Andre M Oliveira

Summary

Affiliation: Mayo Clinic
Country: USA

Publications

  1. doi request reprint Frequency of USP6 rearrangements in myositis ossificans, brown tumor, and cherubism: molecular cytogenetic evidence that a subset of "myositis ossificans-like lesions" are the early phases in the formation of soft-tissue aneurysmal bone cyst
    William R Sukov
    Department of Laboratory Medicine and Pathology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Skeletal Radiol 37:321-7. 2008
  2. pmc USP6 and CDH11 oncogenes identify the neoplastic cell in primary aneurysmal bone cysts and are absent in so-called secondary aneurysmal bone cysts
    Andre M Oliveira
    Department of Pathology, Brigham and Women s Hospital, 75 Francis Street, Boston, MA 02115, USA
    Am J Pathol 165:1773-80. 2004
  3. doi request reprint USP6-induced neoplasms: the biologic spectrum of aneurysmal bone cyst and nodular fasciitis
    Andre M Oliveira
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA Department of Orthopedics, Mayo Clinic, Rochester, MN, USA Electronic address
    Hum Pathol 45:1-11. 2014
  4. ncbi request reprint Aneurysmal bone cyst variant translocations upregulate USP6 transcription by promoter swapping with the ZNF9, COL1A1, TRAP150, and OMD genes
    Andre M Oliveira
    Department of Pathology, Brigham and Women s Hospital, Boston, MA 02115, USA
    Oncogene 24:3419-26. 2005
  5. doi request reprint Nodular fasciitis: a novel model of transient neoplasia induced by MYH9-USP6 gene fusion
    Michele R Erickson-Johnson
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Lab Invest 91:1427-33. 2011
  6. ncbi request reprint USP6 (Tre2) fusion oncogenes in aneurysmal bone cyst
    Andre M Oliveira
    Department of Pathology, Brigham and Women s Hospital, 75 Francis Street, Boston, MA 02115, USA
    Cancer Res 64:1920-3. 2004
  7. ncbi request reprint Cytogenetic and molecular cytogenetic findings in dedifferentiated liposarcoma with neural-like whorling pattern and metaplastic bone formation
    Ricardo S Macarenco
    Department of Laboratory Medicine and Pathology, Mayo Clinic, 200 First Street NW, Rochester, MN 55905, USA
    Cancer Genet Cytogenet 172:147-50. 2007
  8. doi request reprint Carboxypeptidase M: a biomarker for the discrimination of well-differentiated liposarcoma from lipoma
    Michele R Erickson-Johnson
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Mod Pathol 22:1541-7. 2009
  9. doi request reprint High-resolution genomic mapping reveals consistent amplification of the fibroblast growth factor receptor substrate 2 gene in well-differentiated and dedifferentiated liposarcoma
    Xiaoke Wang
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA
    Genes Chromosomes Cancer 50:849-58. 2011
  10. doi request reprint Successful treatment of a child with T/myeloid acute bilineal leukemia associated with TLX3/BCL11B fusion and 9q deletion
    Jennifer L Oliveira
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Pediatr Blood Cancer 56:467-9. 2011

Collaborators

Detail Information

Publications57

  1. doi request reprint Frequency of USP6 rearrangements in myositis ossificans, brown tumor, and cherubism: molecular cytogenetic evidence that a subset of "myositis ossificans-like lesions" are the early phases in the formation of soft-tissue aneurysmal bone cyst
    William R Sukov
    Department of Laboratory Medicine and Pathology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Skeletal Radiol 37:321-7. 2008
    ..Several lesions show histologic features that may overlap with ABC, including myositis ossificans (MO), brown tumor, and cherubism. The objective of this study was to assess whether these lesions harbored USP6 rearrangements...
  2. pmc USP6 and CDH11 oncogenes identify the neoplastic cell in primary aneurysmal bone cysts and are absent in so-called secondary aneurysmal bone cysts
    Andre M Oliveira
    Department of Pathology, Brigham and Women s Hospital, 75 Francis Street, Boston, MA 02115, USA
    Am J Pathol 165:1773-80. 2004
    ..By contrast, secondary ABC lack CDH11 and USP6 rearrangements, and although morphological mimics of primary ABC, appear to represent a non-specific morphological pattern of a diverse group of non-ABC neoplasms...
