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Genomes and GenesSpecies | Hannah C KinneySummaryAffiliation: Harvard University Country: USA Publications
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Publications
Serotonergic brainstem abnormalities in Northern Plains Indians with the sudden infant death syndromeHannah C Kinney
Department of Pathology, Children s Hospital and Harvard Medical School, Boston, Massachusetts 02115, USA
J Neuropathol Exp Neurol 62:1178-91. 2003..011) and alcohol (p = 0.075), during the periconceptional period or throughout pregnancy. Prenatal exposure to cigarette smoke and/or alcohol may contribute to abnormal fetal medullary 5-HT development in SIDS infants...
Late development of the GABAergic system in the human cerebral cortex and white matterGang Xu
Department of Pathology, Children s Hospital Boston, Boston, Massachusetts 02115, USA
J Neuropathol Exp Neurol 70:841-58. 2011..This period corresponds to the peak window of vulnerability to perinatal hypoxia-ischemia in which GABAergic neurons are potentially developmentally susceptible, including in the preterm infant...- Myelin abnormalities without oligodendrocyte loss in periventricular leukomalaciaSaraid S Billiards
Department of Pathology, Children s Hospital Boston, Harvard Medical School, Boston, MA 02115, USA
Brain Pathol 18:153-63. 2008..OL migration toward the "core" of injury may occur to replenish OL cell number. This study provides new insight into the cellular basis of the myelin deficits observed in survivors of PVL... - Thalamic damage in periventricular leukomalacia: novel pathologic observations relevant to cognitive deficits in survivors of prematurityPoonam Ligam
Department of Pathology, Harvard Medical School, Boston, Massachusetts 02115, USA
Pediatr Res 65:524-9. 2009..01), with substantial involvement of the mediodorsal, and reticular nuclei in PVL. The prevention of thalamic damage may be required for the eradication of defects in survivors with PVL... - The effect of maternal smoking and drinking during pregnancy upon (3)H-nicotine receptor brainstem binding in infants dying of the sudden infant death syndrome: initial observations in a high risk populationJhodie R Duncan
Department of Pathology, Children s Hospital and Harvard Medical School, Boston, MA 02115, USA
Brain Pathol 18:21-31. 2008..Future studies are needed to establish the role of adverse prenatal exposures in altered brainstem neurochemistry in SIDS... - The serotonergic anatomy of the developing human medulla oblongata: implications for pediatric disorders of homeostasisHannah C Kinney
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, MA 02115, United States
J Chem Neuroanat 41:182-99. 2011..The delineation of the development and organization of the human caudal 5-HT system provides the critical foundation for the neuropathologic elucidation of its disorders directly in the human brain... - Lack of association of the serotonin transporter polymorphism with the sudden infant death syndrome in the San Diego DatasetDavid S Paterson
Department of Pathology, Children s Hospital Boston, Harvard Medical School, Boston, Massachusetts 02115, USA
Pediatr Res 68:409-13. 2010..These observations do not support previous findings that the L allele and/or LL genotype of the 5-HTTLPR are associated with SIDS... - The cerebral cortex overlying periventricular leukomalacia: analysis of pyramidal neuronsSarah E Andiman
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, Mass, USA
Brain Pathol 20:803-14. 2010..This study underscores the potential role of secondary cortical injury in the encephalopathy of prematurity... - Brainstem serotonergic deficiency in sudden infant death syndromeJhodie R Duncan
Department of Pathology, Children s Hospital Boston, 300 Longwood Ave, Boston, MA 02115, USA
JAMA 303:430-7. 2010..Abnormalities of serotonin (5-hydroxytryptamine [5-HT]) receptor binding in regions of the medulla oblongata involved in this control have been reported in infants dying from SIDS... - Glutamate transporter EAAT2 expression is up-regulated in reactive astrocytes in human periventricular leukomalaciaTara M Desilva
Department of Neurology, Children s Hospital and Harvard Medical School, Boston, Massachusetts 02115, USA
