Max A Tischfield

Summary

Affiliation: Johns Hopkins University
Country: USA

Publications

  1. ncbi Distinct alpha- and beta-tubulin isotypes are required for the positioning, differentiation and survival of neurons: new support for the 'multi-tubulin' hypothesis
    Max A Tischfield
    Department of Neurology and Ophthalmology, Manten Center for Orphan Disease Research, Children s Hospital Boston, Harvard Medical School, MA, USA
    Biosci Rep 30:319-30. 2010
  2. ncbi Human TUBB3 mutations perturb microtubule dynamics, kinesin interactions, and axon guidance
    Max A Tischfield
    Department of Neurology, Children s Hospital Boston, Boston, MA 02115, USA
    Cell 140:74-87. 2010
  3. ncbi An inherited TUBB2B mutation alters a kinesin-binding site and causes polymicrogyria, CFEOM and axon dysinnervation
    Gustav Y Cederquist
    Department of Neurology, Boston Children s Hospital, Boston, MA, USA
    Hum Mol Genet 21:5484-99. 2012

Collaborators

  • Joseph L Demer
  • Wei He
  • Gustav Y Cederquist
  • Manoj P Menezes
  • Anna Luchniak
  • Maree Flaherty
  • PATRICIA ELLEN GRANT
  • Sheena Chew
  • Mohan L Gupta
  • Maya Peeva
  • Elizabeth C Engle
  • Yuyu Song
  • Lavier Gomes
  • Wai Man Chan
  • Robyn V Jamieson
  • Caroline Andrews

Detail Information

Publications3

  1. ncbi Distinct alpha- and beta-tubulin isotypes are required for the positioning, differentiation and survival of neurons: new support for the 'multi-tubulin' hypothesis
    Max A Tischfield
    Department of Neurology and Ophthalmology, Manten Center for Orphan Disease Research, Children s Hospital Boston, Harvard Medical School, MA, USA
    Biosci Rep 30:319-30. 2010
    ....
  2. ncbi Human TUBB3 mutations perturb microtubule dynamics, kinesin interactions, and axon guidance
    Max A Tischfield
    Department of Neurology, Children s Hospital Boston, Boston, MA 02115, USA
    Cell 140:74-87. 2010
    ..These findings demonstrate that normal TUBB3 is required for axon guidance and maintenance in mammals...
  3. ncbi An inherited TUBB2B mutation alters a kinesin-binding site and causes polymicrogyria, CFEOM and axon dysinnervation
    Gustav Y Cederquist
    Department of Neurology, Boston Children s Hospital, Boston, MA, USA
    Hum Mol Genet 21:5484-99. 2012
    ..Together with previous studies, these findings highlight that β-tubulin isotypes function in both conserved and divergent ways to support proper human nervous system development...