  3. doi request reprint USP6-induced neoplasms: the biologic spectrum of aneurysmal bone cyst and nodular fasciitis
    Andre M Oliveira
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA Department of Orthopedics, Mayo Clinic, Rochester, MN, USA Electronic address
    Hum Pathol 45:1-11. 2014
    ..We also discuss the possible line of differentiation of ABC and its relationship to nodular fasciitis and other lesions. ..
  4. ncbi request reprint Aneurysmal bone cyst variant translocations upregulate USP6 transcription by promoter swapping with the ZNF9, COL1A1, TRAP150, and OMD genes
    Andre M Oliveira
    Department of Pathology, Brigham and Women s Hospital, Boston, MA 02115, USA
    Oncogene 24:3419-26. 2005
    ..In summary, these studies show that USP6 oncogenic activation results from heterogeneous genomic mechanisms involving USP6 transcriptional upregulation by juxtaposition with ectopic promoters...
  5. doi request reprint Nodular fasciitis: a novel model of transient neoplasia induced by MYH9-USP6 gene fusion
    Michele R Erickson-Johnson
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Lab Invest 91:1427-33. 2011
    ..Considering the self-limited nature of the lesion, NF may represent a model of 'transient neoplasia', as it is, to our knowledge, the first example of a self-limited human disease characterized by a recurrent somatic gene fusion event...
  6. ncbi request reprint USP6 (Tre2) fusion oncogenes in aneurysmal bone cyst
    Andre M Oliveira
    Department of Pathology, Brigham and Women s Hospital, 75 Francis Street, Boston, MA 02115, USA
    Cancer Res 64:1920-3. 2004
    ..CDH11 is expressed strongly in bone, and our findings implicate a novel oncogenic mechanism in which deregulated USP6 transcription results from juxtaposition to the highly active CDH11 promoter...
  7. ncbi request reprint Cytogenetic and molecular cytogenetic findings in dedifferentiated liposarcoma with neural-like whorling pattern and metaplastic bone formation
    Ricardo S Macarenco
    Department of Laboratory Medicine and Pathology, Mayo Clinic, 200 First Street NW, Rochester, MN 55905, USA
    Cancer Genet Cytogenet 172:147-50. 2007
    ..The specific cytogenetics and molecular determinants of these peculiar morphologic findings, however, remain unknown...
  8. doi request reprint Carboxypeptidase M: a biomarker for the discrimination of well-differentiated liposarcoma from lipoma
    Michele R Erickson-Johnson
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Mod Pathol 22:1541-7. 2009
    ..These findings suggest that identification of CPM amplification could be used as an alternative diagnostic tool for the diagnosis of well-differentiated liposarcoma/atypical lipomatous tumors...
  9. doi request reprint High-resolution genomic mapping reveals consistent amplification of the fibroblast growth factor receptor substrate 2 gene in well-differentiated and dedifferentiated liposarcoma
    Xiaoke Wang
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA
    Genes Chromosomes Cancer 50:849-58. 2011
    ..Considering the critical role of FRS2 in mediating fibroblast growth factor receptor signaling, our findings indicate that FRS2 signaling should be further investigated as a potential therapeutic target for liposarcoma...
  10. doi request reprint Successful treatment of a child with T/myeloid acute bilineal leukemia associated with TLX3/BCL11B fusion and 9q deletion
    Jennifer L Oliveira
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Pediatr Blood Cancer 56:467-9. 2011
    ..Our patient has been in extended remission following ALL-based chemotherapy and a matched unrelated cord blood transplant. Inc...
  11. doi request reprint Endobronchial lipomatous tumors: clinicopathologic analysis of 12 cases with molecular cytogenetic evidence supporting classification as "lipoma"
    Jennifer M Boland
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester MN Department of Laboratory Medicine and Pathology, Mayo Clinic, Scottsdale, AZ
    Am J Surg Pathol 37:1715-21. 2013
    ..Ancillary fluorescence in situ hybridization testing may be very valuable in the analysis of these rare tumors, as true ALT/WDL seem to be very rare or nonexistent at this anatomic site...
  12. doi request reprint Fusion of HMGA2 to COG5 in uterine leiomyoma
    Gopalrao V N Velagaleti
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55906, USA
    Cancer Genet Cytogenet 202:11-6. 2010
    ..Since the general structure of the fusion gene is similar to other previously described HMGA2 fusions, its biologic activity is predicted to be likely similar...
