HARRY ALAN QUIGLEY

Summary

Affiliation: Johns Hopkins University
Country: USA

Publications

  1. pmc Mice with an induced mutation in collagen 8A2 develop larger eyes and are resistant to retinal ganglion cell damage in an experimental glaucoma model
    Matthew R Steinhart
    Glaucoma Center of Excellence, Wilmer Ophthalmological Institute, Johns Hopkins University, Baltimore, MD, USA
    Mol Vis 18:1093-106. 2012
  2. doi request reprint Clinical trials for glaucoma neuroprotection are not impossible
    Harry A Quigley
    Glaucoma Services and Dana Center for Preventive Ophthalmology, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Curr Opin Ophthalmol 23:144-54. 2012
  3. pmc Evaluation of a combined index of optic nerve structure and function for glaucoma diagnosis
    Michael V Boland
    Glaucoma Service, Dana Center for Preventive Ophthalmology, Wilmer Eye Institute, Johns Hopkins University, Baltimore, MD, USA
    BMC Ophthalmol 11:6. 2011
  4. pmc Long term results of glaucoma surgery among participants in an east African population survey
    H A Quigley
    Dana Center for Preventive Ophthalmology, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, MD, USA
    Br J Ophthalmol 84:860-4. 2000
  5. doi request reprint Improving eye drop treatment for glaucoma through better adherence
    Harry A Quigley
    Wilmer Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205 2020, USA
    Optom Vis Sci 85:374-5. 2008
  6. ncbi request reprint How are glaucoma patients identified?
    Harry A Quigley
    Glaucoma Service and Dana Center for Preventive Ophthalmology Wilmer Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland 21287, USA
    J Glaucoma 12:451-5. 2003
  7. ncbi request reprint Evaluation of practice patterns for the care of open-angle glaucoma compared with claims data: the Glaucoma Adherence and Persistency Study
    Harry A Quigley
    Glaucoma Service and Dana Center for Preventive Ophthalmology, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Ophthalmology 114:1599-606. 2007
  8. ncbi request reprint New paradigms in the mechanisms and management of glaucoma
    H A Quigley
    Wilmer Ophthalmological Institute, Johns Hopkins Hospital, Baltimore, MD, USA
    Eye (Lond) 19:1241-8. 2005
  9. pmc The number of people with glaucoma worldwide in 2010 and 2020
    H A Quigley
    The Glaucoma Service and the Dana Center for Preventive Opthalmalogy, Wilmer Opthalmological Institute, Johns Hopkins Hospital, 600 N Wolfe Street, Baltimore, MD 21287, USA
    Br J Ophthalmol 90:262-7. 2006
  10. ncbi request reprint Possible mechanisms of primary angle-closure and malignant glaucoma
    Harry A Quigley
    Glaucoma Service and the Dana Center for Preventive Ophthamology, Wilmer Ophthamological Institute, John Hopkins University School of Medicine, Baltimore, Maryland 21287, USA
    J Glaucoma 12:167-80. 2003

Research Grants

Detail Information

Publications54

  1. pmc Mice with an induced mutation in collagen 8A2 develop larger eyes and are resistant to retinal ganglion cell damage in an experimental glaucoma model
    Matthew R Steinhart
    Glaucoma Center of Excellence, Wilmer Ophthalmological Institute, Johns Hopkins University, Baltimore, MD, USA
    Mol Vis 18:1093-106. 2012
    ..To study susceptibility to glaucoma injury as it may be affected by mutations in ocular connective tissue components...
  2. doi request reprint Clinical trials for glaucoma neuroprotection are not impossible
    Harry A Quigley
    Glaucoma Services and Dana Center for Preventive Ophthalmology, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Curr Opin Ophthalmol 23:144-54. 2012
    ..To discuss issues that affect development of new glaucoma treatments known as neuroprotection...
