Eduardo Benarroch

Summary

Affiliation: Mayo Clinic
Country: USA

Publications

  1. ncbi request reprint Thermoregulation: recent concepts and remaining questions
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 69:1293-7. 2007
  2. ncbi request reprint Rho GTPases: role in dendrite and axonal growth, mental retardation, and axonal regeneration
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 68:1315-8. 2007
  3. doi request reprint Brain cholesterol metabolism and neurologic disease
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, Rochester, MN 55905, USA
    Neurology 71:1368-73. 2008
  4. doi request reprint Parabrachial nucleus involvement in multiple system atrophy
    E E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA Electronic address
    Auton Neurosci 177:170-4. 2013
  5. doi request reprint Neocortical interneurons: functional diversity and clinical correlations
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 81:273-80. 2013
  6. doi request reprint Pedunculopontine nucleus: functional organization and clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 80:1148-55. 2013
  7. doi request reprint HCN channels: function and clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 80:304-10. 2013
  8. doi request reprint Insulin-like growth factors in the brain and their potential clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 79:2148-53. 2012
  9. pmc Postural tachycardia syndrome: a heterogeneous and multifactorial disorder
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Mayo Clin Proc 87:1214-25. 2012
  10. doi request reprint Adenosine and its receptors: multiple modulatory functions and potential therapeutic targets for neurologic disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 70:231-6. 2008

Detail Information

Publications67

  1. ncbi request reprint Thermoregulation: recent concepts and remaining questions
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 69:1293-7. 2007
  2. ncbi request reprint Rho GTPases: role in dendrite and axonal growth, mental retardation, and axonal regeneration
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 68:1315-8. 2007
  3. doi request reprint Brain cholesterol metabolism and neurologic disease
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, Rochester, MN 55905, USA
    Neurology 71:1368-73. 2008
  4. doi request reprint Parabrachial nucleus involvement in multiple system atrophy
    E E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA Electronic address
    Auton Neurosci 177:170-4. 2013
    ..Neurons of the parabrachial nucleus (PBN) control respiratory rhythmogenesis and airway resistance...
  5. doi request reprint Neocortical interneurons: functional diversity and clinical correlations
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 81:273-80. 2013
    ..There are many comprehensive reviews on GABAergic interneuron heterogeneity, development, plasticity, function in shaping cortical activity, and involvement in disease.(1-13.) ..
  6. doi request reprint Pedunculopontine nucleus: functional organization and clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 80:1148-55. 2013
    ..There have been several recent reviews on the PPN focused on its neurochemical organization and connectivity, physiology, involvement in parkinsonian syndromes, and as a target for DBS.(1-12.)..
  7. doi request reprint HCN channels: function and clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, USA
    Neurology 80:304-10. 2013
    ..Experimental evidence indicates that HCN channels may also contribute to mechanisms of epilepsy and pain. The physiologic functions of HCN channels and their implications for neurologic disorders have been recently reviewed.(1-10)...
  8. doi request reprint Insulin-like growth factors in the brain and their potential clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 79:2148-53. 2012
    ..The insulin-like peptides, their receptors, effects in the nervous system, and potential clinical correlations have been the subject of several recent reviews.(1-6)...
  9. pmc Postural tachycardia syndrome: a heterogeneous and multifactorial disorder
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Mayo Clin Proc 87:1214-25. 2012
    ..Studies were limited to those published in English. Other articles were identified from bibliographies of the retrieved articles...
  10. doi request reprint Adenosine and its receptors: multiple modulatory functions and potential therapeutic targets for neurologic disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 70:231-6. 2008
  11. ncbi request reprint Aquaporin-4, homeostasis, and neurologic disease
    Eduardo E Benarroch
    Mayo Clinic, 200 First Street SW, West 8A Mayo Bldg or 811 Guggenheim Bldg, Rochester, MN 55905, USA
    Neurology 69:2266-8. 2007
  12. ncbi request reprint Enteric nervous system: functional organization and neurologic implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 69:1953-7. 2007
  13. ncbi request reprint Lipid rafts, protein scaffolds, and neurologic disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, West 8A Mayo Bldg or 811 Guggenheim Bldg, Rochester, MN 55905, USA
    Neurology 69:1635-9. 2007
  14. ncbi request reprint Tissue plasminogen activator: beyond thrombolysis
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 69:799-802. 2007
  15. ncbi request reprint Rostral raphe involvement in Lewy body dementia and multiple system atrophy
    E E Benarroch
    Department of Neurology, Mayo Clinic, 811 Guggenheim Building, 200 First Street SW, Rochester, MN 55905, USA
    Acta Neuropathol 114:213-20. 2007
    ..Although loss of rostral raphe neurons may contribute to depression in DLB, this appears to be less likely in MSA. Factors other than the neurochemical phenotype determine neuronal vulnerability in MSA...
