Susan C Abraham

Summary

Affiliation: Mayo Clinic
Country: USA

Publications

  1. ncbi Achlorhydria, parietal cell hyperplasia, and multiple gastric carcinoids: a new disorder
    Susan C Abraham
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 29:969-75. 2005
  2. ncbi Diffuse lymphoplasmacytic chronic cholecystitis is highly specific for extrahepatic biliary tract disease but does not distinguish between primary and secondary sclerosing cholangiopathy
    Susan C Abraham
    Department of Pathology, Hilton 11 Mayo Clinic, 220 First Street SW, Rochester, MN, USA
    Am J Surg Pathol 27:1313-20. 2003
  3. ncbi Lymphoplasmacytic chronic cholecystitis and biliary tract disease in patients with lymphoplasmacytic sclerosing pancreatitis
    Susan C Abraham
    Department of Pathology, Hilton 11, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Am J Surg Pathol 27:441-51. 2003
  4. ncbi Eosinophilic pancreatitis and increased eosinophils in the pancreas
    Susan C Abraham
    Department of Pathology, Hilton 11, Mayo Clinic, 220 First Street SW, Rochester, MN 55905, USA
    Am J Surg Pathol 27:334-42. 2003
  5. ncbi Pancreaticoduodenectomy (Whipple resections) in patients without malignancy: are they all 'chronic pancreatitis'?
    Susan C Abraham
    Department of Pathology, Mayo Clinic, Rochester, Minnesota, 55905, USA
    Am J Surg Pathol 27:110-20. 2003
  6. ncbi Microsatellite instability in intraductal papillary neoplasms of the biliary tract
    Susan C Abraham
    Division of Gastrointestinal Liver Pathology, Department of Pathology, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Mod Pathol 15:1309-17. 2002
  7. ncbi Genetic alterations in gastric adenomas of intestinal and foveolar phenotypes
    Susan C Abraham
    Department of Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Mod Pathol 16:786-95. 2003
  8. ncbi "Seedling" mesenchymal tumors (gastrointestinal stromal tumors and leiomyomas) are common incidental tumors of the esophagogastric junction
    Susan C Abraham
    Division of Anatomic Pathology, Department of Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 31:1629-35. 2007
  9. ncbi Distinguishing gastrointestinal stromal tumors from their mimics: an update
    Susan C Abraham
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Adv Anat Pathol 14:178-88. 2007
  10. ncbi Liver transplantation in precirrhotic biliary tract disease: Portal hypertension is frequently associated with nodular regenerative hyperplasia and obliterative portal venopathy
    Susan C Abraham
    Department of Pathology and Gastroenterology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 30:1454-61. 2006

Detail Information

Publications74

  1. ncbi Achlorhydria, parietal cell hyperplasia, and multiple gastric carcinoids: a new disorder
    Susan C Abraham
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 29:969-75. 2005
    ....
  2. ncbi Diffuse lymphoplasmacytic chronic cholecystitis is highly specific for extrahepatic biliary tract disease but does not distinguish between primary and secondary sclerosing cholangiopathy
    Susan C Abraham
    Department of Pathology, Hilton 11 Mayo Clinic, 220 First Street SW, Rochester, MN, USA
    Am J Surg Pathol 27:1313-20. 2003
    ..These results suggest that a pattern of diffuse lymphoplasmacytic chronic cholecystitis is highly specific for extrahepatic biliary tract disease but does not distinguish between primary and secondary cholangiopathies...
  3. ncbi Lymphoplasmacytic chronic cholecystitis and biliary tract disease in patients with lymphoplasmacytic sclerosing pancreatitis
    Susan C Abraham
    Department of Pathology, Hilton 11, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Am J Surg Pathol 27:441-51. 2003
    ....
  4. ncbi Eosinophilic pancreatitis and increased eosinophils in the pancreas
    Susan C Abraham
    Department of Pathology, Hilton 11, Mayo Clinic, 220 First Street SW, Rochester, MN 55905, USA
    Am J Surg Pathol 27:334-42. 2003
    ..True eosinophilic pancreatitis, although a fascinating clinicopathologic entity, is one of the rarest causes of pancreatic eosinophilia...