  3. pmc Evaluation of a combined index of optic nerve structure and function for glaucoma diagnosis
    Michael V Boland
    Glaucoma Service, Dana Center for Preventive Ophthalmology, Wilmer Eye Institute, Johns Hopkins University, Baltimore, MD, USA
    BMC Ophthalmol 11:6. 2011
    ..We have therefore defined and tested a new approach to glaucoma diagnosis by combining imaging and visual field data, using the anatomical organization of retinal ganglion cells...
  4. pmc Long term results of glaucoma surgery among participants in an east African population survey
    H A Quigley
    Dana Center for Preventive Ophthalmology, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, MD, USA
    Br J Ophthalmol 84:860-4. 2000
    ..To evaluate the long term results of glaucoma surgery among people in East Africa...
  5. doi request reprint Improving eye drop treatment for glaucoma through better adherence
    Harry A Quigley
    Wilmer Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205 2020, USA
    Optom Vis Sci 85:374-5. 2008
  6. ncbi request reprint How are glaucoma patients identified?
    Harry A Quigley
    Glaucoma Service and Dana Center for Preventive Ophthalmology Wilmer Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland 21287, USA
    J Glaucoma 12:451-5. 2003
    ..To determine the events that lead to the diagnosis of glaucoma...
  7. ncbi request reprint Evaluation of practice patterns for the care of open-angle glaucoma compared with claims data: the Glaucoma Adherence and Persistency Study
    Harry A Quigley
    Glaucoma Service and Dana Center for Preventive Ophthalmology, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Ophthalmology 114:1599-606. 2007
    ..To perform a chart review to measure the validity of large claims databases in estimating patient cooperation with eyedrop therapy and to assess physician adherence with guidelines for a preferred practice pattern (PPP) using a new metric...
  8. ncbi request reprint New paradigms in the mechanisms and management of glaucoma
    H A Quigley
    Wilmer Ophthalmological Institute, Johns Hopkins Hospital, Baltimore, MD, USA
    Eye (Lond) 19:1241-8. 2005
    ..Diagnostic methods for glaucoma need improvement. The pathogenetic steps to loss of neurons in glaucoma are increasingly understood and non-pressure lowering therapies are on the horizon...
  9. pmc The number of people with glaucoma worldwide in 2010 and 2020
    H A Quigley
    The Glaucoma Service and the Dana Center for Preventive Opthalmalogy, Wilmer Opthalmological Institute, Johns Hopkins Hospital, 600 N Wolfe Street, Baltimore, MD 21287, USA
    Br J Ophthalmol 90:262-7. 2006
    ..To estimate the number of people with open angle (OAG) and angle closure glaucoma (ACG) in 2010 and 2020...
  10. ncbi request reprint Possible mechanisms of primary angle-closure and malignant glaucoma
    Harry A Quigley
    Glaucoma Service and the Dana Center for Preventive Ophthamology, Wilmer Ophthamological Institute, John Hopkins University School of Medicine, Baltimore, Maryland 21287, USA
    J Glaucoma 12:167-80. 2003
  11. doi request reprint Angle-closure glaucoma-simpler answers to complex mechanisms: LXVI Edward Jackson Memorial Lecture
    Harry A Quigley
    Glaucoma Service and Dana Center for Preventive Ophthalmology, Wilmer Institute, Johns Hopkins University School of Medicine, Baltimore, MD 21287, USA
    Am J Ophthalmol 148:657-669.e1. 2009
    ..To present evidence-based concepts that will explain mechanisms of angle-closure glaucoma...
  12. pmc Lack of neuroprotection against experimental glaucoma in c-Jun N-terminal kinase 3 knockout mice
    Harry A Quigley
    Glaucoma Research Laboratory, Wilmer Eye Institute, 600 North Wolfe Street, Johns Hopkins University School of Medicine, Baltimore, MD 21287 9205, USA
    Exp Eye Res 92:299-305. 2011
    ..Counts of RGC layer cells and optic nerve axons indicate that Jnk3(-/-) mice have an increased number of amacrine cells compared to WT controls...