J Comp Neurol 508:238-48. 2008..The previously unrecognized up-regulation of EAAT2 in reactive astrocytes and its presence in macrophages in PVL reported here may reflect a response to either hypoxic-ischemic injury or inflammation... - Neuroanatomic relationships between the GABAergic and serotonergic systems in the developing human medullaKevin G Broadbelt
Department of Pathology, Children s Hospital Boston, Boston, MA 02115, USA
Auton Neurosci 154:30-41. 2010..The developmental profile of GABAergic markers changed dramatically relative to the 5-HT markers. These data provide baseline information for medullary studies of human pediatric disorders, such as sudden infant death syndrome... - Serotonergic and glutamatergic neurons at the ventral medullary surface of the human infant: Observations relevant to central chemosensitivity in early human lifeDavid S Paterson
Department of Pathology, Enders 1111, Children s Hospital, 300 Longwood Avenue, Boston, MA 02115, United States Harvard Medical School, Boston, MA 02115, USA
Auton Neurosci 124:112-24. 2006..These data are important towards delineating the role of the human Arc in modulation of homeostasis, and its dysfunction in brainstem-associated pathologies such as the sudden infant death syndrome... - Multiple serotonergic brainstem abnormalities in sudden infant death syndromeDavid S Paterson
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, MA, USA
JAMA 296:2124-32. 2006..Previously, abnormalities in 5-HT receptor binding in the medullae of infants dying from sudden infant death syndrome (SIDS) were identified, suggesting that medullary 5-HT dysfunction may be responsible for a subset of SIDS cases... - Developmental lag in superoxide dismutases relative to other antioxidant enzymes in premyelinated human telencephalic white matterRebecca D Folkerth
From Departments of Pathology Neuropathology, Children s Hospital, Boston, USA
J Neuropathol Exp Neurol 63:990-9. 2004..The previously unrecognized dissociation between the expression of the SODs and that of catalase and GPx in the fetal period has potential implications for future antioxidant therapy in PVL... - Decreased GABAA receptor binding in the medullary serotonergic system in the sudden infant death syndromeKevin G Broadbelt
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, MA 02115, USA
J Neuropathol Exp Neurol 70:799-810. 2011..026). These data suggest that medullary GABAA receptors are abnormal in SIDS infants and that SIDS is a complex disorder of a homeostatic network in the medulla that involves deficits of the GABAergic and 5-HT systems... - Brainstem deficiency of the 14-3-3 regulator of serotonin synthesis: a proteomics analysis in the sudden infant death syndromeKevin G Broadbelt
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, Massachusetts 02115, USA
Mol Cell Proteomics 11:M111.009530. 2012..These data suggest a potential molecular defect in SIDS related to TPH2 regulation, as 14-3-3 is critical in this process... - The glutamate transporter EAAT2 is transiently expressed in developing human cerebral white matterTara M Desilva
Neurobiology Program, Children s Hospital Boston, Department of Neurology, Harvard Medical School, Boston, Massachusetts 02115, USA
J Comp Neurol 501:879-90. 2007.... 
Brainstem mechanisms underlying the sudden infant death syndrome: evidence from human pathologic studiesHannah C Kinney
Department of Pathology, Children s Hospital Boston and Harvard Medical School, 300 Longwood Avenue, Boston, MA 02115, USA
Dev Psychobiol 51:223-33. 2009..The possible fetal origin of brainstem defects in SIDS is reviewed, including evidence for adverse effects of prenatal exposure to maternal cigarette smoking and alcohol upon the postnatal development of human brainstem 5-HT pathways...- Diffuse axonal injury in periventricular leukomalacia as determined by apoptotic marker fractinRobin L Haynes
Department of Pathology, Children s Hospital and Harvard Medical School, Boston, Massachusetts 02115, USA
Pediatr Res 63:656-61. 2008..We conclude that diffuse axonal injury, as determined by apoptotic marker fractin, occurs in PVL and that its cause likely includes primary ischemia and trophic degeneration secondary to corticothalamic neuronal damage... - Sudden death, febrile seizures, and hippocampal and temporal lobe maldevelopment in toddlers: a new entityHannah C Kinney
Department of Pathology Neuropathology, Children s Hospital and Harvard Medical School, Enders Building, Room 1112, 300 Longwood Avenue, Boston, MA, 02115, USA