  5. ncbi Pancreaticoduodenectomy (Whipple resections) in patients without malignancy: are they all 'chronic pancreatitis'?
    Susan C Abraham
    Department of Pathology, Mayo Clinic, Rochester, Minnesota, 55905, USA
    Am J Surg Pathol 27:110-20. 2003
    ..These findings indicate that "benign but clinically suspicious" Whipple resections are relatively common in high-volume centers (9.2%) and reveal a diverse group of clinicopathologically distinctive pancreatic and biliary tract disease...
  6. ncbi Microsatellite instability in intraductal papillary neoplasms of the biliary tract
    Susan C Abraham
    Division of Gastrointestinal Liver Pathology, Department of Pathology, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Mod Pathol 15:1309-17. 2002
    ....
  7. ncbi Genetic alterations in gastric adenomas of intestinal and foveolar phenotypes
    Susan C Abraham
    Department of Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Mod Pathol 16:786-95. 2003
    ..Although intestinal-type and foveolar-type gastric adenomas display divergent biologic behavior, the specific genetic events accounting for these differences in morphology and biologic behavior are unclear...
  8. ncbi "Seedling" mesenchymal tumors (gastrointestinal stromal tumors and leiomyomas) are common incidental tumors of the esophagogastric junction
    Susan C Abraham
    Division of Anatomic Pathology, Department of Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 31:1629-35. 2007
    ..The common occurrence of microscopic GISTs compared with the rarity of clinically manifest and malignant esophagogastric GISTs suggests that additional genetic or epigenetic alterations must happen for neoplastic progression...
  9. ncbi Distinguishing gastrointestinal stromal tumors from their mimics: an update
    Susan C Abraham
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Adv Anat Pathol 14:178-88. 2007
    ..It is important to separate GISTs from these potential mimics because their treatment and prognosis can differ markedly...
  10. ncbi Liver transplantation in precirrhotic biliary tract disease: Portal hypertension is frequently associated with nodular regenerative hyperplasia and obliterative portal venopathy
    Susan C Abraham
    Department of Pathology and Gastroenterology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 30:1454-61. 2006
    ..The occurrence of NRH in some patients transplanted for other indications suggests it is a histologic pattern that can precede the development of clinically significant portal hypertension...
  11. ncbi Chronic antral ulcer associated with gastroduodenal lymphocytic phlebitis
    Susan C Abraham
    Department of Pathology and Laboratory Medicine, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 28:1659-63. 2004
    ..We hypothesize that there may be more cases of upper gastrointestinal ELP than are diagnosed as such, in part because the diagnosis can be made only on surgical resections specimens...
  12. ncbi Frequent CpG island methylation in precursor lesions and early gastric adenocarcinomas
    Jae Hyuk Lee
    Department of Pathology, MD Anderson Cancer Center, Houston, TX 77030, USA
    Oncogene 23:4646-54. 2004
    ..Methylation of the p16 gene may contribute to the malignant transformation of gastric precursor lesions...
  13. doi Duplicated muscularis mucosae invasion has similar risk of lymph node metastasis and recurrence-free survival as intramucosal esophageal adenocarcinoma
    Jeannelyn S Estrella
    Department of Pathology, The University of Texas MD Anderson Cancer Center, Houston, USA
    Am J Surg Pathol 35:1045-53. 2011
    ..EAC invasion into the space between duplicated MM confers a similar risk of lymph node metastasis and recurrence as those of intramucosal EAC, and LVI is the best predictor of lymph node status and RFS in pT1 EAC...
  14. ncbi Molecular and immunohistochemical analysis of intraductal papillary neoplasms of the biliary tract
    Susan C Abraham
    Department of Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Hum Pathol 34:902-10. 2003
    ..Finally, the frequency of allelic loss on 18q suggests that a locus on 18q is involved in the molecular pathogenesis of biliary IPNs, but this locus is not DPC4...