  13. doi request reprint HMGA1 and HMGA2 rearrangements in mass-forming endometriosis
    Fabiola Medeiros
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Genes Chromosomes Cancer 49:630-4. 2010
    ..The present findings support the concept that endometriosis is clonal and that rearrangement of HMGA genes likely contributes to its pathogenesis...
  14. ncbi request reprint Frequency and characterization of HMGA2 and HMGA1 rearrangements in mesenchymal tumors of the lower genital tract
    Fabiola Medeiros
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Genes Chromosomes Cancer 46:981-90. 2007
    ..The diagnostic usefulness of HMGA2 rearrangements to differentiate between AAM and other tumors of the lower genital tract may be limited due to the their low frequency...
  15. doi request reprint HMGA2 expression analysis in cytological and paraffin-embedded tissue specimens of thyroid tumors by relative quantitative RT-PCR
    Long Jin
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA
    Diagn Mol Pathol 20:71-80. 2011
    ..The 1-step qRT-PCR method is a sensitive, accurate, and reliable technique for gene expression analysis of thyroid tumors...
  16. doi request reprint Identification of novel HMGA2 fusion sequences in lipoma: evidence that deletion of let-7 miRNA consensus binding site 1 in the HMGA2 3' UTR is not critical for HMGA2 transcriptional upregulation
    Xiaoke Wang
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Genes Chromosomes Cancer 48:673-8. 2009
    ....
  17. doi request reprint Correlation of IHC and FISH for ALK gene rearrangement in non-small cell lung carcinoma: IHC score algorithm for FISH
    Eunhee S Yi
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota, USA
    J Thorac Oncol 6:459-65. 2011
    ..Fluorescent in situ hybridization (FISH) is used to detect ALK+, but it is expensive and not routinely available. We explored the potential of an immunohistochemistry (IHC) scoring system as an affordable, accessible approach...
  18. ncbi request reprint Gains of COL1A1-PDGFB genomic copies occur in fibrosarcomatous transformation of dermatofibrosarcoma protuberans
    Jared J Abbott
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Mod Pathol 19:1512-8. 2006
    ..Copy number of COL1A1-PDGFB fusion gene in the classic dermatofibrosarcoma protuberans areas does not seem to be a major predisposing mechanism for fibrosarcomatous transformation...
  19. doi request reprint HMGA gene rearrangement is a recurrent somatic alteration in polypoid endometriosis
    Fabiola Medeiros
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Hum Pathol 43:1243-8. 2012
    ..These findings suggest that HMGA rearrangements likely contribute to the pathogenesis of endometriosis. However, additional studies are needed to better define the biologic role of this genetic alteration...
  20. doi request reprint Anaplastic lymphoma kinase immunoreactivity correlates with ALK gene rearrangement and transcriptional up-regulation in non-small cell lung carcinomas
    Jennifer M Boland
    Department of Laboratory Medicine and Pathology, Mayo Medical School, Rochester, MN 55905, USA
    Hum Pathol 40:1152-8. 2009
    ..Anaplastic lymphoma kinase immunohistochemistry may have utility as a screening tool or as a surrogate marker for the molecular techniques to detect the EML4-ALK fusion gene in these tumors...
  21. doi request reprint Worse disease-free survival in never-smokers with ALK+ lung adenocarcinoma
    Ping Yang
    Division of Epidemiology, Health Sciences Research, Mayo Clinic, Rochester, Minnesota 55905, USA
    J Thorac Oncol 7:90-7. 2012
    ....
  22. doi request reprint Diagnostic utility of IMP3 expression in thyroid neoplasms: a quantitative RT-PCR study
    Long Jin
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Diagn Mol Pathol 19:63-9. 2010
    ..These results indicate that detection of IMP3 mRNA expression levels by qRT-PCR may be a useful molecular marker to assist in the diagnosis of well-differentiated thyroid carcinomas...
  23. doi request reprint MET and EGFR mutations identified in ALK-rearranged pulmonary adenocarcinoma: molecular analysis of 25 ALK-positive cases
    Jennifer M Boland
    Departments of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    J Thorac Oncol 8:574-81. 2013
    ..However, the mutation status of other genes is not widely known in ALK+ tumors. The aim of this study is to survey for mutations involving other genes in 25 ALK+ cases confirmed by fluorescent in situ hybridization...