Pediatr Dev Pathol 12:455-63. 2009..This study mandates further research into the potential link between simple febrile seizures and death... - Lipid peroxidation during human cerebral myelinationRobin L Haynes
Department of Pathology, Children s Hospital Boston, Massachusetts 02115, USA
J Neuropathol Exp Neurol 65:894-904. 2006.... - The development of nicotinic receptors in the human medulla oblongata: inter-relationship with the serotonergic systemJhodie R Duncan
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, Massachusetts 02115, USA
Auton Neurosci 144:61-75. 2008..This study indicates parallel dynamic and complex changes in the medullary nicotinic and 5-HT systems throughout early life, i.e., the period of risk for SIDS... - Development of microglia in the cerebral white matter of the human fetus and infantSaraid S Billiards
Department of Pathology, Children s Hospital Boston and Harvard Medical School, MA 02115, USA
J Comp Neurol 497:199-208. 2006..j... - Subtle autonomic and respiratory dysfunction in sudden infant death syndrome associated with serotonergic brainstem abnormalities: a case reportHannah C Kinney
Department of Pathology, Children s Hospital and Harvard Medical School, Boston, Massachusetts 02115, USA
J Neuropathol Exp Neurol 64:689-94. 2005..Replication of these findings in a large population may lead to the development of predictive cardiorespiratory assessment tools for future screening to identify infants with medullary 5-HT abnormalities and SIDS risk... - Neuron deficit in the white matter and subplate in periventricular leukomalaciaHannah C Kinney
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, MA 02115, USA
Ann Neurol 71:397-406. 2012..We tested the hypothesis that the overall density of neurons in the white matter and subplate region is significantly lower in PVL cases compared to non-PVL controls... - Nitrosative stress and inducible nitric oxide synthase expression in periventricular leukomalaciaRobin L Haynes
Departments of Pathology, Children s Hospital Boston, Boston, MA 02115, USA
Acta Neuropathol 118:391-9. 2009..This study supports an important role for iNOS-induced nitrosative stress in the reactive/inflammatory component of PVL... - The development of the medullary serotonergic system in early human lifeHannah C Kinney
Department of Pathology, Children s Hospital and Harvard Medical School, Boston, MA 02115, USA
Auton Neurosci 132:81-102. 2007..Thus, protracted changes occur from the prenatal period through infancy. These data provide a foundation for 5-HT neuronal analysis in pediatric brainstem disorders, as proposed in the sudden infant death syndrome... - Prenatal nicotine-exposure alters fetal autonomic activity and medullary neurotransmitter receptors: implications for sudden infant death syndromeJhodie R Duncan
Dept of Pathology, Enders 1112 1, Children s Hospital Boston, 300 Longwood Ave, Boston, MA 02115, USA
J Appl Physiol 107:1579-90. 2009..Thus increased risk for SIDS due to maternal smoking may be partly related to the effects of nicotine on 5-HT and/or nicotinic receptors... - Gray matter injury associated with periventricular leukomalacia in the premature infantChristopher R Pierson
Department of Pathology, Children s Hospital Boston, Boston, MA, USA
Acta Neuropathol 114:619-31. 2007.... - Serotonin-related FEV gene variant in the sudden infant death syndrome is a common polymorphism in the African-American populationKevin G Broadbelt
Department of Pathology, Enders Building Room 1111, Children s Hospital Boston, 300 Longwood Avenue, Boston, MA 02115, USA
Pediatr Res 66:631-5. 2009..128-(191_192)dupA). The polymorphism seems to be a common, likely nonpathogenic, variant in the African-American population... - Interferon-gamma expression in periventricular leukomalacia in the human brainRebecca D Folkerth
Department of Pathology Neuropathology, Children s Hospital and Harvard Medical School, Boston, MA, USA
Brain Pathol 14:265-74. 2004..In PVL, IFN-gamma produced by macrophages and reactive astrocytes may play a role in cytokine-induced toxicity to premyelinating oligodendrocytes as part of a cytokine response stimulated by ischemia and/or infection... - Is the late preterm infant more vulnerable to gray matter injury than the term infant?Saraid S Billiards
Department of Pathology, Enders Building, Room 1109, Children s Hospital Boston, Boston, MA 02115, USA