  16. ncbi request reprint Endocannabinoids in basal ganglia circuits: implications for Parkinson disease
    Eduardo Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 69:306-9. 2007
  17. ncbi request reprint Brainstem respiratory chemosensitivity: new insights and clinical implications
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 68:2140-3. 2007
  18. doi request reprint TRP channels: functions and involvement in neurologic disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 70:648-52. 2008
  19. pmc Loss of A5 noradrenergic neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Acta Neuropathol 115:629-34. 2008
    ..The severity of cell loss was comparable to that of locus ceruleus neurons in these cases. Our results indicate that A5 neurons are severely affected in MSA and this may contribute to some of the manifestations of this synucleinopathy...
  20. pmc Differential involvement of the periaqueductal gray in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN, United States
    Auton Neurosci 158:111-7. 2010
    ..There was accumulation of glial cytoplasmic inclusions in all PAG columns in MSA. Our findings indicate involvement of the PAG columns in MSA, which may contribute to autonomic disturbances in this disorder...
  21. pmc Dopamine cell loss in the periaqueductal gray in multiple system atrophy and Lewy body dementia
    E E Benarroch
    Mayo Clinic, Department of Neurology, 811 Guggenheim Bldg, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 73:106-12. 2009
    ..Excessive daytime sleepiness (EDS) is a common manifestation of multiple system atrophy (MSA) and dementia with Lewy bodies (DLB) but involvement of these neurons has not yet been explored...
  22. doi request reprint The midline and intralaminar thalamic nuclei: anatomic and functional specificity and implications in neurologic disease
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 71:944-9. 2008
  23. doi request reprint Suprachiasmatic nucleus and melatonin: reciprocal interactions and clinical correlations
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 71:594-8. 2008
  24. doi request reprint Descending monoaminergic pain modulation: bidirectional control and clinical relevance
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 71:217-21. 2008
  25. doi request reprint Subthalamic nucleus and its connections: Anatomic substrate for the network effects of deep brain stimulation
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 70:1991-5. 2008
  26. doi request reprint N-acetylaspartate and N-acetylaspartylglutamate: neurobiology and clinical significance
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 70:1353-7. 2008
  27. doi request reprint Metabotropic glutamate receptors: synaptic modulators and therapeutic targets for neurologic disease
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, 200 First Street SW, West 8A Mayo Bldg, Rochester, MN 55905, USA
    Neurology 70:964-8. 2008
  28. ncbi request reprint Depletion of putative chemosensitive respiratory neurons in the ventral medullary surface in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Rochester, MN, USA
    Brain 130:469-75. 2007
    ..This may provide a possible morphological basis for impaired respiratory chemosensitivity and central sleep apnoea in this disorder...
  29. ncbi request reprint Dysautonomia in fatal familial insomnia as an indicator of the potential role of the thalamus in autonomic control
    E E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Brain Pathol 8:527-30. 1998
    ..The medial portion of the MD may share with the PVT a relay function for circuits controlling autonomic responses. MD involvement in FFI suggests a role of the thalamus in central autonomic and other integrative functions...
  30. ncbi request reprint Paraventricular nucleus, stress response, and cardiovascular disease
    Eduardo E Benarroch
    Mayo Clinic, Dept of Neurology, 811 Guggenheim Building, 200 First Street SW, Rochester, MN 55905, USA
    Clin Auton Res 15:254-63. 2005
    ..The neurochemical control of the different subgroups of PVN neurons is complex. The PVN has been implicated in the pathophysiology of congestive heart failure and the metabolic syndrome...