  15. ncbi Hepatic adenomas: analysis of sex steroid receptor status and the Wnt signaling pathway
    Michael Torbenson
    Division of Gastrointestinal Liver Pathology, Department of Pathology, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21205 2196, USA
    Mod Pathol 15:189-96. 2002
    ..Activation of the Wnt signaling pathway appears to be important in a subset of hepatic adenomas but does not result from common beta-catenin or APC mutations and does not appear to be directly linked to hormonal receptor status...
  16. doi Significance of central perivenulitis in pediatric liver transplantation
    Susan C Abraham
    Department of Pathology, M D Anderson Cancer Center, Houston, TX, USA
    Am J Surg Pathol 32:1479-88. 2008
    ..CP is most common in conjunction with portal ACR, where it carries a significant risk for the development of zone 3 fibrosis and a trend toward the development of ductopenic chronic rejection...
  17. ncbi Duplication of the muscularis mucosae in Barrett esophagus: an underrecognized feature and its implication for staging of adenocarcinoma
    Susan C Abraham
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN, USA
    Am J Surg Pathol 31:1719-25. 2007
    ..The 17% rate of angiolymphatic invasion and 10% rate of lymph node metastases in our patients with invasion into duplicated MM suggest that these tumors can behave aggressively despite their technically intramucosal location...
  18. ncbi Frequent CpG island methylation in sporadic and syndromic gastric fundic gland polyps
    Susan C Abraham
    Department of Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Clin Pathol 122:740-6. 2004
    ..87). Epigenetic alterations involving methylation of CpG islands might have a role in the development of some FGPs, particularly those with a PPI effect. They do not account for the presence or absence of a dysplastic phenotype in FGPs...
  19. pmc Solid-pseudopapillary tumors of the pancreas are genetically distinct from pancreatic ductal adenocarcinomas and almost always harbor beta-catenin mutations
    Susan C Abraham
    Department of Pathology, Division of Gastrointestinal Liver Pathology, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21205 2196, USA
    Am J Pathol 160:1361-9. 2002
    ..Overexpression of p53 was limited to only 3 of 19 (15.8%) SPTs. These results emphasize the two distinct, divergent genetic pathways of neoplastic progression in pancreatic ductal and nonductal neoplasms...
  20. doi Muscularis mucosae duplication and the musculo-fibrous anomaly in endoscopic mucosal resections for barrett esophagus: implications for staging of adenocarcinoma
    Jason T Lewis
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 32:566-71. 2008
    ....
  21. pmc Results of pancreaticoduodenectomy for lymphoplasmacytic sclerosing pancreatitis
    Jeffrey M Hardacre
    Department of Surgery, The Johns Hopkins Hospital, 600 N Wolfe Street, Baltimore, MD 21287 4688, USA
    Ann Surg 237:853-8; discussion 858-9. 2003
    ....
  22. pmc Sporadic fundic gland polyps with epithelial dysplasia : evidence for preferential targeting for mutations in the adenomatous polyposis coli gene
    Susan C Abraham
    Department of Pathology, Division of Gastrointestinal Liver Pathology, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Am J Pathol 161:1735-42. 2002
    ..The lack of microsatellite instability in FGPs in this study and of K-ras mutations in a previous study suggests that secondary genetic alterations are rare in both dysplastic and nondysplastic FGPs...
  23. pmc Genetic and immunohistochemical analysis of pancreatic acinar cell carcinoma: frequent allelic loss on chromosome 11p and alterations in the APC/beta-catenin pathway
    Susan C Abraham
    Department of Pathology, Division of GI LiverPathology, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21205 2196, USA
    Am J Pathol 160:953-62. 2002
    ..These results indicate that ACCs are genetically distinct from pancreatic ductal adenocarcinomas, but some cases contain genetic alterations common to histologically similar pancreatoblastomas...
  24. doi Histologic abnormalities are common in protocol liver allograft biopsies from patients with normal liver function tests
    Susan C Abraham
    Department of Pathology, M D Anderson Cancer Center, Houston, TX, USA
    Am J Surg Pathol 32:965-73. 2008
    ....