Clin Perinatol 33:915-33; abstract x-xi. 2006..Future research involving closer examination of the late preterm period is essential to provide a greater understanding of the neuronal vulnerability in the face of hypoxic-ischemic injury... - Serotonin transporter abnormality in the dorsal motor nucleus of the vagus in Rett syndrome: potential implications for clinical autonomic dysfunctionDavid S Paterson
Department of Pathology, Children s Hospital and Harvard Medical School, Boston, Massachusetts, USA
J Neuropathol Exp Neurol 64:1018-27. 2005..These data suggest hypotheses concerning 5-HT modulation of vagal function for testing in MeCP2 knockout mice to understand mechanisms underlying autonomic dysfunction in patients with Rett syndrome... - Interleukin-6 and the serotonergic system of the medulla oblongata in the sudden infant death syndromeIngvar Jon Rognum
Department of Pathology, Children s Hospital Boston, Harvard Medical School, Boston, MA, USA
Acta Neuropathol 118:519-30. 2009..Aberrant interactions between IL-6 and the arcuate nucleus may contribute to impaired responses to hypercapnia generated by infection (hyper-metabolism) combined with rebreathing... - Axonal development in the cerebral white matter of the human fetus and infantRobin L Haynes
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, Massachusetts 02115, USA
J Comp Neurol 484:156-67. 2005..These data suggest that immature axons are susceptible to damage in PVL and that the timing of axonal maturation must be considered toward establishing its pathology relative to the oligodendrocyte/myelin/axonal unit... - Potential neuronal repair in cerebral white matter injury in the human neonateRobin L Haynes
Departments of Pathology, Children s Hospital Boston, Harvard Medical School, Boston, Massachusetts 02115, USA
Pediatr Res 69:62-7. 2011..e. 35-42 postconceptional weeks. These data provide the first evidence suggestive of an attempt at neuronal repair or regeneration in human neonatal white matter injury... - The developing oligodendrocyte: key cellular target in brain injury in the premature infantJoseph J Volpe
Department of Neurology, Children s Hospital and Harvard Medical School, Boston, MA 02115, USA
Int J Dev Neurosci 29:423-40. 2011..The critical next step, i.e., clinical trials in the living infant, is now on the horizon... - Inheritance of febrile seizures in sudden unexplained death in toddlersIngrid A Holm
Division of Genetics, Program in Genomics, and Manton Center for Orphan Disease Research, Children s Hospital Boston and Harvard Medical School, Boston, Massachusetts 02115, USA
Pediatr Neurol 46:235-9. 2012..Future studies should develop markers (including genetic) to identify which patients with febrile seizures are at risk for sudden unexplained death in childhood, and to provide guidance for families and physicians... - Sudden and unexpected death in early life: proceedings of a symposium in honor of Dr. Henry F. KrousHannah C Kinney
Department of Pathology, Boston Children s Hospital and Harvard Medical School, 300 Longwood Avenue, Enders Building 1112, Boston, MA 02115, USA
Forensic Sci Med Pathol 8:414-25. 2012..Krous' far-reaching influence upon SIDS and SUDC research... - Expression of EAAT2 in neurons and protoplasmic astrocytes during human cortical developmentTara M Desilva
Department of Neurology and the FM Kirby Neurobiology Center, Children s Hospital Boston and Harvard Medical School, Boston, Massachusetts 02115, USA
J Comp Neurol 520:3912-32. 2012..In addition, by studying the expression of EAAT1 and EAAT2 glutamate transporters, it was possible to document the development of protoplasmic astrocytes... - Neuroanatomic connectivity of the human ascending arousal system critical to consciousness and its disordersBrian L Edlow
Department of Neurology, Brigham and Women s Hospital, 75 Francis St, Boston, MA 02115, USA
J Neuropathol Exp Neurol 71:531-46. 2012.... - Hypoxic-ischemic brain injury in infants with congenital heart disease dying after cardiac surgeryHannah C Kinney
Department of Pathology, Children s Hospital and Harvard Medical School, Enders 1112, 300 Longwood Avenue, Boston, MA 02115, USA
Acta Neuropathol 110:563-78. 2005.... - The near-term (late preterm) human brain and risk for periventricular leukomalacia: a reviewHannah C Kinney
Department of Pathology, Children s Hospital Boston and Harvard Medical School, MA 02115, USA