  13. doi request reprint Iris cross-sectional area decreases with pupil dilation and its dynamic behavior is a risk factor in angle closure
    Harry A Quigley
    Wilmer Institute, Johns Hopkins University School of Medicine, Baltimore, MD, USA
    J Glaucoma 18:173-9. 2009
    ..To estimate the change in iris cross-sectional (CS) area with pupil dilation using anterior segment optical coherence tomography comparing eyes with angle closure (AC) to open angle glaucoma (OAG)...
  14. doi request reprint Glaucoma
    Harry A Quigley
    Glaucoma Service and Dana Center for Preventive Ophthalmology, Wilmer Ophthalmological Institute, Johns Hopkins School of Medicine, Baltimore, MD 21287, USA
    Lancet 377:1367-77. 2011
    ..Successful gene therapy, which has been used for other eye diseases might be possible for the treatment of glaucoma in the future...
  15. ncbi request reprint Community screening for eye disease by laypersons: the Hoffberger program
    Harry A Quigley
    Glaucoma Service and the Dana Center for Preventive Ophthalmology and the Department of Ophthalmology, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Am J Ophthalmol 133:386-92. 2002
    ..To describe the results of a community-based eye screening program in Baltimore...
  16. ncbi request reprint The prevalence of glaucoma in a population-based study of Hispanic subjects: Proyecto VER
    H A Quigley
    Dana Center for Preventive Ophthalmology, Wilmer Institute, The Johns Hopkins Hospital, 600 N Wolfe St, Baltimore, MD 21287, USA
    Arch Ophthalmol 119:1819-26. 2001
    ..To determine the prevalence of glaucoma in a population-based sample of Hispanic adults older than 40 years...
  17. ncbi request reprint Retrograde axonal transport of BDNF in retinal ganglion cells is blocked by acute IOP elevation in rats
    H A Quigley
    Glaucoma Research Laboratory, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Invest Ophthalmol Vis Sci 41:3460-6. 2000
    ..To determine whether acute experimental glaucoma in rats obstructs retrograde transport of brain-derived neurotrophic factor (BDNF) to retinal ganglion cells (RGCs)...
  18. ncbi request reprint Glaucoma: macrocosm to microcosm the Friedenwald lecture
    Harry A Quigley
    Glaucoma Service and Dana Center for Preventive Ophthalmology, Wilmer Institute, Johns Hopkins University School of Medicine, Baltimore, MD 21287, USA
    Invest Ophthalmol Vis Sci 46:2662-70. 2005
  19. ncbi request reprint Gene therapy with brain-derived neurotrophic factor as a protection: retinal ganglion cells in a rat glaucoma model
    Keith R G Martin
    Wilmer Eye Institute, Johns Hopkins University, Baltimore, Maryland 21287, USA
    Invest Ophthalmol Vis Sci 44:4357-65. 2003
    ....
  20. ncbi request reprint Comparison of optic nerve head topography and visual field in eyes with open-angle and angle-closure glaucoma
    Michael V Boland
    Dana Center for Preventive Ophthalmology, Wilmer Eye Institute, Johns Hopkins University, Baltimore, Maryland, USA
    Ophthalmology 115:239-245.e2. 2008
    ..To compare the optic disc and visual field (VF) alterations in primary open-angle glaucoma (POAG) and primary angle-closure glaucoma (PACG)...
  21. pmc Differential susceptibility to experimental glaucoma among 3 mouse strains using bead and viscoelastic injection
    Frances E Cone
    Glaucoma Research Laboratory, Johns Hopkins University School of Medicine, Wilmer Institute, Wilmer 122, 600 North Wolfe Street, Baltimore, MD 21287, USA
    Exp Eye Res 91:415-24. 2010
    ..Factors that determine sensitivity to RGC injury can be studied using transgenic mouse strains with inducible models...
  22. ncbi request reprint Changes in gene expression in experimental glaucoma and optic nerve transection: the equilibrium between protective and detrimental mechanisms
    Zhiyong Yang
    Department of Ophthalmology, Johns Hopkins University School of Medicine, Baltimore, MD 21287 9019, USA
    Invest Ophthalmol Vis Sci 48:5539-48. 2007
    ..The authors studied retinal gene expression changes in rats after experimental intraocular pressure elevation and optic nerve transection to elucidate molecular mechanisms of retinal ganglion cell (RGC) death...
