hearts by unmasking adenosine signaling
    Jennifer L Strande
    Division of Cardiovascular Medicine, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    J Pharmacol Exp Ther 324:1045-54. 2008
    ..Thrombin receptor blockade by PAR4 inhibition provides protection against injury from myocardial I/R by unmasking adenosine receptor signaling and supports the hypothesis of a coupling between thrombin receptors and adenosine receptors...
  36. ncbi request reprint Delayed cardioprotection with isoflurane: role of reactive oxygen and nitrogen
    Yang Shi
    Division of Pediatric Surgery, Free Radical Research Center, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    Am J Physiol Heart Circ Physiol 288:H175-84. 2005
    ..8% but not in 1.0% isoflurane-treated hearts. We conclude that isoflurane confers delayed cardioprotection in the adult rat, triggered by ROS and RNS...
  37. ncbi request reprint Increased resistance to myocardial ischemia in the Brown Norway vs. Dahl S rat: role of nitric oxide synthase and Hsp90
    Yang Shi
    Division of Pediatric Surgery, Medical College of Wisconsin, 8701 Watertown Plank Road, Milwaukee, WI 53226, USA
    J Mol Cell Cardiol 38:625-35. 2005
    ..NO production and decreases uncoupled NOS3 activity. We conclude increased association of hsp90 with NOS3 is a major mechanism by which BN/Mcw hearts are more resistant to ischemia than SS/Mcw hearts...
  38. pmc SCH 79797, a selective PAR1 antagonist, limits myocardial ischemia/reperfusion injury in rat hearts
    Jennifer L Strande
    Division of Cardiovascular Medicine, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, USA
    Basic Res Cardiol 102:350-8. 2007
    ....
  39. pmc Monomeric structure of the cardioprotective chemokine SDF-1/CXCL12
    Christopher T Veldkamp
    Department of Biochemistry, Medical College of Wisconsin, Milwaukee, Wisconsin 53226, USA
    Protein Sci 18:1359-69. 2009
    ..We conclude that interactions with the alignment medium biased the previous structure, masking flexibility in the helix position that may be essential for the distinct functional properties of the SDF-1 monomer...
  40. ncbi request reprint Chronic hypoxia activates lung 15-lipoxygenase, which catalyzes production of 15-HETE and enhances constriction in neonatal rabbit pulmonary arteries
    Daling Zhu
    Department of Medicine, Medical College of Wisconsin, Milwaukee, Wis 53226, USA
    Circ Res 92:992-1000. 2003
    ..Although a clear causal relationship remains to be demonstrated, these data suggest a previously unrecognized role for 15-LO in hypoxic vasoconstriction in neonatal mammals...
  41. ncbi request reprint Antagonizing reactive oxygen by treatment with a manganese (III) metalloporphyrin-based superoxide dismutase mimetic in cardiac transplants
    Vani Nilakantan
    Department of Surgery, Division of Transplant Surgery, Medical College of Wisconsin, Milwaukee, Wis 53226, USA
    J Thorac Cardiovasc Surg 131:898-906. 2006
    ....

Research Grants18

  1. CHRONIC HYPOXIA AND RESISTANCE TO MYOCARDIAL ISCHEMIA
    John E Baker; Fiscal Year: 2010
    ..The results of these studies are expected to have an impact on our understanding of the biology of Tpo in heart and may lead to the development of new therapeutics for the treatment of myocardial infarction. ..
  2. CHRONIC HYPOXIA AND RESISTANCE TO MYOCARDIAL ISCHEMIA
    John Baker; Fiscal Year: 2009
    ..The results of these studies are expected to have an impact on our understanding of the biology of Tpo in heart and may lead to the development of new therapeutics for the treatment of myocardial infarction. ..
  3. Radiation injury to the heart
    John Baker; Fiscal Year: 2009
    ..1 ..
  4. CHRONIC HYPOXIA AND RESISTANCE TO MYOCARDIAL ISCHEMIA
    John Baker; Fiscal Year: 2007
    ..The results of these studies are expected to have an impact on our understanding of the biology of Tpo in heart and may lead to the development of new therapeutics for the treatment of myocardial infarction. ..
  5. CHRONIC HYPOXIA AND RESISTANCE TO MYOCARDIAL ISCHEMIA
    John Baker; Fiscal Year: 2005
    ..abstract_text> ..
  6. CHRONIC HYPOXIA AND RESISTANCE TO MYOCARDIAL ISCHEMIA
    John Baker; Fiscal Year: 2004
    ..abstract_text> ..
  7. GENETICS AND INTERMITTENT MYOCARDIAL HYPOXIA
    John Baker; Fiscal Year: 2003
    ....
  8. CHRONIC HYPOXIA AND RESISTANCE TO MYOCARDIAL ISCHEMIA
    John Baker; Fiscal Year: 2003
    ..abstract_text> ..
  9. GENETICS AND INTERMITTENT MYOCARDIAL HYPOXIA
    John Baker; Fiscal Year: 2002
    ....
  10. CHRONIC HYPOXIA AND RESISTANCE TO MYOCARDIAL ISCHEMIA
    John Baker; Fiscal Year: 2002
    ..abstract_text> ..
  11. GENETICS AND INTERMITTENT MYOCARDIAL HYPOXIA
    John Baker; Fiscal Year: 2001
    ....
  12. GENETICS AND INTERMITTENT MYOCARDIAL HYPOXIA
    John Baker; Fiscal Year: 2000
    ....
  13. Radiation injury to the heart
    John E Baker; Fiscal Year: 2010
    ..1 ..