  24. doi request reprint Fusion of HMGA1 to the LPP/TPRG1 intergenic region in a lipoma identified by mapping paraffin-embedded tissues
    Xiaoke Wang
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Cancer Genet Cytogenet 196:64-7. 2010
    ..No rearrangement of HMGA2 was found. The biological function of this novel fusion could be similar to the role of HMGA2-LPP in tumorigenesis...
  25. doi request reprint FOXL2 mutations in granulosa cell tumors occurring in males
    Joema F Lima
    Division of Anatomic Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota, USA
    Arch Pathol Lab Med 136:825-8. 2012
    ..Recently, a somatic mutation of FOXL2 was reported in virtually all adult-type granulosa cell tumors in women...
  26. doi request reprint Fusion of dynactin 1 to anaplastic lymphoma kinase in inflammatory myofibroblastic tumor
    Xiaoke Wang
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Hum Pathol 43:2047-52. 2012
    ..This novel fusion gene is structurally similar to other previously described anaplastic lymphoma kinase fusion genes and may be associated with the unusual morphology and immunophenotype of this tumor...
  27. ncbi request reprint Cytogenetic and molecular cytogenetic findings of intimal sarcoma
    Hongying Zhang
    Division of Laboratory Medicine and Pathology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Cancer Genet Cytogenet 179:146-9. 2007
    ..Further studies are needed to determine whether the cytogenetic abnormalities found in this case are recurrent events for this poorly characterized malignancy...
  28. pmc Synovial sarcoma of the sellar region
    Bernd W Scheithauer
    Department of Laboratory Medicine and Pathology, Mayo Clinic, 200 First St, SW, Rochester, MN 55905, USA
    Neuro Oncol 9:454-9. 2007
    ..Subtotal resection and radiosurgery proved to be efficacious. The spectrum of primary sellar region sarcomas is summarized...
  29. ncbi request reprint Clonal cytogenetic abnormalities in Erdheim-Chester disease
    Eneida F Vencio
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 31:319-21. 2007
    ..However, additional studies are warranted to confirm whether the chromosomal abnormalities found in this case represent recurrent cytogenetic events...
  30. doi request reprint Ependymal tumors with sarcomatous change ("ependymosarcoma"): a clinicopathologic and molecular cytogenetic study
    Fausto J Rodriguez
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 32:699-709. 2008
    ..Although rare, ependymal neoplasms must be included among the gliomas prone to undergo sarcomatous change and we propose the term "ependymosarcoma" for these tumors...
  31. doi request reprint KIT is an independent prognostic marker for pancreatic endocrine tumors: a finding derived from analysis of islet cell differentiation markers
    Lizhi Zhang
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA
    Am J Surg Pathol 33:1562-9. 2009
    ..These results indicate that KIT is a new and independent prognostic marker for PETs. The classification system derived from KIT and CK19 was able to predict clinical behavior of PET...
  32. doi request reprint Genome-wide transcriptome analyses reveal p53 inactivation mediated loss of miR-34a expression in malignant peripheral nerve sheath tumours
    Subbaya Subramanian
    Department of Laboratory Medicine and Pathology, University of Minnesota, Minneapolis, Minnesota, USA
    J Pathol 220:58-70. 2010
    ..Collectively, our findings suggest that deregulation of miRNAs has a potential role in the malignant transformation process in peripheral nerve sheath tumours...
  33. doi request reprint Liposarcomas with mixed well-differentiated and pleomorphic features: a clinicopathologic study of 12 cases
    Jennifer M Boland
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 34:837-43. 2010
    ..The presence of MDM2/CPM amplification in the PL component of mixed WDL/PL suggests that a subset of PL may arise through tumor progression of WDL or may represent a "transitional" or partially differentiated step toward classic DL...
  34. doi request reprint Low-grade sinonasal sarcoma with neural and myogenic features: a clinicopathologic analysis of 28 cases
    Jason T Lewis
    Department of Laboratory Medicine, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 36:517-25. 2012
    ..On the basis of the locally aggressive nature of this lesion we believe it is best considered a low-grade sarcoma and suggest the term low-grade sinonasal sarcoma with neural and myogenic features...
  35. ncbi request reprint Cytogenetic and molecular genetic findings in dedifferentiated liposarcoma with neural-like whorling pattern and metaplastic bone formation
    Ricardo S Macarenco
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Cancer Genet Cytogenet 171:126-9. 2006
    ..However, the specific cytogenetic and molecular determinants of these peculiar morphologic findings remain unknown...