  23. doi request reprint Glaucomatous optic nerve injury involves early astrocyte reactivity and late oligodendrocyte loss
    Janice L Son
    Hugo W Moser Research Institute at Kennedy Krieger, Baltimore, Maryland, USA
    Glia 58:780-9. 2010
    ..In contrast, a large increase in astrocyte reactivity occurred early in both animal models. These results are consistent with astrocytes playing a prominent role in regulating axon loss in glaucoma...
  24. ncbi request reprint Translimbal laser photocoagulation to the trabecular meshwork as a model of glaucoma in rats
    Hana Levkovitch-Verbin
    Glaucoma Research Laboratory, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland 21287, USA
    Invest Ophthalmol Vis Sci 43:402-10. 2002
    ..To develop and characterize a model of pressure-induced optic neuropathy in rats...
  25. ncbi request reprint Optic nerve dynein motor protein distribution changes with intraocular pressure elevation in a rat model of glaucoma
    Keith R G Martin
    Glaucoma Research Laboratory, Wilmer Eye Institute, Johns Hopkins University, Baltimore, MD, USA
    Exp Eye Res 83:255-62. 2006
    ..The effect of IOP elevation on other motor proteins deserves further investigation in the future...
  26. ncbi request reprint Long-term outcomes in fellow eyes after acute primary angle closure in the contralateral eye
    David S Friedman
    Wilmer Eye Institute, Johns Hopkins University, Johns Hopkins Hospital, Baltimore, Maryland 21287, USA
    Ophthalmology 113:1087-91. 2006
    ..To determine the long-term outcome of the contralateral eye in Asian persons with a unilateral attack of acute primary angle closure (APAC)...
  27. pmc The effect of experimental glaucoma and optic nerve transection on amacrine cells in the rat retina
    Jennifer L Kielczewski
    Glaucoma Research Laboratory, Wilmer Eye Institute, Johns Hopkins University School of Medicine, 600 North Wolfe Street, Baltimore, MD, USA
    Invest Ophthalmol Vis Sci 46:3188-96. 2005
    ..To detect alterations in amacrine cells associated with retinal ganglion cell (RGC) depletion caused by experimental optic nerve transection and glaucoma...
  28. ncbi request reprint Using pharmacy claims data to study adherence to glaucoma medications: methodology and findings of the Glaucoma Adherence and Persistency Study (GAPS)
    David S Friedman
    Wilmer Eye Institute, Baltimore, MD, USA
    Invest Ophthalmol Vis Sci 48:5052-7. 2007
    ..To develop methods for investigating adherence to glaucoma medications by using a modified claims data-based measure of adherence, validation by chart review, and patient and physician interviews...
  29. doi request reprint Risk factors for poor adherence to eyedrops in electronically monitored patients with glaucoma
    David S Friedman
    Wilmer Eye Institute, Department of Ophthalmology, Johns Hopkins University School of Medicine, Baltimore, MD 21210, USA
    Ophthalmology 116:1097-105. 2009
    ..To identify risk factors for poor adherence to topical once daily therapy for glaucoma...
  30. ncbi request reprint A model to study differences between primary and secondary degeneration of retinal ganglion cells in rats by partial optic nerve transection
    Hana Levkovitch-Verbin
    Department of Ophthalmology, Wilmer Institute, Johns Hopkins Hospital, Baltimore, Maryland, USA
    Invest Ophthalmol Vis Sci 44:3388-93. 2003
    ..To use a rat model of optic nerve injury to differentiate primary and secondary retinal ganglion cell (RGC) injury...