  25. ncbi Prevalence and risk factors for gallbladder neoplasia in patients with primary sclerosing cholangitis: evidence for a metaplasia-dysplasia-carcinoma sequence
    Jason T Lewis
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 31:907-13. 2007
    ..The strong correlation between gallbladder dysplasia/adenocarcinoma and bile duct dysplasia/CC supports the concept of a neoplastic "field effect" along the intrahepatic and extrahepatic biliary tract in PSC...
  26. doi Precancerous bile duct pathology in end-stage primary sclerosing cholangitis, with and without cholangiocarcinoma
    Jason T Lewis
    Division of Anatomic Pathology, Department of Pathology, DeKalb Medical Center, Decatur, GA 30033, USA
    Am J Surg Pathol 34:27-34. 2010
    ..Dysplasia, however, is generally confined to large and septal-size bile ducts and its presence may not be recognized unless multiple sections specifically targeted to the biliary tree are examined...
  27. ncbi Sporadic fundic gland polyposis: a clinical, histological, and molecular analysis
    Michael Torbenson
    Department of Pathology, The Johns Hopkins Hospital, Baltimore, Maryland, USA
    Mod Pathol 15:718-23. 2002
    ..The fundic gland polyps were histologically and genetically identical to single sporadic fundic gland polyps and demonstrated frequent somatic activating mutations in exon 3 of the beta-catenin gene...
  28. ncbi High-level microsatellite instability in appendiceal carcinomas
    Melissa W Taggart
    Department of Pathology, University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA
    Am J Surg Pathol 37:1192-200. 2013
    ....
  29. pmc Inverse relationship between APC gene mutation in gastric adenomas and development of adenocarcinoma
    Jae Hyuk Lee
    Department of Pathology, MD Anderson Cancer Center, Houston, Texas 77030, USA
    Am J Pathol 161:611-8. 2002
    ..Gastric adenomas or dysplasias without APC mutations but with or without MSI may have a different biological behavior, and are precursors of intestinal-type of gastric adenocarcinomas...
  30. doi Paget cells in the esophagus: assessment of their histopathologic features and near-universal association with underlying esophageal adenocarcinoma
    Susan C Abraham
    Department of Pathology, M D Anderson Cancer Center, Houston, TX, USA
    Am J Surg Pathol 32:1068-74. 2008
    ..A commonality among cases with Paget cells is the presence of focal or diffuse, poorly differentiated adenocarcinoma with discohesive cells. E-cadherin alterations seem to play a less significant role...
  31. ncbi Excellent interobserver agreement on grading the extent of residual carcinoma after preoperative chemoradiation in esophageal and esophagogastric junction carcinoma: a reliable predictor for patient outcome
    Tsung Teh Wu
    Department of Pathology, University of Texas M D Anderson Cancer Center, 1515 Holcombe Boulevard, Houston, TX 77030, USA
    Am J Surg Pathol 31:58-64. 2007
    ..The extent of residual carcinoma is a reliable and reproducible predictor of survival; this grading system may allow a novel and early means of comparing outcomes after different neoadjuvant treatment regimens...
  32. ncbi Patchy distribution of pathologic abnormalities in autoimmune pancreatitis: implications for preoperative diagnosis
    Vishal S Chandan
    Department of Pathology, Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 32:1762-9. 2008
    ..IgG4 immunostaining of apparently negative biopsies may be helpful, but only in a small minority of cases...
  33. doi Biliary intraepithelial neoplasia in patients without chronic biliary disease: analysis of liver explants with alcoholic cirrhosis, hepatitis C infection, and noncirrhotic liver diseases
    Tsung Teh Wu
    Division of Anatomic Pathology, Mayo Clinic, Rochester, Minnesota, USA
    Cancer 115:4564-75. 2009
    ....
  34. doi The prevalence and natural history of untreated isolated central perivenulitis in adult allograft livers
    Alyssa M Krasinskas
    Department of Pathology, University of Pittsburgh, Pittsburgh, PA 15213 2546, USA
    Liver Transpl 14:625-32. 2008
    ..In summary, "transplant-associated" isolated CP occurs in 28% of adult patients, early CP is predictive of late CP, and late CP (often present as isolated CP) is associated with long-term liver injury in some patients...
