S Sethi

Summary

Affiliation: Mayo Clinic
Country: USA

Publications

  1. ncbi request reprint C3 glomerulonephritis: clinicopathological findings, complement abnormalities, glomerular proteomic profile, treatment, and follow-up
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Kidney Int 82:465-73. 2012
  2. doi request reprint Etiology-based diagnostic approach to proliferative glomerulonephritis
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN Electronic address
    Am J Kidney Dis 63:561-6. 2014
  3. doi request reprint Monoclonal gammopathy-associated proliferative glomerulonephritis
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN Electronic address
    Mayo Clin Proc 88:1284-93. 2013
  4. pmc Laser microdissection and proteomic analysis of amyloidosis, cryoglobulinemic GN, fibrillary GN, and immunotactoid glomerulopathy
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Clin J Am Soc Nephrol 8:915-21. 2013
  5. pmc Atypical postinfectious glomerulonephritis is associated with abnormalities in the alternative pathway of complement
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Kidney Int 83:293-9. 2013
  6. doi request reprint Renal amyloidosis associated with a novel sequence variant of gelsolin
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 61:161-6. 2013
  7. doi request reprint Secondary focal and segmental glomerulosclerosis associated with single-nucleotide polymorphisms in the genes encoding complement factor H and C3
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 60:316-21. 2012
  8. pmc Laser microdissection and mass spectrometry-based proteomics aids the diagnosis and typing of renal amyloidosis
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Kidney Int 82:226-34. 2012
  9. doi request reprint Medullary amyloidosis associated with apolipoprotein A-IV deposition
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Kidney Int 81:201-6. 2012
  10. doi request reprint Renal failure with intracapillary thrombi. Lipoprotein glomerulopathy
    S Sethi
    Laboratory Medicine and Pathology, Mayo Clinic, 200 1st Street SW, Rochester, New York 55905, USA
    Kidney Int 73:1097-8. 2008

Detail Information

Publications63

  1. ncbi request reprint C3 glomerulonephritis: clinicopathological findings, complement abnormalities, glomerular proteomic profile, treatment, and follow-up
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Kidney Int 82:465-73. 2012
    ..Thus, C3GN results from diverse abnormalities of the alternative complement pathway leading to subsequent glomerular injury...
  2. doi request reprint Etiology-based diagnostic approach to proliferative glomerulonephritis
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN Electronic address
    Am J Kidney Dis 63:561-6. 2014
    ..The second aim is to simplify and standardize the classification of proliferative GN. ..
  3. doi request reprint Monoclonal gammopathy-associated proliferative glomerulonephritis
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN Electronic address
    Mayo Clin Proc 88:1284-93. 2013
    ....
  4. pmc Laser microdissection and proteomic analysis of amyloidosis, cryoglobulinemic GN, fibrillary GN, and immunotactoid glomerulopathy
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Clin J Am Soc Nephrol 8:915-21. 2013
    ..Organized deposits are present in amyloidosis, fibrillary GN, and immunotactoid glomerulopathy. However, the constituents of the deposits are not known...
  5. pmc Atypical postinfectious glomerulonephritis is associated with abnormalities in the alternative pathway of complement
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Kidney Int 83:293-9. 2013
    ..Hence, the sequela is continual glomerular deposition of complement factors with resultant inflammation and development of an 'atypical' postinfectious glomerulonephritis...
  6. doi request reprint Renal amyloidosis associated with a novel sequence variant of gelsolin
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 61:161-6. 2013
    ..The novel gelsolin variant may be responsible for the unusual clinical and pathologic presentation. The report also highlights the usefulness of laser microdissection and mass spectrometry in the typing of difficult cases of amyloidosis...
  7. doi request reprint Secondary focal and segmental glomerulosclerosis associated with single-nucleotide polymorphisms in the genes encoding complement factor H and C3
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 60:316-21. 2012
    ..This case also expands the list of genetic polymorphisms that can be associated with an FSGS phenotype...
  8. pmc Laser microdissection and mass spectrometry-based proteomics aids the diagnosis and typing of renal amyloidosis
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Kidney Int 82:226-34. 2012
    ....