  31. ncbi request reprint Glaucoma management among individuals enrolled in a single comprehensive insurance plan
    David S Friedman
    Dana Center for Preventive Ophthalmology, Wilmer Eye Institute, Johns Hopkins Medical Institutions, Baltimore, Maryland 21287, USA
    Ophthalmology 112:1500-4. 2005
    ..To determine the management patterns for glaucoma and suspect glaucoma in a nationally representative sample of newly treated persons...
  32. ncbi request reprint Variations in treatment among adult-onset open-angle glaucoma patients
    David S Friedman
    Dana Center for Preventive Ophthalmology, Wilmer Eye Institute, Johns Hopkins Medical Institutions, Baltimore, Maryland 21287, USA
    Ophthalmology 112:1494-9. 2005
    ..To determine the predictors of treatment for glaucoma and suspect glaucoma in a nationally representative sample of diagnosed persons...
  33. ncbi request reprint Gene expression profiling of purified rat retinal ganglion cells
    Ronald H Farkas
    Department of Ophthalmology, Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD 21287, USA
    Invest Ophthalmol Vis Sci 45:2503-13. 2004
    ..Retinal ganglion cells (RGCs) are of wide interest in neuroscience and die in glaucoma and other optic neuropathies. In this study the genes expressed by RGCs were profiled by expressed sequence tag (EST) analysis...
  34. ncbi request reprint Increased expression of iron-regulating genes in monkey and human glaucoma
    Ronald H Farkas
    Guerrieri Center for Genetic Engineering and Molecular Ophthalmology, Wilmer Ophthalmological Institute, Baltimore, Maryland 21287, USA
    Invest Ophthalmol Vis Sci 45:1410-7. 2004
    ..To understand the mechanisms mediating retinal ganglion cell loss in glaucoma, the gene expression patterns were compared for transferrin, ceruloplasmin, and ferritin between normal and glaucomatous retina in monkey and human eyes...
  35. ncbi request reprint Retinal glutamate transporter changes in experimental glaucoma and after optic nerve transection in the rat
    Keith R G Martin
    Glaucoma Research Laboratory, Wilmer Eye Institute, Johns Hopkins University, 600 North Wolfe Street, Baltimore, MD 21287, USA
    Invest Ophthalmol Vis Sci 43:2236-43. 2002
    ..This study was conducted to investigate whether glutamate transporter changes occur with two models of optic nerve injury in the rat...
  36. ncbi request reprint The impact of visual impairment and eye disease on vision-related quality of life in a Mexican-American population: proyecto VER
    Aimee Teo Broman
    Dana Center for Preventive Ophthalmology, Johns Hopkins School of Medicine, Baltimore, Maryland, USA
    Invest Ophthalmol Vis Sci 43:3393-8. 2002
    ....
  37. ncbi request reprint Central corneal thickness and corneal hysteresis associated with glaucoma damage
    Nathan G Congdon
    Wilmer Eye Institute, Johns Hopkins University School of Medicine, 600 N Wolfe Street, Baltimore, MD 21287, USA
    Am J Ophthalmol 141:868-75. 2006
    ..We sought to measure the impact of central corneal thickness (CCT), a possible risk factor for glaucoma damage, and corneal hysteresis, a proposed measure of corneal resistance to deformation, on various indicators of glaucoma damage...
  38. ncbi request reprint Gaze behavior of the visually impaired during street crossing
    Duane R Geruschat
    The Maryland School for the Blind, Baltimore, MD, USA
    Optom Vis Sci 83:550-8. 2006
    ..This study explored the gaze patterns of fully sighted and visually impaired subjects during the high-risk activity of crossing the street...
  39. pmc The presence and distribution of elastin in the posterior and retrobulbar regions of the mouse eye
    Scott Gelman
    Glaucoma Research Laboratory, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, MD 21287, USA
    Exp Eye Res 90:210-5. 2010
    ..Elastin comprises a portion of the mouse sclera and its distribution in the peripapillary area is similar to that in human eyes...