  35. ncbi Fibromatosis of the breast and mutations involving the APC/beta-catenin pathway
    Susan C Abraham
    Department of Pathology, The Johns Hopkins University School of Medicine, Baltimore, MD 21205 2196, USA
    Hum Pathol 33:39-46. 2002
    ..The spectrum of beta-catenin and APC alterations is similar to that described for desmoid tumors of the abdomen, paraspinal region, and extremities...
  36. pmc Adult autoimmune enteropathy: Mayo Clinic Rochester experience
    Salma Akram
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, Mayo Clinic, Rochester, Minnesota, USA
    Clin Gastroenterol Hepatol 5:1282-90; quiz 1245. 2007
    ..Autoimmune enteropathy is a rare cause of intractable diarrhea associated with circulating gut autoantibodies and a predisposition to autoimmunity. It is rarely observed in adults, with only 11 cases reported to date...
  37. doi Pyloric gland adenoma: an entity distinct from gastric foveolar type adenoma
    Zong Ming Chen
    Department of Pathology, Johns Hopkins University, Baltimore, MD 21231 2410, USA
    Am J Surg Pathol 33:186-93. 2009
    ..Despite its bland histologic appearance, it is much more likely to be accompanied by background IM and autoimmune gastritis and can evolve into invasive adenocarcinoma displaying pyloric gland differentiation...
  38. ncbi Analysis of novel tumor markers in pancreatic and biliary carcinomas using tissue microarrays
    Sharon L Swierczynski
    Department of Pathology, The Johns Hopkinds University School of Medicine, Baltimore, MD 21231, USA
    Hum Pathol 35:357-66. 2004
    ..Therefore, these markers are potentially useful in developing diagnostic tests and treatment paradigms for tumors involving the biliary system...
  39. ncbi Colchicine effect in a colonic hyperplastic polyp. A lesion mimicking serrated adenoma
    Michael Torbenson
    Division of Gastrointestinal Liver Pathology, Department of Pathology, The Johns Hopkins University School of Medicine, Baltimore, MD 21205 2196, USA
    Arch Pathol Lab Med 126:615-7. 2002
    ..Distinguishing between hyperplastic polyp and serrated adenoma is important because of the different management implications and the increased potential for neoplastic progression in the latter...
  40. ncbi Tumor suppressor gene hypermethylation as a predictor of gastric stromal tumor behavior
    Michael G House
    Department of Surgery, The Johns Hopkins Medical Institutions, Baltimore, MD 21231 1000, USA
    J Gastrointest Surg 7:1004-14; discussion 1014. 2003
    ..This study supports the methylation status of E-cadherin as a prognostic marker for early GIST recurrence and survival...
  41. doi IgG4+ plasma cell infiltrates in liver explants with primary sclerosing cholangitis
    Lizhi Zhang
    Department of Laboratory Medicine, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 34:88-94. 2010
    ..PSC with tissue IgG4 positivity has a more aggressive clinical course manifested by shorter time to transplant and a higher likelihood of recurrence than IgG4 negative PSC...
  42. ncbi CD44 loss in gastric stromal tumors as a prognostic marker
    Elizabeth Montgomery
    Johns Hopkins Medical Institutions, Baltimore, MD, USA
    Am J Surg Pathol 28:168-77. 2004
    ..Thus, the prognostic value of CD44s and isoform expression in GIST were evaluated by immunohistochemistry of tissue microarrays...
  43. ncbi Bile duct dysplasia in the setting of chronic hepatitis C and alcohol cirrhosis
    Michael Torbenson
    Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, MD 21231, USA
    Am J Surg Pathol 31:1410-3. 2007
    ..Alcohol also seems to be a risk factor. The dysplastic changes are multifocal, involve septal sized bile ducts, and are typically papillary...
  44. doi Mucosal colonization by metastatic carcinoma in the gastrointestinal tract: a potential mimic of primary neoplasia
    Jeannelyn S Estrella
    Department of Pathology, The University of Texas M D Anderson Cancer Center, Houston, TX, USA
    Am J Surg Pathol 35:563-72. 2011
    ..In situ growth and presence of an apparent adenoma cannot be taken as prima facie evidence of a primary neoplasm...