  9. doi request reprint Medullary amyloidosis associated with apolipoprotein A-IV deposition
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Kidney Int 81:201-6. 2012
    ..A high degree of suspicion is required for the diagnosis of apolipoprotein A-IV amyloidosis as it may be missed if a renal biopsy consists only of cortex...
  10. doi request reprint Renal failure with intracapillary thrombi. Lipoprotein glomerulopathy
    S Sethi
    Laboratory Medicine and Pathology, Mayo Clinic, 200 1st Street SW, Rochester, New York 55905, USA
    Kidney Int 73:1097-8. 2008
  11. pmc Glomeruli of Dense Deposit Disease contain components of the alternative and terminal complement pathway
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Kidney Int 75:952-60. 2009
    ..Our study shows that in addition to fluid-phase dysregulation of the alternative pathway, soluble components of the terminal complement complex contribute to glomerular lesions found in DDD...
  12. doi request reprint Dense deposit disease associated with monoclonal gammopathy of undetermined significance
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 56:977-82. 2010
    ..Thus, DDD in some older patients may be a distinct clinicopathologic entity that represents an uncommon complication of MGUS...
  13. pmc Mass spectrometry-based proteomic diagnosis of renal immunoglobulin heavy chain amyloidosis
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Clin J Am Soc Nephrol 5:2180-7. 2010
    ..We also describe the clinicopathologic findings of four problematic cases of renal Ig heavy chain amyloidosis that required LMD/MS proteomic analysis for accurate typing of the amyloid...
  14. pmc Proliferative glomerulonephritis secondary to dysfunction of the alternative pathway of complement
    Sanjeev Sethi
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Clin J Am Soc Nephrol 6:1009-17. 2011
    ..A recently described entity referred to as GN with C3 deposition (GN-C3) bears many similarities to DDD. The purpose of this study was to evaluate AP function in cases of GN-C3...
  15. doi request reprint AA amyloidosis associated with hepatitis B
    Abhijeet Saha
    Division of Pediatric Nephrology, PGIMER and Associated Dr RML Hospital, New Delhi, India
    Nephrol Dial Transplant 26:2407-12. 2011
    ..Furthermore, mass spectrometry also detected a variant of SAA protein (SAA W71R)...
  16. doi request reprint Membranoproliferative glomerulonephritis: pathogenetic heterogeneity and proposal for a new classification
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Semin Nephrol 31:341-8. 2011
    ..Initial AP screening tests should include serum membrane attack complex levels, an AP functional assay, and a hemolytic assay, followed by tests for mutations and autoantibodies to complement-regulating proteins...
  17. pmc Membranoproliferative glomerulonephritis secondary to monoclonal gammopathy
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, 200 1st Street SW, Rochester, MN 55905, USA
    Clin J Am Soc Nephrol 5:770-82. 2010
    ..Autoimmune diseases and chronic infections, such as hepatitis C, are commonly recognized causes of MPGN; however, monoclonal gammopathy is a less widely recognized cause of MPGN...
  18. ncbi request reprint Linear anti-glomerular basement membrane IgG but no glomerular disease: Goodpasture's syndrome restricted to the lung
    Sanjeev Sethi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, 200 1st Street SW, Rochester, MN 55905, USA
    Nephrol Dial Transplant 22:1233-5. 2007
  19. ncbi request reprint Renal thrombotic microangiopathy in a genetic model of hypertension in mice
    Sanjeev Sethi
    Department of Pathology, Mayo Clinic, 200 1st Street S W, Rochester, MN 55905, USA
    Exp Biol Med (Maywood) 231:196-203. 2006
    ..In summary, these studies characterize vascular and glomerular lesions in R+/A+ mice fed high salt, L-NAME, or both high salt and L-NAME, and provide a murine model of malignant hypertension with renal thrombotic microangiopathy...
  20. ncbi request reprint Acute renal failure in a renal allograft: an unusual infectious cause of thrombotic microangiopathy
    Sanjeev Sethi
    Department of Pathology, University of Iowa Hospitals and Clinics, Iowa City, IA, USA
    Am J Kidney Dis 46:159-62. 2005
  21. doi request reprint The pathology and clinical features of early recurrent membranous glomerulonephritis
    E F Rodriguez
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA
    Am J Transplant 12:1029-38. 2012
    ..A total of 11/16 patients showed progression of disease by EM on follow-up biopsy. Recognition of early allograft biopsy features aids in diagnosis of recurrent MGN before patients develop significant proteinuria...