  31. ncbi request reprint Differential involvement of hypothalamic vasopressin neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Brain 129:2688-96. 2006
    ..Loss of AVP neurons in the SCN may contribute to impaired circadian regulation of endocrine and autonomic functions...
  32. ncbi request reprint Pain-autonomic interactions
    E E Benarroch
    Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurol Sci 27:S130-3. 2006
    ..This review will focus on some central pain-autonomic interactions potentially relevant for the pathophysiology of primary headache...
  33. ncbi request reprint Involvement of medullary regions controlling sympathetic output in Lewy body disease
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Brain 128:338-44. 2005
    ..In LBD, orthostatic hypotension may be due primarily to involvement of sympathetic ganglion neurons rather than ventrolateral medulla neurons...
  34. ncbi request reprint Involvement of medullary serotonergic groups in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 811 Guggenheim Building, 200 First Street SW, Rochester, MN 55905, USA
    Ann Neurol 55:418-22. 2004
    ..Depletion of serotonergic neurons may contribute to impaired control of sympathetic outflow and other abnormalities in MSA...
  35. ncbi request reprint Brainstem in multiple system atrophy: clinicopathological correlations
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, Minnesota 55905, USA
    Cell Mol Neurobiol 23:519-26. 2003
    ..6. Loss of cholinergic mesopontine neurons, in the setting of loss of locus ceruleus neurons and preservation of rostral raphe neurons, may contribute to REM sleep abnormalities in MSA...
  36. ncbi request reprint Involvement of vagal autonomic nuclei in multiple system atrophy and Lewy body disease
    E E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 66:378-83. 2006
    ..The relationship between DMV and NAmb involvement and the gastrointestinal or cardiovagal manifestations in MSA and LBD is unclear...
  37. ncbi request reprint Preservation of branchimotor neurons of the nucleus ambiguus in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 60:115-7. 2003
    ..The number of neurons in the dorsal NA was not significantly different among the three groups. Neurons in the ventrolateral portion of the NA were depleted in MSA. Laryngeal stridor cannot be explained solely by neuronal loss in the NA...
  38. ncbi request reprint Involvement of hypocretin neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Acta Neuropathol 113:75-80. 2007
    ..This is the first demonstration of loss of Hcrt/Orx neurons in MSA, which is consistent with a system degeneration of neurons involved in homeostatic function, including sleep and autonomic regulation, in this disorder...
  39. ncbi request reprint Depletion of mesopontine cholinergic and sparing of raphe neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 59:944-6. 2002
    ..001) and laterodorsal tegmental nucleus (18 +/- 3 vs 47 +/- 4 cells/section, p < 0.001) in MSA. Whereas there was also depletion of locus ceruleus neurons, there was a striking preservation of rostral raphe neurons in MSA...
  40. ncbi request reprint Brainstem respiratory control: substrates of respiratory failure of multiple system atrophy
    Eduardo E Benarroch
    Mayo Clinic, Department of Neurology, Rochester, Minnesota 55905, USA
    Mov Disord 22:155-61. 2007
    ..This finding may provide potential anatomical substrates for the respiratory manifestations of the disease...
  41. ncbi request reprint New findings on the neuropathology of multiple system atrophy
    Eduardo E Benarroch
    Neurophysiological Laboratory, Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Auton Neurosci 96:59-62. 2002
    ....
  42. ncbi request reprint Sodium channels and pain
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Neurology 68:233-6. 2007
  43. ncbi request reprint Pain-autonomic interactions: a selective review
    E E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Clin Auton Res 11:343-9. 2001
    ..These interactions should be taken into account to understand the complex pathophysiology of chronic pain disorders...
  44. ncbi request reprint Depletion of corticotrophin-releasing factor neurons in the pontine micturition area in multiple system atrophy
    E E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Ann Neurol 50:640-5. 2001
    ..7 +/- 4 neurons/section in controls). Our results suggest that depletion of CRF neurons in the putative pontine micturition center may contribute to the severe bladder dysfunction that characterizes multiple system atrophy...