  45. ncbi Epidermal growth factor receptor, p53 mutation, and pathological response predict survival in patients with locally advanced esophageal cancer treated with preoperative chemoradiotherapy
    Michael K Gibson
    Division of Medical Oncology, The Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins, Baltimore, Maryland 21231, USA
    Clin Cancer Res 9:6461-8. 2003
    ..This study evaluates the role of clinical and cellular markers in predicting CR and OS in patients with esophageal cancer...
  46. doi Expression of melanoma antigens in epithelioid gastrointestinal stromal tumors: a potential diagnostic pitfall
    Mehmet L Guler
    Department of Pathology, Johns Hopkins University, Baltimore, MD 21231, USA
    Arch Pathol Lab Med 132:1302-6. 2008
    ..Most gastric gastrointestinal stromal tumors (GISTs) express CD117/c-kit, as do a subset of metastatic melanomas, leading to a diagnostic dilemma in some cases...
  47. doi Metastatic neuroendocrine tumour in the breast: a potential mimic of in-situ and invasive mammary carcinoma
    Kyle D Perry
    Division of Anatomic Pathology, Department of Pathology, Mayo Clinic, Rochester, MN, USA
    Histopathology 59:619-30. 2011
    ....
  48. doi Observer variability in the histologic diagnosis of microscopic colitis
    David Limsui
    Division of Gastroenterology and Hepatology, Miles and Shirley Fiterman Center for Digestive Diseases, Mayo Clinic, Rochester, Minnesota, USA
    Inflamm Bowel Dis 15:35-8. 2009
    ..There has been no investigation of the reproducibility of the histologic diagnosis of microscopic colitis. Our aim was to evaluate interobserver and intraobserver variation in this diagnosis...
  49. ncbi Atrophic autoimmune pangastritis: A distinctive form of antral and fundic gastritis associated with systemic autoimmune disease
    Dragan Jevremovic
    Department of Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 30:1412-9. 2006
    ..The development of multifocal low-grade dysplasia in 1 patient, a 19-year-old woman, suggests that this condition might have neoplastic potential...
  50. doi Small intestinal bacterial overgrowth: histopathologic features and clinical correlates in an underrecognized entity
    Paul J Lappinga
    Departments of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Arch Pathol Lab Med 134:264-70. 2010
    ..Small intestinal bacterial overgrowth (SIBO) is a common cause of chronic diarrhea and malabsorption. Morphologic changes associated with this condition have not, to our knowledge, been studied in detail...
  51. ncbi Oxyntic mucosa pseudopolyps: a presentation of atrophic autoimmune gastritis
    Alyssa M Krasinskas
    Department of Pathology, University of Pennsylvania Medical Center, Philadelphia, USA
    Am J Surg Pathol 27:236-41. 2003
    ..Although a limited number of previous studies have suggested this type of polypoid presentation may represent "early" atrophic gastritis, its persistence in three of our patients argues against this hypothesis...
  52. ncbi Beta-catenin immunohistochemistry separates mesenteric fibromatosis from gastrointestinal stromal tumor and sclerosing mesenteritis
    Elizabeth Montgomery
    Department of Pathology, Johns Hopkins School of Medicine, Baltimore, Maryland 21205, USA
    Am J Surg Pathol 26:1296-301. 2002
    ..beta-Catenin staining identifies lesions that are, instead, mesenteric fibromatoses...
  53. ncbi An assessment of chromosomal alterations detected by fluorescence in situ hybridization and p16 expression in sporadic and primary sclerosing cholangitis-associated cholangiocarcinomas
    Ryan D DeHaan
    Department of Laboratory Medicine and Pathology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA
    Hum Pathol 38:491-9. 2007
    ..Polysomy and homozygous 9p21 deletion are common in both sporadic and PSC-associated CCAs and are frequently detectable in PSC-associated biliary dysplasia...