Semin Perinatol 30:81-8. 2006.... - Human myelination and perinatal white matter disordersHannah C Kinney
Department of Neuroscience, Harvard Medical School/Children's Hospital, Enders Building, Room 206, 300 Longwood Avenue, Boston, MA 02115, USA
J Neurol Sci 228:190-2. 2005 - The brainstem and serotonin in the sudden infant death syndromeHannah C Kinney
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, MA 02115, USA
Annu Rev Pathol 4:517-50. 2009..These emerging data suggest an important underlying mechanism in SIDS that may help lead to identification of infants at risk and specific interventions to prevent death... - Sudden death in toddlers associated with developmental abnormalities of the hippocampus: a report of five casesHannah C Kinney
Department of Pathology, Children s Hospital and Harvard Medical School, Boston, MA, USA
Pediatr Dev Pathol 10:208-23. 2007..Future studies are needed to confirm these initial findings and to define the putative links between sudden death, hippocampal anomalies, and febrile seizures in toddlers... - Arrested oligodendrocyte lineage progression during human cerebral white matter development: dissociation between the timing of progenitor differentiation and myelinogenesisStephen A Back
Department of Neurology, Children's Hospital and Harvard Medical School, Boston, Massachusetts, USA
J Neuropathol Exp Neurol 61:197-211. 2002.... - Differential development of 5-HT receptor and the serotonin transporter binding in the human infant medullaDavid S Paterson
Department of Neurology, Children s Hospital and Harvard Medical School, Boston, Massachusetts 02115, USA
J Comp Neurol 472:221-31. 2004..This study provides important baseline data that serve as a foundation for future work in pediatric 5-HT brainstem disorders, including sudden infant death syndrome... - Sudden death in toddlers with viral meningitis, massive cerebral edema, and neurogenic pulmonary edema and hemorrhage: report of two casesHenry F Krous
Department of Pathology, Rady Children s Hospital San Diego, San Diego, CA, USA
Pediatr Dev Pathol 10:463-9. 2007..They also underscore the importance of a comprehensive autopsy, including detailed neuropathologic examination and viral testing, in determining of the cause of unexpected death in toddlers... - Reprint of "The developing oligodendrocyte: key cellular target in brain injury in the premature infant"Joseph J Volpe
Department of Neurology, Children s Hospital and Harvard Medical School, Boston, MA 02115, USA
Int J Dev Neurosci 29:565-82. 2011..The critical next step, i.e., clinical trials in the living infant, is now on the horizon... - Abnormalities of the brainstem serotonergic system in the sudden infant death syndrome: a reviewHannah C Kinney
Department of Pathology, Children's Hospital Boston, Enders Building 1112, 300 Longwood Avenue, Boston, Massachusetts 02115, USA
Pediatr Dev Pathol 8:507-24. 2005 - The encephalopathy of prematurity: one pediatric neuropathologist's perspectiveHannah C Kinney
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, MA 02115, USA
Semin Pediatr Neurol 16:179-90. 2009.... - Nitrosative and oxidative injury to premyelinating oligodendrocytes in periventricular leukomalaciaRobin L Haynes
Department of Neurology, Children s Hospital and Harvard Medical School, Boston, Massachusetts 02115, USA
J Neuropathol Exp Neurol 62:441-50. 2003..Agents that prevent nitrosative and oxidative stress may play a key role in ameliorating PVL in premature infants in the intensive care nursery... - Sudden Infant Death Syndrome: increased carotid-body dopamine and noradrenaline contentHannah C Kinney
Department of Pathology, Children's Hospital Boston, 300 Longwood Avenue, Enders 1112, Boston, MA 02115, USA
Pediatr Dev Pathol 8:258-67. 2005 - Anatomic relationships of the human nucleus of the solitary tract in the medulla oblongata: a DiI labeling studyNatasa Zec
Department of Neurology, Children's Hospital and Harvard Medical School, 300 Longwood Avenue, Enders Building 206, Boston, MA 02115, USA
Auton Neurosci 105:131-44. 2003.... - Structural abnormalities in the brainstem and cerebellum in congenital central hypoventilation syndrome: commentary on the article by Kumar et al. on page 275Hannah C Kinney
Department of Pathology, Children's Hospital and Harvard Medical School, Boston, Massachusetts 02115, USA
Pediatr Res 64:226-7. 2008 - Modeling the encephalopathy of prematurity in animals: the important role of translational researchHannah C Kinney