  22. doi request reprint The histology of solitary renal allografts at 1 and 5 years after transplantation
    M D Stegall
    Von Liebig Transplant Center, Division of Transplantation Surgery, Division of Nephrology and Hypertension, Department of Anatomic Pathology, Mayo Clinic, Rochester, MN, USA
    Am J Transplant 11:698-707. 2011
    ..These results in the recent era of transplantation demonstrate fewer, less severe and less progressive chronic histologic changes in the first 5 years after transplantation than previously reported...
  23. ncbi request reprint Fibrillary glomerulonephritis: a report of 2 cases with extensive glomerular and tubular deposits
    O A Adeyi
    Department of Pathology, Brigham and Women's Hospital, Boston, MA, USA
    Hum Pathol 32:660-3. 2001
    ..These findings are used to support the view that fibrillary glomerulopathy is not a disease, but rather the morphologic expression of an etiologically diverse group of diseases as yet incompletely defined...
  24. doi request reprint Transplant glomerulopathy
    F G Cosio
    Division of Nephrology and Hypertension, Department of Internal Medicine, Mayo Clinic and Foundation, Rochester, MN, USA
    Am J Transplant 8:492-6. 2008
    ..However, it is also likely that an improved understanding of TG pathogenesis will result in the development of effective therapies for this form of progressive kidney allograft damage...
  25. ncbi request reprint A case of fibrillary glomerulonephritis with linear immunoglobulin G staining of the glomerular capillary walls
    S Sethi
    Department of Pathology, Harvard Medical School, Brigham and Women's Hospital, 75 Francis St, Boston, MA 02115, USA
    Arch Pathol Lab Med 125:534-6. 2001
    ..In such cases, electron microscopy is critical in differentiating the cause of crescentic glomerulonephritis...
  26. ncbi request reprint Mesangial immune complex glomerulonephritis due to complement factor D deficiency
    M A Abrera-Abeleda
    Department of Otolaryngology, University of Iowa, Iowa City, Iowa, USA
    Kidney Int 71:1142-7. 2007
    ..Our studies show that the complement factor D gene knockout mice are a novel model of spontaneous mesangial immune complex glomerulonephritis...
  27. ncbi request reprint Intra-tubular amyloidosis
    Z El-Zoghby
    Division of Nephrology, Department of Internal Medicine, Mayo Clinic, Rochester, MN 55905, USA
    Kidney Int 72:1282-8. 2007
  28. doi request reprint C1q deposition in the renal allograft: a report of 24 cases
    Samar M Said
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Mod Pathol 23:1080-8. 2010
    ..The rate of preceding infection and the prevalence of proteinuria seem to be similar to the renal transplant recipients in general. Most cases show mesangial hypercellularity or no glomerular changes on light microscopy...
  29. ncbi request reprint Transplant glomerulopathy: subclinical incidence and association with alloantibody
    J M Gloor
    Department of Internal Medicine, Division of Nephrology and Hypertension and Transplant Center, Mayo Clinic and Foundation, Rochester, MN, USA
    Am J Transplant 7:2124-32. 2007
    ..We suggest that subclinical TG is an under-recognized cause of antibody-mediated, chronic renal allograft injury which may be mechanistically distinct from other causes of nephropathy...
  30. pmc Variations in the complement regulatory genes factor H (CFH) and factor H related 5 (CFHR5) are associated with membranoproliferative glomerulonephritis type II (dense deposit disease)
    M A Abrera-Abeleda
    Department of Otolaryngology, Division of Nephrology, Carver College of Medicine, University of Iowa, Iowa City, IA, USA
    J Med Genet 43:582-9. 2006
    ....
  31. doi request reprint Recurrent membranoproliferative glomerulonephritis after kidney transplantation
    Elizabeth C Lorenz
    Division of Nephrology and Hypertension, Department of Internal Medicine, Mayo Clinic College of Medicine, Rochester, Minnesota, USA
    Kidney Int 77:721-8. 2010
    ..Our study shows the risk of MPGN recurrence and progression depends on identifiable pretransplant characteristics, has variable clinical impact, and can result in graft failure...
