D A Waltz

Summary

Affiliation: Harvard University
Country: USA

Publications

  1. pmc Plasmin and plasminogen activator inhibitor type 1 promote cellular motility by regulating the interaction between the urokinase receptor and vitronectin
    D A Waltz
    Division of Respiratory Diseases, Children s Hospital, Brigham and Women s Hospital, and Harvard Medical School, Boston, Massachusetts 02115, USA
    J Clin Invest 100:58-67. 1997
  2. ncbi request reprint Nonproteolytic role for the urokinase receptor in cellular migration in vivo
    D A Waltz
    Department of Medicine, Children s Hospital, Boston, Massachusetts 02115, USA
    Am J Respir Cell Mol Biol 22:316-22. 2000
  3. ncbi request reprint Role of urokinase receptor and caveolin in regulation of integrin signaling
    H A Chapman
    Department of Medicine, Brigham and Women s Hospital, Boston, MA 02115, USA
    Thromb Haemost 82:291-7. 1999
  4. ncbi request reprint Pediatric lung transplantation and "lessons from Green Surgery."
    C W Lillehei
    Department of Surgery, Children s Hospital, Harvard Medical School, Boston, Massachusetts 02115, USA
    Ann Thorac Surg 68:S25-7. 1999
  5. ncbi request reprint Lack of association of secretory component with IgA in J chain-deficient mice
    B A Hendrickson
    Division of Infectious Diseases, Children s Hospital, Boston, MA 02115, USA
    J Immunol 157:750-4. 1996

Collaborators

Detail Information

Publications5

  1. pmc Plasmin and plasminogen activator inhibitor type 1 promote cellular motility by regulating the interaction between the urokinase receptor and vitronectin
    D A Waltz
    Division of Respiratory Diseases, Children s Hospital, Brigham and Women s Hospital, and Harvard Medical School, Boston, Massachusetts 02115, USA
    J Clin Invest 100:58-67. 1997
    ..Excess PAI-1 may also promote migration by blocking cellular adhesion and/or promoting detachment, possibly accounting in part for the strong correlation between PAI-1 expression and tumor cell metastasis...
  2. ncbi request reprint Nonproteolytic role for the urokinase receptor in cellular migration in vivo
    D A Waltz
    Department of Medicine, Children s Hospital, Boston, Massachusetts 02115, USA
    Am J Respir Cell Mol Biol 22:316-22. 2000
    ..These observations verify that protease-independent functions of uPAR operate in vivo and identify uPAR as a potential target for regulation of inflammatory processes characterized by neutrophil-mediated injury...
  3. ncbi request reprint Role of urokinase receptor and caveolin in regulation of integrin signaling
    H A Chapman
    Department of Medicine, Brigham and Women s Hospital, Boston, MA 02115, USA
    Thromb Haemost 82:291-7. 1999
    ..Interruption of u-PAR/integrin interactions may be a strategy to regulate cellular migration in these settings...
  4. ncbi request reprint Pediatric lung transplantation and "lessons from Green Surgery."
    C W Lillehei
    Department of Surgery, Children s Hospital, Harvard Medical School, Boston, Massachusetts 02115, USA
    Ann Thorac Surg 68:S25-7. 1999
    ..Our experience parallels that of other frontiers such as early cardiac surgery in which medical and technologic innovations can be applied in a supportive environment to permit surgical progress...
  5. ncbi request reprint Lack of association of secretory component with IgA in J chain-deficient mice
    B A Hendrickson
    Division of Infectious Diseases, Children s Hospital, Boston, MA 02115, USA
    J Immunol 157:750-4. 1996
    ..Further, we suggest that J chain-deficient IgA is transported into secretions by a different mechanism than wild-type IgA...