  54. ncbi Crystal-storing histiocytosis due to massive accumulation of charcot-leyden crystals: a unique association producing colonic polyposis in a 78-year-old woman with eosinophilic colitis
    Jason T Lewis
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Am J Surg Pathol 31:481-5. 2007
    ..This is the first reported case of crystal-storing histiocytosis produced by massive accumulation of Charcot-Leyden crystals in eosinophilic colitis...
  55. ncbi Surgical and chemotherapy treatment outcomes of goblet cell carcinoid: a tertiary cancer center experience
    Tuan H Pham
    Department of Colorectal Surgery, Mayo Clinic, 200 First Street S W, Rochester, Minnesota 55905, USA
    Ann Surg Oncol 13:370-6. 2006
    ..Goblet cell carcinoid (GCC) is a rare malignant tumor with distinct histological and clinical features. Our goals were to review the surgical and chemotherapy outcomes of patients with GCC...
  56. ncbi Hematological malignancy manifesting as ascites
    Konstantinos N Lazaridis
    Division of Gastroenterology and Hepatology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA
    Nat Clin Pract Gastroenterol Hepatol 2:112-6; quiz 117. 2005
    ..Splenectomy was recommended but the patient declined. She was lost to follow-up twice but returned with tense ascites about 2 years after the initial presentation. Despite aggressive medical management, the ascites did not improve...
  57. doi Oesophageal hyperkeratosis: clinicopathological associations
    Melissa W Taggart
    Department of Pathology, University of Texas M D Anderson Cancer Center, Houston, TX, USA
    Histopathology 63:463-73. 2013
    ..In contrast to hyperkeratosis of orolaryngeal mucosa, where its risk factors and association with squamous neoplasia are well-studied, the prevalence and clinicopathological features of oesophageal hyperkeratosis are unknown...
  58. ncbi Remission of severe autoimmune enteropathy after treatment with high-dose cyclophosphamide
    Maria M Oliva-Hemker
    Department of Pediatrics, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21187 2631, USA
    J Pediatr Gastroenterol Nutr 36:639-43. 2003
  59. ncbi Autoimmune gastritis: distinct histological and immunohistochemical findings before complete loss of oxyntic glands
    Michael Torbenson
    Division of Gastrointestinal Liver Pathology, Department of Pathology, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Mod Pathol 15:102-9. 2002
    ....
  60. doi Rosai-Dorfman disease confined to the breast
    Jerzy J Morkowski
    Department of Pathology, The University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA
    Ann Diagn Pathol 14:81-7. 2010
    ....
  61. ncbi The effect of the metabolic syndrome, hepatic steatosis and steatohepatitis on liver fibrosis in hereditary hemochromatosis
    Leon A Adams
    Department of Gastroenterology and Hepatology, Mayo Clinic College of Medicine, Rochester, MI, USA
    Liver Int 26:298-304. 2006
    ..We sought to examine whether the metabolic syndrome, hepatic steatosis or steatohepatitis influenced hepatic fibrosis among patients with HH and iron overload...
  62. ncbi Colonic spirochetosis in children and adults
    Ayman Koteish
    Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA
    Am J Clin Pathol 120:828-32. 2003
    ..While treatment information remains limited, treatment can lead to resolution of symptoms in some cases...
  63. ncbi Gastrointestinal plasmacytoma that caused anemia in a patient with multiple myeloma
    Tuba Esfandyari
    Mayo Clinic, Rochester, MN 55905, USA
    Nat Clin Pract Gastroenterol Hepatol 4:111-5. 2007
    ..He had required more than 15 U of packed red blood cells within a 2-month period for his anemia, despite recombinant erythropoietin treatment, and his hemoglobin level was persistently below 9 g/dl...
  64. doi Esophageal lichen planus
    Vishal S Chandan
    Division of Anatomic Pathology, Mayo Clinic, Rochester, MN 55905, USA
    Arch Pathol Lab Med 132:1026-9. 2008
    ..Esophageal lichen planus may have malignant potential, as evidenced by 3 patients who developed squamous carcinoma of the esophagus after longstanding disease...