  32. doi request reprint Coexistence of myeloma cast nephropathy, light chain deposition disease, and nonamyloid fibrils in a patient with multiple myeloma
    Qi Qian
    Department of Medicine, Division of Nephrology and Hypertension, Mayo Clinic, Rochester, MN 55902, USA
    Am J Kidney Dis 56:971-6. 2010
    ..We show that laser microdissection and mass spectrometry is an extremely useful ancillary test for the diagnosis of heavy and light chain deposition diseases...
  33. ncbi request reprint Fc gamma RIII mediates neutrophil recruitment to immune complexes. a mechanism for neutrophil accumulation in immune-mediated inflammation
    A Coxon
    Department of Pathology, Brigham and Women s Hospital and, Harvard Medical School, 221 Longwood Avenue, 02115, Boston, MA, USA
    Immunity 14:693-704. 2001
    ..Thus, the interaction of immune complexes with Fc gamma RIII may mediate early neutrophil recruitment in immune complex-mediated inflammation...
  34. doi request reprint Idiopathic membranous nephropathy: diagnosis and treatment
    Fernando C Fervenza
    Division of Nephrology and Hypertension, Mayo Clinic College of Medicine, 200 First Street, SW, Rochester, MN 55905, USA
    Clin J Am Soc Nephrol 3:905-19. 2008
    ....
  35. doi request reprint Postinfectious glomerulonephritis in the elderly
    Samih H Nasr
    Mayo Clinic, Division of Anatomic Pathology, Hilton 10 20, 200 First Street, SW, Rochester, MN 55905, USA
    J Am Soc Nephrol 22:187-95. 2011
    ..Prognosis for these older patients is poor, with fewer than 25% recovering full renal function...
  36. doi request reprint Crystalline nephropathy due to 2,8-dihydroxyadeninuria: an under-recognized cause of irreversible renal failure
    Samih H Nasr
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA
    Nephrol Dial Transplant 25:1909-15. 2010
    ..Only rare cases of DHA nephrolithiasis have been reported from the USA. Herein, we report three American patients who developed DHA crystalline nephropathy leading to end-stage renal disease (ESRD) with recurrence in the allograft...
  37. pmc Fibrillary glomerulonephritis: a report of 66 cases from a single institution
    Samih H Nasr
    Mayo Clinic, Division of Anatomic Pathology, Hilton 10 20, 200 First Street SW, Rochester, MN 55905, USA
    Clin J Am Soc Nephrol 6:775-84. 2011
    ..Most previously reported cases were idiopathic. To better define the clinical-pathologic spectrum and prognosis, we report the largest single-center series with the longest follow-up...
  38. doi request reprint Kidney transplant histology after one year of continuous therapy with sirolimus compared with tacrolimus
    Patrick G Dean
    1 Department of Surgery, Mayo Clinic College of Medicine, Rochester, Minnesota 55905, USA
    Transplantation 85:1212-5. 2008
    ..This potential benefit is offset by lower success rate in maintaining the regimen and was not accompanied by superior glomerular filtration rate at 1 year...
  39. doi request reprint The rat femoral arteriovenous fistula model: increased expression of matrix metalloproteinase-2 and -9 at the venous stenosis
    Sanjay Misra
    Department of Radiology, Mayo Clinic College of Medicine, 200 First Street Southwest, Alfred 6460, Rochester, MN 55905, USA
    J Vasc Interv Radiol 19:587-94. 2008
    ....
  40. pmc Proliferative glomerulonephritis with monoclonal IgG deposits recurs in the allograft
    Samih H Nasr
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Clin J Am Soc Nephrol 6:122-32. 2011
    ..Proliferative GN with monoclonal IgG deposits (PGNMID) is a newly described entity resembling immune complex GN. Its potential to recur in the allograft is undefined...
  41. ncbi request reprint Nodular glomerulosclerosis: renal lesions in chronic smokers mimic chronic thrombotic microangiopathy and hypertensive lesions
    Kelly V Liang
    Department of Internal Medicine, Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 49:552-9. 2007
    ..These changes indicate endothelial injury and glomerular capillary wall remodeling; the lesions mimic those seen in patients with chronic hypertension and chronic or healed thrombotic microangiopathies...