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, MA 02115, USA
Neurol Res Int 2012:295389. 2012.... - Neuropathology associated with stillbirthMarjorie R Grafe
Department of Pathology, University of Texas Medical Branch, Galveston 77555-0609, USA
Semin Perinatol 26:83-8. 2002..The fetal brain may be selectively vulnerable to various insults at specific stages of development... - Novel neuropathologic findings in the Haddad syndromeNestor D Tomycz
Department of Pathology, Children s Hospital Boston and Harvard Medical School, Boston, MA, USA
Acta Neuropathol 119:261-9. 2010..This study implicates novel brain findings in Haddad syndrome mimicking those in murine Phox2b null mutants. This case suggests that abnormalities occur in CCHS in a network of sites critical to chemoreception... - Comparative anatomical assessment of the piglet as a model for the developing human medullary serotonergic systemMary M Niblock
Department of Physiology, Dartmouth Medical School, 1 Medical Center Drive, Lebanon, NH 03756, USA
Brain Res Brain Res Rev 50:169-83. 2005..Collectively, when certain species differences are considered, these data support the use of the piglet as a model for the human infant medullary 5-HT system... - Risk factors for sudden infant death syndrome among northern plains IndiansSolomon Iyasu
Division of Reproductive Health, National Center for Chronic Disease Prevention and Health Promotion, Centers for Disease Control and Prevention, Atlanta, GA, USA
JAMA 288:2717-23. 2002..Sudden infant death syndrome (SIDS) is a leading cause of postneonatal mortality among American Indians, a group whose infant death rate is consistently above the US national average... - The development of the medullary serotonergic system in the pigletMary M Niblock
Department of Physiology, Dartmouth Medical School, 1 Medical Center Drive, Lebanon, NH 03753, USA
Auton Neurosci 110:65-80. 2004..Therefore, the first 30 days of life represent a critical period in the development of the 5-HT system and the homeostatic functions it mediates... - Chronic fluoxetine microdialysis into the medullary raphe nuclei of the rat, but not systemic administration, increases the ventilatory response to CO2Natalie C Taylor
Department of Physiology, Dartmouth Medical Center, Borwell Bldg, Lebanon, NH 03756 000, USA
J Appl Physiol 97:1763-73. 2004..Chronic fluoxetine treatment in the medullary raphe increases the ventilatory response to hypercapnia in an unanesthetized rat model, an effect that may be due to facilitation of chemosensitive serotonergic neurons... - Mechanism of JCV entry into oligodendrocytesSandro Santagata
Science 309:381-2. 2005 - Subacute combined degeneration of the spinal cord in cblC disorder despite treatment with B12Sharon E Smith
Division of Genetics, Children's Hospital Boston, MA 02115, USA
Mol Genet Metab 88:138-45. 2006..This is especially important now that the presymptomatic detection of cblC disorder is possible through the expansion of newborn screening... - Sudden unexpected death in childhood associated with cardiac rhabdomyoma, involuting adrenal ganglioneuroma, and megalencephaly: another expression of tuberous sclerosis?Homeyra Masoumi
Rady Children s Hospital and Health Center, San Diego, CA, USA
Pediatr Dev Pathol 10:129-33. 2007..The constellation of her autopsy findings suggested a diagnosis of tuberous sclerosis, for which there are gene defects that can be identified in surviving family members... - Fetal mechanisms in neurodevelopmental disordersSusan L Connors
Department of Neurology and Developmental Medicine, Kennedy Krieger Institute, 707 North Broadway, Baltimore, MD 21205, USA
Pediatr Neurol 38:163-76. 2008..This research emphasizes the diversity of such prenatal mechanisms and the need to expand our understanding of how, when altered, they may lead to disordered development, the signs of which may not appear until long after birth... - The late preterm infant and the control of breathing, sleep, and brainstem development: a reviewRobert A Darnall
Department of Physiology, Dartmouth Hitchcock Medical Center, Lebanon, NH 03756, USA
Clin Perinatol 33:883-914; abstract x. 2006..Research focused on this group of infants will not only further our understanding of brainstem maturation during this high risk period, but will help develop focused plans for their management...