  36. doi request reprint Are meningeal hemangiopericytoma and mesenchymal chondrosarcoma the same?: a study of HEY1-NCOA2 fusion
    Karen J Fritchie
    Mayo Clinic, Dept of Anatomic Pathology, Hilton 11, 200 First St, SW, Rochester, MN 55905
    Am J Clin Pathol 140:670-4. 2013
    ..Recently, a novel HEY1-NCOA2 fusion was reported as a recurrent event in mesenchymal chondrosarcomas...
  37. ncbi request reprint KIT-negative gastrointestinal stromal tumors: proof of concept and therapeutic implications
    Fabiola Medeiros
    Department of Pathology, Brigham and Women s Hospital and Harvard Medical School, Boston, MA 02115, USA
    Am J Surg Pathol 28:889-94. 2004
    ..Notably, some KIT-negative GISTs contain imatinib-sensitive KIT or PDGFRA mutations; therefore, patients with KIT-negative GISTs should not, a priori, be denied imatinib therapy...
  38. ncbi request reprint Formation of the 12q14-q15 amplicon precedes the development of a well-differentiated liposarcoma arising from a nonchondroid pulmonary hamartoma
    Sylvain Trahan
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 30:1326-9. 2006
    ....
  39. ncbi request reprint The TRE17/USP6 oncogene: a riddle wrapped in a mystery inside an enigma
    Andre M Oliveira
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA
    Front Biosci (Schol Ed) 4:321-34. 2012
    ..In this review, we discuss the most up-to-date findings on the molecular functions of TRE17, the role of its USP and TBC domains, and potential models for how it contributes to transformation and ABC pathogenesis...
  40. doi request reprint A cytogenetic analysis of 2 cases of phosphaturic mesenchymal tumor of mixed connective tissue type
    Rondell P Graham
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Hum Pathol 43:1334-8. 2012
    ..2;p14),add(4)(q31.1)[10]/46,sdl,add(13)(q34)[4]/92,sdl2x2[1]. These represent what is, to our knowledge, the first examples of abnormal karyotypes obtained from phosphaturic mesenchymal tumor of mixed connective tissue type...
  41. doi request reprint Molecular cytogenetic analysis for TFE3 rearrangement in Xp11.2 renal cell carcinoma and alveolar soft part sarcoma: validation and clinical experience with 75 cases
    Jennelle C Hodge
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA
    Mod Pathol 27:113-27. 2014
    ..Our TFE3 FISH assay is a useful adjunct to morphologic analysis of such challenging cases and will be applicable to assess the growing spectrum of TFE3-rearranged tumors. ..
  42. ncbi request reprint Tumor suppressor genes in breast cancer: the gatekeepers and the caretakers
    Andre M Oliveira
    Department of Laboratory Medicine and Pathology, Mayo Clinic and Mayo Foundation, Rochester, MN, USA
    Am J Clin Pathol 124:S16-28. 2005
    ..Other tumor suppressor genes of interest in breast cancer include the retinoblastoma gene (pRb), PTEN, p16, nm23, and maspin...
  43. ncbi request reprint Primary synovial sarcoma of the mitral valve
    Dylan V Miller
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Cardiovasc Pathol 14:331-3. 2005
    ..The histopathologic diagnosis was supported by the immunohistochemical profile and the presence of the SYT-SSX1 fusion transcript identified by molecular genetic studies...
  44. ncbi request reprint Angiomyxofibromatous tumor of the falx cerebri
    Fabiola Medeiros
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 30:545-7. 2006
    ....
  45. ncbi request reprint The prognostic significance of fibrosarcomatous transformation in dermatofibrosarcoma protuberans
    Jared J Abbott
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 30:436-43. 2006
    ..We demonstrate that FS change in DFSP is a form of tumor progression that carries an increased risk of metastasis over classic DFSP and is associated with gains of p53 mutations and increased proliferative activity...
  46. ncbi request reprint Pleomorphic liposarcoma: clinicopathologic analysis of 57 cases
    Jason L Hornick
    Department of Pathology, Brigham and Women s Hospital and Harvard Medical School, 75 Francis Street, Boston, MA 02115, USA
    Am J Surg Pathol 28:1257-67. 2004
    ..By multivariate analysis, wide local excision or amputation and postoperative radiotherapy protected against local recurrence...
