D Traver

Summary

Affiliation: Harvard University
Country: USA

Publications

  1. ncbi request reprint Fetal liver myelopoiesis occurs through distinct, prospectively isolatable progenitor subsets
    D Traver
    Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Blood 98:627-35. 2001
  2. ncbi request reprint A clonogenic common myeloid progenitor that gives rise to all myeloid lineages
    K Akashi
    Department of Pathology, Stanford University School of Medicine, California 94305, USA
    Nature 404:193-7. 2000
  3. ncbi request reprint Dendritic cell potentials of early lymphoid and myeloid progenitors
    M G Manz
    Department of Pathology, Stanford University School of Medicine, 279 Campus Dr, Stanford, CA 94305 5428, USA
    Blood 97:3333-41. 2001
  4. ncbi request reprint Dendritic cell development from common myeloid progenitors
    M G Manz
    Department of Pathology and Developmental Biology, B261 Beckman Center, Stanford University School of Medicine, 279 Campus Drive, Stanford, California 94305 5428, USA
    Ann N Y Acad Sci 938:167-73; discussion 173-4. 2001
  5. ncbi request reprint The Hox cofactor and proto-oncogene Pbx1 is required for maintenance of definitive hematopoiesis in the fetal liver
    J F DiMartino
    Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Blood 98:618-26. 2001
  6. ncbi request reprint Lymphoid development from hematopoietic stem cells
    K Akashi
    Department of Pathology, Stanford University School of Medicine, California 94305, USA
    Int J Hematol 69:217-26. 1999
  7. ncbi request reprint Mice defective in two apoptosis pathways in the myeloid lineage develop acute myeloblastic leukemia
    D Traver
    Department of Pathology, Stanford University School of Medicine, California 94305, USA
    Immunity 9:47-57. 1998

Collaborators

Detail Information

Publications7

  1. ncbi request reprint Fetal liver myelopoiesis occurs through distinct, prospectively isolatable progenitor subsets
    D Traver
    Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Blood 98:627-35. 2001
    ..Blood. 2001;98:627-635)..
  2. ncbi request reprint A clonogenic common myeloid progenitor that gives rise to all myeloid lineages
    K Akashi
    Department of Pathology, Stanford University School of Medicine, California 94305, USA
    Nature 404:193-7. 2000
    ....
  3. ncbi request reprint Dendritic cell potentials of early lymphoid and myeloid progenitors
    M G Manz
    Department of Pathology, Stanford University School of Medicine, 279 Campus Dr, Stanford, CA 94305 5428, USA
    Blood 97:3333-41. 2001
    ..Blood. 2001;97:3333-3341)..
  4. ncbi request reprint Dendritic cell development from common myeloid progenitors
    M G Manz
    Department of Pathology and Developmental Biology, B261 Beckman Center, Stanford University School of Medicine, 279 Campus Drive, Stanford, California 94305 5428, USA
    Ann N Y Acad Sci 938:167-73; discussion 173-4. 2001
    ..On the basis of transplantation studies, it seems likely that most of the DCs in secondary lymphoid organs and a substantial fraction of thymic DCs are myeloid-derived...
  5. ncbi request reprint The Hox cofactor and proto-oncogene Pbx1 is required for maintenance of definitive hematopoiesis in the fetal liver
    J F DiMartino
    Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305, USA
    Blood 98:618-26. 2001
    ..Thus, Pbx1 is required for the maintenance, but not the initiation, of definitive hematopoiesis and contributes to the mitotic amplifications of progenitor subsets through which mature erythrocytes are generated. (Blood. 2001;98:618-626)..
  6. ncbi request reprint Lymphoid development from hematopoietic stem cells
    K Akashi
    Department of Pathology, Stanford University School of Medicine, California 94305, USA
    Int J Hematol 69:217-26. 1999
    ..Thus, common lymphoid progenitors exist in early hematopoiesis, and expression of the IL-7R is a critical step in the initiation of lymphoid development from HSC...
  7. ncbi request reprint Mice defective in two apoptosis pathways in the myeloid lineage develop acute myeloblastic leukemia
    D Traver
    Department of Pathology, Stanford University School of Medicine, California 94305, USA
    Immunity 9:47-57. 1998
    ..Taken together, these data suggest that Fas has a novel role in the regulation of myelopoiesis and that Fas may act as a tumor suppressor to control leukemogenic transformation in myeloid progenitor cells...