  42. doi request reprint De novo AL amyloidosis in the kidney allograft
    Q Qian
    Department of Medicine, Division of Nephrology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Am J Transplant 11:606-12. 2011
    ..In conclusion, this study shows that AL amyloidosis is an uncommon but important cause of late onset proteinuria in the kidney allograft that results in kidney allograft failure...
  43. ncbi request reprint Renal vascular sclerosis is associated with inherited thrombophilias
    R L Goforth
    Department of Pathology, University of Iowa Hospitals and Clinics, Iowa, IA, USA
    Kidney Int 70:743-50. 2006
    ..001 compared to controls). These findings indicate that renal vascular lesions, in the absence of diabetes, hypertension, or smoking appears to be associated with inherited thrombophilias...
  44. doi request reprint Transplant glomerulopathy: risk and prognosis related to anti-human leukocyte antigen class II antibody levels
    Naim Issa
    Division of Nephrology and Hypertension and Transplant Center, Department of Internal Medicine, Mayo Clinic College of Medicine, Rochester, MN 55905, USA
    Transplantation 86:681-5. 2008
    ..The goal of this study was to determine the relationships among antibody characteristics (level and specificity), risk for TG, and graft survival...
  45. ncbi request reprint Disseminated histoplasmosis presenting as thrombotic microangiopathy
    Denis M Dwyre
    Department of Pathology, Roy J and Lucille A Carver College of Medicine, University of Iowa, Iowa City, Iowa 52242, USA
    Transfusion 46:1221-5. 2006
    ..Rapid and reliable diagnosis of TMA is critical. The diagnosis is complicated by a lack of objective and sensitive laboratory testing as well as multiple concurrent diseases, including infectious processes...
  46. ncbi request reprint New approaches to the treatment of dense deposit disease
    Richard J H Smith
    Department of Internal Medicine and Otolaryngology, University of Iowa Carver College of Medicine, Iowa City, Iowa, USA
    J Am Soc Nephrol 18:2447-56. 2007
    ..As the understanding of DDD increases, novel therapies should be integrated into existing protocols for DDD and evaluated using an open-label Bayesian study design...
  47. pmc Mycophenolate mofetil for induction and maintenance of remission in microscopic polyangiitis with mild to moderate renal involvement--a prospective, open-label pilot trial
    Francisco Silva
    Division of Nephrology and Hypertension, Mayo Clinic College of Medicine, 200 First Street, SW, Rochester, MN 55905, USA
    Clin J Am Soc Nephrol 5:445-53. 2010
    ..This prospective pilot trial explored whether mycophenolate mofetil (MMF) represents an effective alternative to CYC for induction and maintenance of remission in MPA with mild to moderate renal involvement...
  48. ncbi request reprint An unusual cause of acute renal failure
    M Lewin
    Mayo Clinic, Department of Laboratory Medicine and Pathology, Rochester, Minnesota 55905, USA
    Kidney Int 70:1534. 2006
  49. pmc Hemolysis and acute kidney failure
    Qi Qian
    Department of Medicine, Division of Nephrology and Hypertension, Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 56:780-4. 2010
    ..We conclude that tubular injury resulting from heme proteins should be considered as a cause of decreased kidney function in all patients with a cardiac valvular disease or prosthesis...
  50. ncbi request reprint Crystal-storing histiocytosis involving the kidney in a low-grade B-cell lymphoproliferative disorder
    Sanjeev Sethi
    Division of Renal Pathology and Electron Microscopy, Department of Pathology, Brigham and Women s Hospital, Harvard Medical School, Boston, MA 02115, USA
    Am J Kidney Dis 39:183-8. 2002
    ....
  51. ncbi request reprint ANCA-positive crescentic glomerulonephritis associated with minocycline therapy
    Sanjeev Sethi
    Department of Pathology, University of Iowa Hospitals and Clinics, Iowa City, IA 52242, USA
    Am J Kidney Dis 42:E27-31. 2003
    ..A high degree of suspicion, testing for ANCA titers, prompt discontinuation of the drug, and initiation of immunosuppressive treatment are crucial to the diagnosis and treatment of drug-induced ANCA-associated glomerulonephritis...