  47. doi request reprint Molecular genetics of soft-tissue sarcomas: a brief overview for clinical oncologists
    Jorge Torres-Mora
    From the Department of Laboratory Medicine and Pathology, Division of Medical Oncology, and Department of Orthopedics, Mayo Clinic, Rochester, MN
    Cancer J 20:73-9. 2014
    ..Herein, we provide a brief overview of clinically relevant molecular abnormalities of STSs for the general clinical oncologist. ..
  48. doi request reprint Rare ALK expression but no ALK rearrangement in pancreatic ductal adenocarcinoma and neuroendocrine tumors
    Rondell P D Graham
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Pancreas 42:949-51. 2013
    ..We performed ALK immunohistochemistry on samples of pancreatic ductal adenocarcinoma and pancreatic neuroendocrine tumors using 2 tissue microarrays...
  49. ncbi request reprint Successful treatment of a child with T/myeloid acute bilineal leukemia associated with TLX3/BCL11B fusion and 9q deletion
    Jennifer L Oliveira
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota
    Pediatr Blood Cancer 56:467-9. 2011
    ..Our patient has been in extended remission following ALL-based chemotherapy and a matched unrelated cord blood transplant. Pediatr Blood Cancer 2011;56:467-469. © 2010 Wiley-Liss, Inc...
  50. ncbi request reprint Applications of fluorescence in situ hybridization in cytopathology: a review
    Andre M Oliveira
    Department of Pathology, Brigham and Women s Hospital, Harvard Medical School, Boston, Massachusetts 02115, USA
    Acta Cytol 49:587-94. 2005
    ..Furthermore, due to its feasibility for virtually all types of cytologic specimens, a new and exciting era in both investigative and clinical cytopathology is expected...
  51. ncbi request reprint Aneurysmal bone cyst: a neoplasm driven by upregulation of the USP6 oncogene
    Andre M Oliveira
    J Clin Oncol 24:e1; author reply e2. 2006
  52. ncbi request reprint Genomic gains of COL1A1-PDFGB occur in the histologic evolution of giant cell fibroblastoma into dermatofibrosarcoma protuberans
    Ricardo S Macarenco
    CIPAX, Medicina Diagnostica, Sao Jose dos Campos SP, Brazil
    Genes Chromosomes Cancer 47:260-5. 2008
    ....
  53. ncbi request reprint Myxoinflammatory fibroblastic sarcoma showing t(2;6)(q31;p21.3) as a sole cytogenetic abnormality
    Cristiane M Ida
    Division of Anatomic Pathology, Hospital das Clinicas C M I, Sao Paulo University School of Medicine, Sao Paulo, SP, Brazil
    Cancer Genet Cytogenet 177:139-42. 2007
    ..3) as the only cytogenetic abnormality. This finding is distinct from the two cases previously reported. Additional studies are needed to verify whether any of these chromosome rearrangements are involved recurrently in MIFS...
  54. ncbi request reprint Molecular prognostication for soft tissue sarcomas: are we ready yet?
    Andre M Oliveira
    J Clin Oncol 22:4031-4. 2004
  55. ncbi request reprint Primary sarcomas of the brain and spinal cord: a study of 18 cases
    Andre M Oliveira
    Division of Anatomic Pathology, Mayo Clinic, Rochester, Minnesota 55905, U S A
    Am J Surg Pathol 26:1056-63. 2002
    ..Most involve the cerebrum and show fibrous, "fibrohistiocytic," or no specific differentiation. The prognosis for high-grade sarcomas seems better than that for glioblastoma multiforme...
  56. ncbi request reprint Sustained complete remission of metastatic dermatofibrosarcoma protuberans with imatinib mesylate
    Stefanos V Labropoulos
    First Department of Medical Oncology, Hygeia Hospital, Marousi, Greece
    Anticancer Drugs 16:461-6. 2005
    ..We conclude that sustained complete remission of metastatic DFSP with specific FISH abnormalities involving the PDGFB locus can be obtained with imatinib mesylate with minimal toxicity for the patient...
  57. doi request reprint HMGA2 rearrangements are rare in benign lipomatous lesions of the nervous system
    Fausto J Rodriguez
    Acta Neuropathol 116:337-8. 2008