D Brian Foster

Summary

Affiliation: Johns Hopkins University
Country: USA

Publications

  1. pmc Redox signaling and protein phosphorylation in mitochondria: progress and prospects
    D Brian Foster
    Division of Cardiology, Department of Medicine, The Johns Hopkins University School of Medicine, Ross Research Building, Room 847, 720 Rutland Avenue, Baltimore, MD 21205, USA
    J Bioenerg Biomembr 41:159-68. 2009
  2. pmc A mighty small heart: the cardiac proteome of adult Drosophila melanogaster
    Anthony Cammarato
    Development and Aging Program, NASCR Center, Sanford Burnham Medical Research Institute, La Jolla, California, United States of America
    PLoS ONE 6:e18497. 2011
  3. pmc The cardiac acetyl-lysine proteome
    D Brian Foster
    Division of Cardiology, Department of Medicine, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    PLoS ONE 8:e67513. 2013
  4. pmc Mitochondrial ROMK channel is a molecular component of mitoK(ATP)
    D Brian Foster
    Division of Cardiology, The Johns Hopkins University School of Medicine, Baltimore, MD, USA
    Circ Res 111:446-54. 2012
  5. pmc Redox regulation of mitochondrial ATP synthase: implications for cardiac resynchronization therapy
    Sheng bing Wang
    Department of Medicine, Johns Hopkins University, Baltimore, MD 21224, USA
    Circ Res 109:750-7. 2011
  6. pmc Identification and characterization of a functional mitochondrial angiotensin system
    Peter M Abadir
    Division of Geriatric Medicine and Gerontology, Biology of Healthy Aging Program, Johns Hopkins University School of Medicine, Baltimore, MD 21224, USA
    Proc Natl Acad Sci U S A 108:14849-54. 2011
  7. pmc Nitroxyl-mediated disulfide bond formation between cardiac myofilament cysteines enhances contractile function
    Wei Dong Gao
    Department of Anesthesiology and Critical Care Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA
    Circ Res 111:1002-11. 2012
  8. doi Mitochondrial protein phosphorylation as a regulatory modality: implications for mitochondrial dysfunction in heart failure
    BRIAN O'ROURKE
    Department of Medicine, Division of Cardiology, The Johns Hopkins University, Baltimore, MD 21205 2195, USA
    Congest Heart Fail 17:269-82. 2011
  9. pmc Creatine kinase-mediated improvement of function in failing mouse hearts provides causal evidence the failing heart is energy starved
    Ashish Gupta
    Department of Medicine, Cardiology Division, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    J Clin Invest 122:291-302. 2012
  10. pmc CAPON modulates cardiac repolarization via neuronal nitric oxide synthase signaling in the heart
    Kuan Cheng Chang
    Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, MD 21205, USA
    Proc Natl Acad Sci U S A 105:4477-82. 2008

Collaborators

Detail Information

Publications12

  1. pmc Redox signaling and protein phosphorylation in mitochondria: progress and prospects
    D Brian Foster
    Division of Cardiology, Department of Medicine, The Johns Hopkins University School of Medicine, Ross Research Building, Room 847, 720 Rutland Avenue, Baltimore, MD 21205, USA
    J Bioenerg Biomembr 41:159-68. 2009
    ....
  2. pmc A mighty small heart: the cardiac proteome of adult Drosophila melanogaster
    Anthony Cammarato
    Development and Aging Program, NASCR Center, Sanford Burnham Medical Research Institute, La Jolla, California, United States of America
    PLoS ONE 6:e18497. 2011
    ....
  3. pmc The cardiac acetyl-lysine proteome
    D Brian Foster
    Division of Cardiology, Department of Medicine, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    PLoS ONE 8:e67513. 2013
    ..New sites suggest a host of potential mechanisms by which excitation-contraction coupling may also be modulated. ..
  4. pmc Mitochondrial ROMK channel is a molecular component of mitoK(ATP)
    D Brian Foster
    Division of Cardiology, The Johns Hopkins University School of Medicine, Baltimore, MD, USA
    Circ Res 111:446-54. 2012
    ..Objective: To use an unbiased proteomic analysis of the mitochondrial inner membrane to identify the mitochondrial K(+) channel underlying mitoK(ATP)...
  5. pmc Redox regulation of mitochondrial ATP synthase: implications for cardiac resynchronization therapy
    Sheng bing Wang
    Department of Medicine, Johns Hopkins University, Baltimore, MD 21224, USA
    Circ Res 109:750-7. 2011
    ..Our recent studies have revealed that mitochondrial posttranslational modifications (PTM) may contribute to its benefits, motivating the present study of the oxidative regulation of mitochondrial ATP synthase...
  6. pmc Identification and characterization of a functional mitochondrial angiotensin system
    Peter M Abadir
    Division of Geriatric Medicine and Gerontology, Biology of Healthy Aging Program, Johns Hopkins University School of Medicine, Baltimore, MD 21224, USA
    Proc Natl Acad Sci U S A 108:14849-54. 2011
    ....
  7. pmc Nitroxyl-mediated disulfide bond formation between cardiac myofilament cysteines enhances contractile function
    Wei Dong Gao
    Department of Anesthesiology and Critical Care Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA
    Circ Res 111:1002-11. 2012
    ..Nitroxyl (HNO), the one-electron-reduced form of nitric oxide, enhances cardiac function in a manner that suggests reversible cysteine modifications of the contractile machinery...
  8. doi Mitochondrial protein phosphorylation as a regulatory modality: implications for mitochondrial dysfunction in heart failure
    BRIAN O'ROURKE
    Department of Medicine, Division of Cardiology, The Johns Hopkins University, Baltimore, MD 21205 2195, USA
    Congest Heart Fail 17:269-82. 2011
    ..The authors review phosphorylation as a mitochondrial regulatory strategy and highlight its possible role in the pathophysiology of cardiac hypertrophy and failure...
  9. pmc Creatine kinase-mediated improvement of function in failing mouse hearts provides causal evidence the failing heart is energy starved
    Ashish Gupta
    Department of Medicine, Cardiology Division, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
    J Clin Invest 122:291-302. 2012
    ..In addition, these data identify CK as a promising therapeutic target for preventing and treating HF and possibly diseases involving energy-dependent dysfunction in other organs with temporally varying energy demands...
  10. pmc CAPON modulates cardiac repolarization via neuronal nitric oxide synthase signaling in the heart
    Kuan Cheng Chang
    Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, MD 21205, USA
    Proc Natl Acad Sci U S A 105:4477-82. 2008
    ..Our findings provide a rationale for the association of CAPON gene variants with extremes of the QT interval in human populations...
  11. pmc Constitutive HIF-1α expression blunts the beneficial effects of cardiosphere-derived cell therapy in the heart by altering paracrine factor balance
    Michael Bonios
    Johns Hopkins University, Baltimore, MD 21205, USA
    J Cardiovasc Transl Res 4:363-72. 2011
    ..HIF-1α expression in CDCs blunted the beneficial functional effects of CDC transplantation, suggesting that paracrine factor balance may play an important role in cardiac regeneration...
  12. pmc Is Kir6.1 a subunit of mitoK(ATP)?
    D Brian Foster
    Institute of Molecular Cardiobiology, Division of Cardiology, Johns Hopkins School of Medicine, Baltimore, MD, USA
    Biochem Biophys Res Commun 366:649-56. 2008
    ..1D-, 2D-, and native gel analyses were consistent with these assignments. The data suggest it is premature to assign Kir6.1 a role in mitoK(ATP) on the basis of immunoreactivity alone...