Elise B Robinson

Summary

Affiliation: Harvard University
Country: USA

Publications

  1. pmc Stability of autistic traits in the general population: further evidence for a continuum of impairment
    Elise B Robinson
    Harvard School of Public Health, Boston, MA, USA
    J Am Acad Child Adolesc Psychiatry 50:376-84. 2011
  2. pmc A multivariate twin study of autistic traits in 12-year-olds: testing the fractionable autism triad hypothesis
    Elise B Robinson
    Department of Epidemiology, Harvard School of Public Health, 185 Cambridge St, Boston, MA 02114, USA
    Behav Genet 42:245-55. 2012
  3. pmc Psychosocial predictors of lifestyle management in adults with epilepsy
    Elise Robinson
    Department of Society Human Development and Health, Harvard University School of Public Health, 677 Huntington Avenue, Boston, MA 02115, USA
    Epilepsy Behav 13:523-8. 2008
  4. pmc Brief report: no association between parental age and extreme social-communicative autistic traits in the general population
    Elise B Robinson
    Department of Epidemiology, Harvard School of Public Health, Simches Research Building, Boston, MA 02114, USA
    J Autism Dev Disord 41:1733-7. 2011
  5. pmc Evidence that autistic traits show the same etiology in the general population and at the quantitative extremes (5%, 2.5%, and 1%)
    Elise B Robinson
    Departments of Epidemiology, and Society, Harvard School of Public Health, Boston, Massachusetts 02114, USA
    Arch Gen Psychiatry 68:1113-21. 2011
  6. pmc Examining and interpreting the female protective effect against autistic behavior
    Elise B Robinson
    Analytic and Translational Genetics Unit, Massachusetts General Hospital, Boston, MA 02114, USA
    Proc Natl Acad Sci U S A 110:5258-62. 2013
  7. doi request reprint Bipolar polygenic loading and bipolar spectrum features in major depressive disorder
    Anna Wiste
    Psychiatric and Neurodevelopmental Genetics Unit, Center for Human Genetic Research, Boston, MA, USA Center for Experimental Drugs and Diagnostics, Department of Psychiatry, Boston, MA, USA
    Bipolar Disord 16:608-16. 2014
  8. doi request reprint A framework for the interpretation of de novo mutation in human disease
    Kaitlin E Samocha
    1 Analytic and Translational Genetics Unit, Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA 2 Program in Medical and Population Genetics, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA 3 Stanley Center for Psychiatric Research, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA 4 Program in Genetics and Genomics, Biological and Biomedical Sciences, Harvard Medical School, Boston, Massachusetts, USA
    Nat Genet 46:944-50. 2014
  9. pmc Bringing a developmental perspective to anxiety genetics
    Lauren M McGrath
    Massachusetts General Hospital, Boston, MA 02114, USA
    Dev Psychopathol 24:1179-93. 2012

Collaborators

Detail Information

Publications9

  1. pmc Stability of autistic traits in the general population: further evidence for a continuum of impairment
    Elise B Robinson
    Harvard School of Public Health, Boston, MA, USA
    J Am Acad Child Adolesc Psychiatry 50:376-84. 2011
    ..This study investigated the developmental course of autistic traits in a nationally representative sample of subjects 7 to 13 years of age...
  2. pmc A multivariate twin study of autistic traits in 12-year-olds: testing the fractionable autism triad hypothesis
    Elise B Robinson
    Department of Epidemiology, Harvard School of Public Health, 185 Cambridge St, Boston, MA 02114, USA
    Behav Genet 42:245-55. 2012
    ..40) and the extremes (0.24-0.67), as well as even lower unique environmental correlations, all suggested considerable fractionation of genetic and environmental influences across autistic trait domains, in line with previous findings...
  3. pmc Psychosocial predictors of lifestyle management in adults with epilepsy
    Elise Robinson
    Department of Society Human Development and Health, Harvard University School of Public Health, 677 Huntington Avenue, Boston, MA 02115, USA
    Epilepsy Behav 13:523-8. 2008
    ..Social support was related to self-efficacy. These findings suggest that lifestyle management is influenced by a number of relationships between psychosocial variables, particularly by self-efficacy...
  4. pmc Brief report: no association between parental age and extreme social-communicative autistic traits in the general population
    Elise B Robinson
    Department of Epidemiology, Harvard School of Public Health, Simches Research Building, Boston, MA 02114, USA
    J Autism Dev Disord 41:1733-7. 2011
    ..Neither maternal nor paternal age was associated with extreme scores. These findings suggest that advanced parental age does not confer increased risk for extreme social and communication impairment assessed quantitatively...
  5. pmc Evidence that autistic traits show the same etiology in the general population and at the quantitative extremes (5%, 2.5%, and 1%)
    Elise B Robinson
    Departments of Epidemiology, and Society, Harvard School of Public Health, Boston, Massachusetts 02114, USA
    Arch Gen Psychiatry 68:1113-21. 2011
    ..Genetic factors play an important role in the etiology of both autism spectrum disorders and autistic traits. However, little is known about the etiologic consistency of autistic traits across levels of severity...
  6. pmc Examining and interpreting the female protective effect against autistic behavior
    Elise B Robinson
    Analytic and Translational Genetics Unit, Massachusetts General Hospital, Boston, MA 02114, USA
    Proc Natl Acad Sci U S A 110:5258-62. 2013
    ..It provides empirical support for the hypothesis of a female protective effect against autistic behavior and can be used to inform and interpret future gene finding efforts in autism spectrum disorders...
  7. doi request reprint Bipolar polygenic loading and bipolar spectrum features in major depressive disorder
    Anna Wiste
    Psychiatric and Neurodevelopmental Genetics Unit, Center for Human Genetic Research, Boston, MA, USA Center for Experimental Drugs and Diagnostics, Department of Psychiatry, Boston, MA, USA
    Bipolar Disord 16:608-16. 2014
    ..Family and genetic studies indicate overlapping liability for major depressive disorder and bipolar disorder. The purpose of the present study was to determine whether this shared genetic liability influences clinical presentation...
  8. doi request reprint A framework for the interpretation of de novo mutation in human disease
    Kaitlin E Samocha
    1 Analytic and Translational Genetics Unit, Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA 2 Program in Medical and Population Genetics, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA 3 Stanley Center for Psychiatric Research, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA 4 Program in Genetics and Genomics, Biological and Biomedical Sciences, Harvard Medical School, Boston, Massachusetts, USA
    Nat Genet 46:944-50. 2014
    ..We also used our model to identify ∼1,000 genes that are significantly lacking in functional coding variation in non-ASD samples and are enriched for de novo loss-of-function mutations identified in ASD cases. ..
  9. pmc Bringing a developmental perspective to anxiety genetics
    Lauren M McGrath
    Massachusetts General Hospital, Boston, MA 02114, USA
    Dev Psychopathol 24:1179-93. 2012
    ..Expanding the range of phenotypic approaches will be important for advancing studies of the genetic architecture of anxiety disorders...