  45. ncbi request reprint Depletion of cholinergic neurons of the medullary arcuate nucleus in multiple system atrophy
    E E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Auton Neurosci 87:293-9. 2001
    ..alpha-Synuclein-containing inclusions were present in the ArcN of MSA patients. Depletion of cholinergic neurons may provide a substrate for disturbances in automatic respiration in MSA patients...
  46. ncbi request reprint Immunohistochemical localization of the angiotensin II type 1 receptor in human hypothalamus and brainstem
    E E Benarroch
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Brain Res 812:292-6. 1998
    ..This provides direct evidence of neuronal localization of the AT-1 receptor in autonomic and motor areas in human brain...
  47. ncbi request reprint Neuron-astrocyte interactions: partnership for normal function and disease in the central nervous system
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic College of Medicine, 200 First St SW, Rochester, MN 55905, USA
    Mayo Clin Proc 80:1326-38. 2005
    ..Disturbances of these neuron-astrocyte interactions are likely to play an important role in neurologic disorders including cerebral ischemia, neurodegeneration, migraine, cerebral edema, and hepatic encephalopathy...
  48. ncbi request reprint Depletion of ventromedullary NK-1 receptor-immunoreactive neurons in multiple system atrophy
    Eduardo E Benarroch
    Department of Neurology, Mayo Foundation, Rochester, MN 55904, USA
    Brain 126:2183-90. 2003
    ....
  49. ncbi request reprint Glutamic acid decarboxylase autoimmunity with brainstem, extrapyramidal, and spinal cord dysfunction
    Sean J Pittock
    Department of Neurology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA
    Mayo Clin Proc 81:1207-14. 2006
    ..To describe novel neurological manifestations associated with glutamic acid decarboxylase (GAD65) autoimmunity...
  50. ncbi request reprint Neurosteroids: endogenous modulators of neuronal excitability and plasticity
    Eduardo E Benarroch
    Mayo Clinic, Rochester, MN 55905, USA
    Neurology 68:945-7. 2007
  51. ncbi request reprint Dynamic phases of peroneal and tibial intraneural ganglia formation: a new dimension added to the unifying articular theory
    Robert J Spinner
    Department of Neurologic Surgery, Mayo Clinic, Rochester, Minnesota 55905, USA
    J Neurosurg 107:296-307. 2007
    ..Atypical features, which occasionally are observed, have offered an opportunity to verify further and expand on the authors' proposed theory...
  52. ncbi request reprint EEG changes in a patient with steroid-responsive encephalopathy associated with antibodies to thyroperoxidase (SREAT, Hashimoto's encephalopathy)
    Alcibiades J Rodriguez
    Mayo Clinic Department of Neurology, Rochester, Minnesota, USA
    J Clin Neurophysiol 23:371-3. 2006
    ..These patients often show an excellent clinical improvement to immunosuppressive therapy, including corticosteroids. This case report documents the dramatic clinical and EEG improvement with steroid therapy...
  53. ncbi request reprint GABAA receptor heterogeneity, function, and implications for epilepsy
    Eduardo E Benarroch
    Department of Neurology, Mayo Clinic, Rochester, MN 55905, USA
    Neurology 68:612-4. 2007
  54. ncbi request reprint Clinical cues for detecting ictal asystole
    Gena R Ghearing
    Dept of Neurology, Mayo Clinic Rochester, Rochester, MN 55905, USA
    Clin Auton Res 17:221-6. 2007
    ..Recognition of ictal asystole is important as treatment with both antiepileptic drugs and cardiac pacing may be necessary. The purpose of this study was to identify clinical cues to aid in the detection of ictal asystole...
  55. ncbi request reprint Pattern-specific loss of aquaporin-4 immunoreactivity distinguishes neuromyelitis optica from multiple sclerosis
    Shanu F Roemer
    Department of Neurology, Mayo Clinic, College of Medicine, 200 First St SW, Rochester, MN 55905, USA
    Brain 130:1194-205. 2007
    ..These findings strongly support a role for a complement activating AQP4-specific autoantibody as the initiator of the NMO lesion, and further distinguish NMO from MS...
  56. ncbi request reprint The ictal bradycardia syndrome: localization and lateralization
    Jeffrey W Britton
    Divisions of Epilepsy and Clinical Neurophysiology EEG, Department of Neurology, Mayo Clinic Rochester, Rochester, Minnesota 55905, USA
    Epilepsia 47:737-44. 2006
    ..However, the evidence for this is inconsistent. We sought to assess critically the predictable value of ictal bradycardia for seizure localization and lateralization...
