Gay Rudow

Summary

Affiliation: Johns Hopkins University
Country: USA

Publications

  1. ncbi Morphometry of the human substantia nigra in ageing and Parkinson's disease
    Gay Rudow
    Neuropathology, Johns Hopkins School of Medicine, Ross Research Building 555, 720 Rutland Avenue, Baltimore, MD 21205, USA
    Acta Neuropathol 115:461-70. 2008
  2. ncbi Neuronal hypertrophy in asymptomatic Alzheimer disease
    Diego Iacono
    Division of Neuropathology, Department of Pathology, School of Medicine, Johns Hopkins University, Baltimore, Maryland 21205, USA
    J Neuropathol Exp Neurol 67:578-89. 2008
  3. ncbi Neuropathologic studies of the Baltimore Longitudinal Study of Aging (BLSA)
    RICHARD J O'BRIEN
    Department of Neurology, School of Medicine, Johns Hopkins University, Baltimore, MD 21224, USA
    J Alzheimers Dis 18:665-75. 2009
  4. ncbi Correspondence between in vivo (11)C-PiB-PET amyloid imaging and postmortem, region-matched assessment of plaques
    Ira Driscoll
    National Institute on Aging, Baltimore, MD, USA
    Acta Neuropathol 124:823-31. 2012
  5. ncbi Resistance to Alzheimer's pathology is associated with nuclear hypertrophy in neurons
    Miguel Angel Riudavets
    Division of Neuropathology, Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA
    Neurobiol Aging 28:1484-92. 2007
  6. ncbi Striatal neuronal loss correlates with clinical motor impairment in Huntington's disease
    Zhihong Guo
    Division of Neuropathology, Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205, USA
    Mov Disord 27:1379-86. 2012

Collaborators

Detail Information

Publications6

  1. ncbi Morphometry of the human substantia nigra in ageing and Parkinson's disease
    Gay Rudow
    Neuropathology, Johns Hopkins School of Medicine, Ross Research Building 555, 720 Rutland Avenue, Baltimore, MD 21205, USA
    Acta Neuropathol 115:461-70. 2008
    ..Presumably, this compensatory mechanism breaks down or is overwhelmed by the pathological events of PD leading to the onset of the characteristic motor disturbances...
  2. ncbi Neuronal hypertrophy in asymptomatic Alzheimer disease
    Diego Iacono
    Division of Neuropathology, Department of Pathology, School of Medicine, Johns Hopkins University, Baltimore, Maryland 21205, USA
    J Neuropathol Exp Neurol 67:578-89. 2008
    ....
  3. ncbi Neuropathologic studies of the Baltimore Longitudinal Study of Aging (BLSA)
    RICHARD J O'BRIEN
    Department of Neurology, School of Medicine, Johns Hopkins University, Baltimore, MD 21224, USA
    J Alzheimers Dis 18:665-75. 2009
    ..e., neuronal hypertrophy and synaptic plasticity) whereas a failure of compensation may culminate in disease...
  4. ncbi Correspondence between in vivo (11)C-PiB-PET amyloid imaging and postmortem, region-matched assessment of plaques
    Ira Driscoll
    National Institute on Aging, Baltimore, MD, USA
    Acta Neuropathol 124:823-31. 2012
    ..The strong associations of PiB retention with region-matched, quantitative analyses of Aβ in postmortem tissue offer support for the validity of (11)C-PiB-PET imaging as a method for evaluation of plaque burden in vivo...
  5. ncbi Resistance to Alzheimer's pathology is associated with nuclear hypertrophy in neurons
    Miguel Angel Riudavets
    Division of Neuropathology, Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA
    Neurobiol Aging 28:1484-92. 2007
    ..This nuclear hypertrophy may represent an early neuronal reaction to Abeta or Tau, or a compensatory mechanism which forestalls the progression of AD and allows the brain to resist the development of dementia...
  6. ncbi Striatal neuronal loss correlates with clinical motor impairment in Huntington's disease
    Zhihong Guo
    Division of Neuropathology, Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205, USA
    Mov Disord 27:1379-86. 2012
    ..Our findings suggest that neuronal loss and atrophy in striatum and neuronal loss in subthalamic nuclei contribute specifically to the motor impairment of HD, but not to chorea...