Wilbert A Derbigny

Summary

Affiliation: Indiana University
Country: USA

Publications

  1. ncbi The Chlamydia muridarum-induced IFN-β response is TLR3-dependent in murine oviduct epithelial cells
    Wilbert A Derbigny
    Department of Microbiology and Immunology, Indiana University School of Medicine, Indianapolis, IN 46202, USA
    J Immunol 185:6689-97. 2010
  2. ncbi Identifying a role for Toll-like receptor 3 in the innate immune response to Chlamydia muridarum infection in murine oviduct epithelial cells
    Wilbert A Derbigny
    Department of Microbiology and Immunology, Indiana University School of Medicine, Indianapolis, IN, USA
    Infect Immun 80:254-65. 2012
  3. ncbi Chlamydia muridarum infection elicits a beta interferon response in murine oviduct epithelial cells dependent on interferon regulatory factor 3 and TRIF
    Wilbert A Derbigny
    Department of Medicine, Indiana University School of Medicine, Indianapolis, IN 46202, USA
    Infect Immun 75:1280-90. 2007
  4. ncbi Pattern recognition molecules activated by Chlamydia muridarum infection of cloned murine oviduct epithelial cell lines
    Wilbert A Derbigny
    Department of Medicine, Indiana University School of Medicine, Indianapolis, IN 46202, USA
    J Immunol 175:6065-75. 2005

Research Grants

  1. Chlamydia-infected Epithelial Cell Secretion of Immunoregulatory Cytokines
    WILBERT DERBIGNY; Fiscal Year: 2007

Detail Information

Publications4

  1. ncbi The Chlamydia muridarum-induced IFN-β response is TLR3-dependent in murine oviduct epithelial cells
    Wilbert A Derbigny
    Department of Microbiology and Immunology, Indiana University School of Medicine, Indianapolis, IN 46202, USA
    J Immunol 185:6689-97. 2010
    ....
  2. ncbi Identifying a role for Toll-like receptor 3 in the innate immune response to Chlamydia muridarum infection in murine oviduct epithelial cells
    Wilbert A Derbigny
    Department of Microbiology and Immunology, Indiana University School of Medicine, Indianapolis, IN, USA
    Infect Immun 80:254-65. 2012
    ..Our OE cell data implicate TLR3 as an essential inducer of IFN-β and other inflammatory mediators by epithelial cells during Chlamydia infection and highlight the contribution of TLR3 to the inflammatory cytokine response...
  3. ncbi Chlamydia muridarum infection elicits a beta interferon response in murine oviduct epithelial cells dependent on interferon regulatory factor 3 and TRIF
    Wilbert A Derbigny
    Department of Medicine, Indiana University School of Medicine, Indianapolis, IN 46202, USA
    Infect Immun 75:1280-90. 2007
    ..Antagonism of either IRF-3 or TRIF signaling significantly decreased IFN-beta production. These data implicate TLR3, or an unknown PRR utilizing TRIF, as the source of IFN-beta production by Chlamydia-infected oviduct epithelial cells...
  4. ncbi Pattern recognition molecules activated by Chlamydia muridarum infection of cloned murine oviduct epithelial cell lines
    Wilbert A Derbigny
    Department of Medicine, Indiana University School of Medicine, Indianapolis, IN 46202, USA
    J Immunol 175:6065-75. 2005
    ..muridarum-infected oviduct epithelial cell lines. The pattern recognition molecule responsible for infection-induced IFN-beta secretion by oviduct epithelial cells remains to be determined...

Research Grants2

  1. Chlamydia-infected Epithelial Cell Secretion of Immunoregulatory Cytokines
    WILBERT DERBIGNY; Fiscal Year: 2007
    ..no abstract was provided by the pi. ..