  57. pmc Serologic profiles aiding the diagnosis of autoimmune gastrointestinal dysmotility
    Radhika Dhamija
    Department of Immunology, Mayo Clinic College of Medicine, Rochester, Minnesota 55905, USA
    Clin Gastroenterol Hepatol 6:988-92. 2008
    ..Here we report 24 Mayo Clinic patients in whom the profile of serum autoantibodies aided this diagnosis...
  58. ncbi request reprint Amphiphysin autoimmunity: paraneoplastic accompaniments
    Sean J Pittock
    Department of Neurology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA
    Ann Neurol 58:96-107. 2005
    ..01). Overall, a minority of women (39%) and men (12%) had stiff-man phenomena. Only 10% of women (some with lung carcinoma) and 4% of men fulfilled diagnostic criteria for stiff-man syndrome...
  59. ncbi request reprint Seizures and syncope: anatomic basis and diagnostic considerations
    Jeffrey W Britton
    Sections of Epilepsy and Clinical Neurophysiology EEG, Dept of Neurology, West 8 Mayo Building, Mayo Clinic Rochester, 200 First Street SW, Rochester, MN 55905, USA
    Clin Auton Res 16:18-28. 2006
    ..This article reviews the use of EEG/ECG telemetry in the evaluation of syncope and seizures, and the neuroanatomic circuitry involved in the production of the cardiovascular manifestations of seizures...
  60. pmc Ventilatory and cardiovascular responses to hypercapnia and hypoxia in multiple-system atrophy
    Axel Lipp
    Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Arch Neurol 67:211-6. 2010
    ..The cardiovascular responses to chemoreflex activation in MSA have not been explored to date...
  61. pmc Focal atrophy in dementia with Lewy bodies on MRI: a distinct pattern from Alzheimer's disease
    Jennifer L Whitwell
    Department of Radiology, Mayo Clinic, Rochester, MN 55905, USA
    Brain 130:708-19. 2007
    ..Damage to this network of structures in DLB may affect a number of different neurotransmitter systems which in turn may contribute to a number of the core clinical features of DLB...
  62. ncbi request reprint Neurodegeneration involving putative respiratory neurons in Perry syndrome
    Yoshio Tsuboi
    Department of Neurology, Fukuoka University, Fukuoka, Japan
    Acta Neuropathol 115:263-8. 2008
    ....
  63. ncbi request reprint Neurovascular unit dysfunction: a vascular component of Alzheimer disease?
    Eduardo E Benarroch
    Neurology 68:1730-2. 2007
  64. ncbi request reprint Dopamine beta-hydroxylase deficiency involves the central autonomic network
    William P Cheshire
    Acta Neuropathol 112:227-9. 2006
  65. doi request reprint Neural control of the gastrointestinal tract: implications for Parkinson disease
    Maria G Cersosimo
    Parkinson s Disease and Movement Disorder Unit, Hospital de Clinicas, University of Buenos Aires, Argentina
    Mov Disord 23:1065-75. 2008
    ..This early involvement provides insights into the pathophysiology of gastrointestinal dysmotility in this disorder and may constitute an important step in the etiopathogenesis of Lewy body disease...
  66. ncbi request reprint Quantitative study of salivary secretion in Parkinson's disease
    Omar R Tumilasci
    Departamento de Fisiologia, Laboratorio de Neurofisiologia, Facultad de Medicina, and Programa de Parkinson y Movimientos Anormales, Instituto de Neurociencias del Hospital de Clínicas, Universidad de Buenos Aires, Buenos Aires, Argentina
    Mov Disord 21:660-7. 2006
    ..Domperidone may have a peripheral effect that potentiates reflex salivary secretion. Salivary composition is abnormal in PD and is not affected by levodopa treatment...
  67. ncbi request reprint Autonomic and cognitive dysfunction in Parkinson's disease
    Juan Idiaquez
    Universidad de Valparaiso, Valparaiso, Chile
    Clin Auton Res 17:93-8. 2007
    ....