  40. ncbi request reprint Manometric calibration and comparison of TonoLab and TonoPen tonometers in rats with experimental glaucoma and in normal mice
    Mary E Pease
    Glaucoma Service, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, MD 21287, USA
    J Glaucoma 15:512-9. 2006
    ..To compare the TonoPen and TonoLab tonometers to each other and to manometrically set intraocular pressure (IOP) in the eyes of normal mice, normal rats, and rats with chronic IOP elevation...
  41. pmc Effect of CNTF on retinal ganglion cell survival in experimental glaucoma
    Mary Ellen Pease
    Department of Ophthalmology, Johns Hopkins University School of Medicine, 600 N Wolfe Street, 175 Woods Research Building, Baltimore, MD 21287, USA
    Invest Ophthalmol Vis Sci 50:2194-200. 2009
    ..To assess the neuroprotective effect of virally mediated overexpression of ciliary-derived neurotrophic factor (CNTF) and brain-derived neurotrophic factor (BDNF) in experimental rat glaucoma...
  42. pmc Estimating the rate of progressive visual field damage in those with open-angle glaucoma, from cross-sectional data
    Aimee Teo Broman
    Dana Center for Preventive Ophthalmology and the Glaucoma Service, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, MD 21287, USA
    Invest Ophthalmol Vis Sci 49:66-76. 2008
    ..To estimate the rate of visual field progression in open-angle glaucoma (OAG) subjects, by using data from population-based cross-sectional studies...
  43. ncbi request reprint The TRAVATAN Dosing Aid accurately records when drops are taken
    David S Friedman
    Glaucoma Division of the Wilmer Eye Institute, Johns Hopkins University, Baltimore, Maryland, USA
    Am J Ophthalmol 143:699-701. 2007
    ..To assess the accuracy of Travatan Dosing Aid recordings...
  44. ncbi request reprint Influence of corneal structure, corneal responsiveness, and other ocular parameters on tonometric measurement of intraocular pressure
    Aimee Teo Broman
    Dana Center for Preventive Ophthalmology, The Johns Hopkins School of Medicine, Baltimore, MD, USA
    J Glaucoma 16:581-8. 2007
    ....
  45. ncbi request reprint Characteristics of progenitor cells derived from adult ciliary body in mouse, rat, and human eyes
    Hangxiu Xu
    Glaucoma Research Laboratory, Wilmer Eye Institute, Johns Hopkins University School of Medicine, 600 North Wolfe Street, Baltimore, MD 21287, USA
    Invest Ophthalmol Vis Sci 48:1674-82. 2007
    ..To isolate and characterize progenitor cells derived from adult mammalian ciliary body...
  46. ncbi request reprint Measurement of amino acid levels in the vitreous humor of rats after chronic intraocular pressure elevation or optic nerve transection
    Hana Levkovitch-Verbin
    Glaucoma Service, Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    J Glaucoma 11:396-405. 2002
    ..To investigate whether the levels of free amino acids and protein in the vitreous of rat eyes are altered with chronic intraocular pressure (IOP) elevation or after optic nerve transection...
  47. pmc The in vitro inflation response of mouse sclera
    Kristin M Myers
    Department of Mechanical Engineering, Johns Hopkins University, 3400 North Charles St, Latrobe 125, Baltimore, MD 21218, USA
    Exp Eye Res 91:866-75. 2010
    ..Further, the specimen-specific scleral displacement profile and thickness measurements will enable future development of specimen-specific finite element models to analyze the inflation data for material properties...
  48. pmc In vivo time-lapse fluorescence imaging of individual retinal ganglion cells in mice
    Mark K Walsh
    Wilmer Eye Institute, Johns Hopkins University School of Medicine, 600 N Wolfe Street, Baltimore, MD 21287, USA
    J Neurosci Methods 169:214-21. 2008
    ..Additionally, the same RGC that was imaged in vivo could then be located and imaged in fixed retinal whole mount preparations. This novel technique has many potential applications...
  49. ncbi request reprint Gene delivery to the eye using adeno-associated viral vectors
    Keith R G Martin
    Wilmer Eye Institute, Wilmer 122, Johns Hopkins Hospital, 600 North Wolfe Street, Baltimore, MD 21287, USA
    Methods 28:267-75. 2002
    ..Our techniques facilitate the study of the pathogenesis of RGC diseases such as glaucoma and the development of novel new treatments based on gene therapy...