  52. ncbi request reprint Impairment of renal function after intravenous immunoglobulin
    Sandra M Soares
    Department of Pathology, Mayo Clinic and Foundation, Rochester, MN, USA
    Nephrol Dial Transplant 21:816-7. 2006
  53. ncbi request reprint Oxidized omega-3 fatty acids inhibit pro-inflammatory responses in glomerular endothelial cells
    Ashok Chaudhary
    Department of Pathology, University of Iowa of Iowa Hospitals and Clinics, Iowa City, Iowa 52242, USA
    Nephron Exp Nephrol 97:e136-45. 2004
    ..Accumulation of leukocytes occurs, in part, due to pro-inflammatory responses in endothelial cells, such as increase in expression of leukocyte adhesion receptors and chemokines, such as MCP-1 and IL-8...
  54. ncbi request reprint Cocaine, loin pain, and renal vein thrombosis
    Ziad Zoghby
    Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 49:859-61. 2007
    ..Cocaine abuse should be considered in the differential diagnosis of renal vein thrombosis in young adults...
  55. doi request reprint Marfan syndrome, MPGN, and bacterial endocarditis
    Peter Boseman
    Department of Internal Medicine, Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 51:697-701. 2008
  56. pmc Neutrophils: game changers in glomerulonephritis?
    Tanya N Mayadas
    Center for Excellence in Vascular Biology, Department of Pathology, Brigham and Women s Hospital and Harvard Medical School, Boston, MA 02115, USA
    Trends Mol Med 16:368-78. 2010
    ..Therefore, neutrophils, which are the first to arrive at a site of inflammation, are potential game changers in GN...
  57. ncbi request reprint Crescentic glomerulonephritis and dense deposit disease in a woman with breast carcinoma on immunosuppressive chemotherapy
    Sanjeev Sethi
    Department of Pathology, University of Iowa Hospitals and Clinics, Iowa City, IA 52242, USA
    Am J Kidney Dis 44:e33-7. 2004
    ....
  58. doi request reprint Sclerosing extramedullary hematopoietic tumor: emphasis on diagnosis by renal biopsy
    William R Sukov
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55902, USA
    Ann Diagn Pathol 13:127-31. 2009
    ....
  59. doi request reprint Successful pregnancy and delivery of a healthy newborn despite transplacental transfer of antimyeloperoxidase antibodies from a mother with microscopic polyangiitis
    Francisco Silva
    Division of Pulmonary and Critical Care Medicine, Mayo Clinic, Rochester, MN 55905, USA
    Am J Kidney Dis 54:542-5. 2009
    ..No clinical manifestation of vasculitis developed in the newborn. This case supports that anti-MPO antibodies alone are not pathogenic without additional cofactors...
  60. doi request reprint Renal failure due to combined cast nephropathy, amyloidosis and light-chain deposition disease
    Elizabeth C Lorenz
    Division of Nephrology and Hypertension, Mayo Clinic, Rochester, MN, USA
    Nephrol Dial Transplant 25:1340-3. 2010
    ..We report a case of renal failure in a patient with MM caused by concurrent CN, amyloidosis and LCDD which has not been previously described...
  61. ncbi request reprint Discovery of a spontaneous genetic mouse model of preeclampsia
    Robin L Davisson
    Department of Anatomy, University of Iowa College of Medicine, Iowa City 52242, USA
    Hypertension 39:337-42. 2002
    ..This is the first report of an animal model that spontaneously develops a syndrome that bears close resemblance to preeclampsia, and it should have an impact on our understanding of the pathophysiology of this disorder...
  62. ncbi request reprint Membranous glomerulonephritis with monoclonal immune deposits and crescents
    Sanjeev Sethi
    Nephrol Dial Transplant 20:1768-9. 2005
  63. ncbi request reprint Membranoproliferative glomerulonephritis type II (dense deposit disease): an update
    Gerald B Appel
    Columbia University, Department of Nephrology, New York, New York, USA
    J Am Soc Nephrol 16:1392-403. 2005
    ..The development of molecular diagnostic tools and new therapies directed at controlling the AP of the complement cascade either locally in the kidney or at the systemic level may lead to effective treatments for MPGN II...