Ruth Serra-Moreno

Summary

Affiliation: Harvard University
Country: USA

Publications

  1. pmc Tetherin/BST-2 antagonism by Nef depends on a direct physical interaction between Nef and tetherin, and on clathrin-mediated endocytosis
    Ruth Serra-Moreno
    Division of Microbiology, New England Primate Research Center, Harvard Medical School, Southborough, Massachusetts, United States of America
    PLoS Pathog 9:e1003487. 2013
  2. pmc Compensatory changes in the cytoplasmic tail of gp41 confer resistance to tetherin/BST-2 in a pathogenic nef-deleted SIV
    Ruth Serra-Moreno
    Department of Microbiology and Molecular Genetics, Harvard Medical School, New England Primate Research Center, Southborough, MA 01772 9102, USA
    Cell Host Microbe 9:46-57. 2011
  3. pmc BST-2/tetherin: a new component of the innate immune response to enveloped viruses
    David T Evans
    Department of Microbiology and Molecular Genetics, Harvard Medical School, New England Primate Research Center, One Pine Hill Drives, Southborough, MA 01772, USA
    Trends Microbiol 18:388-96. 2010
  4. ncbi request reprint Adaptation of human and simian immunodeficiency viruses for resistance to tetherin/BST-2
    Ruth Serra-Moreno
    New England Primate Research Center, Harvard Medical School, Southborough, MA 01772, USA
    Curr HIV Res 10:277-82. 2012
  5. pmc Species-specific activity of SIV Nef and HIV-1 Vpu in overcoming restriction by tetherin/BST2
    Bin Jia
    Department of Microbiology and Molecular Genetics, Harvard Medical School, New England Primate Research Center, Southborough, MA, USA
    PLoS Pathog 5:e1000429. 2009

Detail Information

Publications5

  1. pmc Tetherin/BST-2 antagonism by Nef depends on a direct physical interaction between Nef and tetherin, and on clathrin-mediated endocytosis
    Ruth Serra-Moreno
    Division of Microbiology, New England Primate Research Center, Harvard Medical School, Southborough, Massachusetts, United States of America
    PLoS Pathog 9:e1003487. 2013
    ....
  2. pmc Compensatory changes in the cytoplasmic tail of gp41 confer resistance to tetherin/BST-2 in a pathogenic nef-deleted SIV
    Ruth Serra-Moreno
    Department of Microbiology and Molecular Genetics, Harvard Medical School, New England Primate Research Center, Southborough, MA 01772 9102, USA
    Cell Host Microbe 9:46-57. 2011
    ..These results are consistent with HIV-2 Env antagonism of human tetherin and suggest that the ability to oppose tetherin is important for lentiviral pathogenesis...
  3. pmc BST-2/tetherin: a new component of the innate immune response to enveloped viruses
    David T Evans
    Department of Microbiology and Molecular Genetics, Harvard Medical School, New England Primate Research Center, One Pine Hill Drives, Southborough, MA 01772, USA
    Trends Microbiol 18:388-96. 2010
    ..Here, we review this new host-pathogen relationship and discuss its impact on the evolution of primate lentiviruses and the origins of the HIV pandemic...
  4. ncbi request reprint Adaptation of human and simian immunodeficiency viruses for resistance to tetherin/BST-2
    Ruth Serra-Moreno
    New England Primate Research Center, Harvard Medical School, Southborough, MA 01772, USA
    Curr HIV Res 10:277-82. 2012
    ....
  5. pmc Species-specific activity of SIV Nef and HIV-1 Vpu in overcoming restriction by tetherin/BST2
    Bin Jia
    Department of Microbiology and Molecular Genetics, Harvard Medical School, New England Primate Research Center, Southborough, MA, USA
    PLoS Pathog 5:e1000429. 2009
    ....