  65. ncbi Fine-needle aspiration cytology findings from a case of pancreatic heterotopia at the gastroesophageal junction
    Fausto J Rodriguez
    Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota 55905, USA
    Diagn Cytopathol 31:175-9. 2004
    ..The increasing use of endoscopic ultrasound and fine-needle aspiration for the diagnosis of gastrointestinal tumors makes the recognition of pseudoneoplastic lesions at unusual sites an important step in optimum patient management...
  66. ncbi Hemobilia caused by cytomegalovirus cholangiopathy
    Ganapathy A Prasad
    Division of Gastroenterology and Hepatology, Department of Medicine, Mayo Clinic College of Medicine, Rochester, Minnesota 55901, USA
    Am J Gastroenterol 100:2592-5. 2005
    ..Biliary CMV disease is rare in HIV-negative persons, but should be considered in a patient with unexplained hemobilia. Cholangioscopy may be useful for diagnosis...
  67. ncbi Giant fibrovascular polyp of the esophagus. A lesion causing upper airway obstruction and syncope
    Matthew R Fries
    Department of Pathology, The Johns Hopkins University School of Medicine, Baltimore, MD, USA
    Arch Pathol Lab Med 127:485-7. 2003
    ..Despite their large size, giant fibrovascular polyps should be recognized radiologically and pathologically as benign lesions. However, they can result in significant morbidity...
  68. ncbi Glycogen pseudoground glass change in hepatocytes
    Joshua Wisell
    Department of Pathology, The Johns Hopkins Hospital, Baltimore, MD 21231, USA
    Am J Surg Pathol 30:1085-90. 2006
    ..The changes are generally seen in the background of mild chronic hepatitis with mild or no fibrosis. Glycogen pseudoground glass change can resolve, but may also persist for years...
  69. ncbi Achlorhydria, parietal cell hyperplasia, and multiple gastric carcinoids: a new disorder
    Pedram Argani
    Am J Surg Pathol 31:488. 2007
  70. ncbi Liver transplantation for severe intrahepatic noncirrhotic portal hypertension
    Alyssa M Krasinskas
    Departments of Pathology, University of Pittsburgh, PA, USA
    Liver Transpl 11:627-34; discussion 610-1. 2005
    ..Importantly, however, a diagnosis of cirrhosis is not required in this group of patients to qualify them for OLT, and these patients have good long-term graft function after OLT...
  71. ncbi Clinicopathologic findings and outcomes of liver transplantation using grafts from donors with unrecognized and unusual diseases
    Surakit Pungpapong
    Division of Gastroenterology and Hepatology, Mayo Clinic College of Medicine, Jacksonville, FL 32224, USA
    Liver Transpl 12:310-5. 2006
    ..In conclusion, long-term follow-up is necessary to confirm these findings and understand the impact of using these grafts...
  72. ncbi Globular amyloid deposits isolated to the small bowel: a rare association with AL amyloidosis
    Patrick R Hemmer
    Division of Anatomic Pathology, Department of Hematology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905, USA
    Am J Surg Pathol 31:141-5. 2007
    ..This endoscopic presentation of isolated small bowel polyposis is an uncommon association with AL amyloidosis and to our knowledge this represents the first case of globular gastrointestinal amyloidosis resulting from AL amyloid...
  73. ncbi Multicenter experience with upper gastrointestinal polyps in pediatric patients with familial adenomatous polyposis
    Thomas M Attard
    Department of Pediatrics, The University of Nebraska Medical Center, Omaha, NE 68198 5160, USA
    Am J Gastroenterol 99:681-6. 2004
    ..There are few data describing pediatric upper gastrointestinal FAP resulting in conflicting screening recommendations...
  74. ncbi Gastric adenomas: intestinal-type and gastric-type adenomas differ in the risk of adenocarcinoma and presence of background mucosal pathology
    Susan C Abraham
    Divison of Gastrointerstinal Liver Pathology, Department of Pahology, Johns Hopkins University School of Medicine, Baltimeore, Maryland
    Am J Surg Pathol 26:1276-85. 2002
    ..In any individual patient complete removal of the adenoma should be performed, and thorough biopsy of the surrounding gastric mucosa is essential to understand the clinicopathologic context of the adenoma...