Ramzi M Kafoury

Summary

Affiliation: Jackson State University
Country: USA

Publications

  1. ncbi request reprint Application of quantitative structure activity relationship (QSAR) models to predict ozone toxicity in the lung
    Ramzi M Kafoury
    Center for Environmental Health, College of Science, Engineering, and Technology, Jackson State University, Jackson, Mississippi 39217, USA
    Environ Toxicol 20:441-8. 2005
  2. ncbi request reprint Diesel exhaust particles induce the over expression of tumor necrosis factor-alpha (TNF-alpha) gene in alveolar macrophages and failed to induce apoptosis through activation of nuclear factor-kappaB (NF-kappaB)
    Ramzi M Kafoury
    Center of Environmental Health and Molecular Toxicology Laboratory, Jackson State University, Jackson, Mississippi, USA
    Int J Environ Res Public Health 2:107-13. 2005
  3. ncbi request reprint Activation of transcription factor IL-6 (NF-IL-6) and nuclear factor-kappaB (NF-kappaB) by lipid ozonation products is crucial to interleukin-8 gene expression in human airway epithelial cells
    Ramzi M Kafoury
    Center for Environmental Health, Jackson State University, Jackson, Mississippi, USA
    Environ Toxicol 22:159-68. 2007
  4. ncbi request reprint Ozone enhances diesel exhaust particles (DEP)-induced interleukin-8 (IL-8) gene expression in human airway epithelial cells through activation of nuclear factors- kappaB (NF-kappaB) and IL-6 (NF-IL6)
    Ramzi M Kafoury
    Center of Environmental Health and Molecular Toxicology Laboratory, College of Science, Engineering and Technology, Jackson State University, Jackson, MS 39217, USA
    Int J Environ Res Public Health 2:403-10. 2005

Detail Information

Publications4

  1. ncbi request reprint Application of quantitative structure activity relationship (QSAR) models to predict ozone toxicity in the lung
    Ramzi M Kafoury
    Center for Environmental Health, College of Science, Engineering, and Technology, Jackson State University, Jackson, Mississippi 39217, USA
    Environ Toxicol 20:441-8. 2005
    ..97, 0.98, 0.99, for PLA2, PLC, and PLD, respectively). The results indicate that QSAR models can be utilized to predict the biological activity of the various ozone-derived LOP species in the lung...
  2. ncbi request reprint Diesel exhaust particles induce the over expression of tumor necrosis factor-alpha (TNF-alpha) gene in alveolar macrophages and failed to induce apoptosis through activation of nuclear factor-kappaB (NF-kappaB)
    Ramzi M Kafoury
    Center of Environmental Health and Molecular Toxicology Laboratory, Jackson State University, Jackson, Mississippi, USA
    Int J Environ Res Public Health 2:107-13. 2005
    ..DEP-induced TNF-alpha gene expression is regulated at the transcriptional level by NF-kappaB. Furthermore, DEP-induced increase in NF-kappaB-DNA binding activity appears to protect against apoptosis...
  3. ncbi request reprint Activation of transcription factor IL-6 (NF-IL-6) and nuclear factor-kappaB (NF-kappaB) by lipid ozonation products is crucial to interleukin-8 gene expression in human airway epithelial cells
    Ramzi M Kafoury
    Center for Environmental Health, Jackson State University, Jackson, Mississippi, USA
    Environ Toxicol 22:159-68. 2007
    ..05). The results of the present study indicate that NF-IL-6 interacts with NF-kappaB in regulating the expression of IL-8 in cultured human airway epithelial cells exposed to LOP, the biological products of ozone in the lung...
  4. ncbi request reprint Ozone enhances diesel exhaust particles (DEP)-induced interleukin-8 (IL-8) gene expression in human airway epithelial cells through activation of nuclear factors- kappaB (NF-kappaB) and IL-6 (NF-IL6)
    Ramzi M Kafoury
    Center of Environmental Health and Molecular Toxicology Laboratory, College of Science, Engineering and Technology, Jackson State University, Jackson, MS 39217, USA
    Int J Environ Res Public Health 2:403-10. 2005
    ..The results of the present study indicate that ozone exposure enhances the toxicity of DEP in human airway epithelial cells by augmenting IL-8 gene expression, a potent chemoattractant of neutrophils in the lung...