  50. doi request reprint Doctor-patient communication, health-related beliefs, and adherence in glaucoma results from the Glaucoma Adherence and Persistency Study
    David S Friedman
    Wilmer Eye Institute, Baltimore, Maryland 21287, USA
    Ophthalmology 115:1320-7, 1327.e1-3. 2008
    ..To use multiple data sources to determine drivers of patient adherence to topical ocular hypotensive therapy...
  51. doi request reprint Doctor-patient communication in glaucoma care: analysis of videotaped encounters in community-based office practice
    David S Friedman
    Wilmer Eye Institute, Baltimore, Maryland, USA
    Ophthalmology 116:2277-85.e1-3. 2009
    ..To assess doctor-patient communication in patients with glaucoma...
  52. doi request reprint Adherence and persistence with glaucoma therapy
    Gail F Schwartz
    Greater Baltimore Medical Center, Baltimore, Maryland 21204, USA
    Surv Ophthalmol 53:S57-68. 2008
    ..Greater physician awareness of adherence and persistence issues is necessary in order to help the patient become more adherent...
  53. ncbi request reprint A self-assessment instrument designed for measuring independent mobility in RP patients: generalizability to glaucoma patients
    Kathleen A Turano
    Wilmer Eye Institute, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    Invest Ophthalmol Vis Sci 43:2874-81. 2002
    ....
  54. ncbi request reprint Risk factors and open-angle glaucoma: classification and application
    Michael V Boland
    Dana Center for Preventive Ophthalmology, Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD 21287, USA
    J Glaucoma 16:406-18. 2007
    ..We also describe methods for using risk factors in clinical practice and describe ways that a large and complex body of knowledge can be applied to individual patients...

Research Grants34

  1. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 2009
    ..To complete the project in 2 years, we eliminated from the 5 year proposal Hypotheses 7, 8, 9, and 11. ..
  2. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 2000
    ..In these rats, induced neuronal expression will be studied in a chronic glaucoma model to assess beneficial effects. ..
  3. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 2001
    ..In these rats, induced neuronal expression will be studied in a chronic glaucoma model to assess beneficial effects. ..
  4. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 2002
    ..In these rats, induced neuronal expression will be studied in a chronic glaucoma model to assess beneficial effects. ..
  5. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 2003
    ..The studies proposed here have already suggested several new avenues of non-intraocular pressure lowering therapy for glaucoma. ..
  6. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 2004
    ..The studies proposed here have already suggested several new avenues of non-intraocular pressure lowering therapy for glaucoma. ..
  7. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 2005
    ..The studies proposed here have already suggested several new avenues of non-intraocular pressure lowering therapy for glaucoma. ..
  8. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 2006
    ..The studies proposed here have already suggested several new avenues of non-intraocular pressure lowering therapy for glaucoma. ..
  9. Wilmer Inst. Mentored Clinical Research Scholar Program
    Harry Quigley; Fiscal Year: 2007
    ..The Wilmer CRS will administer didactic courses and personal mentored instruction in a wide variety of research areas to two candidates per year. ..
  10. THE PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 1980
  11. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 1999
    ..In these rats, induced neuronal expression will be studied in a chronic glaucoma model to assess beneficial effects. ..
  12. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 1990
    ....
  13. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 1991
    ....
  14. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 1992
    ....
  15. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 1993
    ....
  16. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    Harry Quigley; Fiscal Year: 2007
    ..The studies proposed here have already suggested several new avenues of non-intraocular pressure lowering therapy for glaucoma. ..
  17. PATHOGENESIS OF GLAUCOMATOUS OPTIC NERVE DAMAGE
    HARRY ALAN QUIGLEY; Fiscal Year: 2010
    ..To complete the project in 2 years, we eliminated from the 5 year proposal Hypotheses 7, 8, 9, and 11. ..