Saravanan Kaliyaperumal

Summary

Affiliation: Harvard University
Country: USA

Publications

  1. ncbi Phosphorylated hMSH6: DNA mismatch versus DNA damage recognition
    Saravanan Kaliyaperumal
    Department of Biochemistry and Cancer Biology, University of Toledo College of Medicine, Toledo, OH 43614, USA
    Mutat Res 706:36-45. 2011
  2. ncbi Prolonged cell cycle response of HeLa cells to low-level alkylation exposure
    Allen G Schroering
    Department of Biochemistry and Cancer Biology, University of Toledo College of Medicine, Toledo, Ohio 43614, USA
    Cancer Res 69:6307-14. 2009
  3. ncbi The ATPase motif in RAD51D is required for resistance to DNA interstrand crosslinking agents and interaction with RAD51C
    Aaron M Gruver
    Department of Physiology and Cardiovascular Genomics, Medical University of Ohio, Toledo, OH 43614 5804, USA
    Mutagenesis 20:433-40. 2005

Collaborators

Detail Information

Publications3

  1. ncbi Phosphorylated hMSH6: DNA mismatch versus DNA damage recognition
    Saravanan Kaliyaperumal
    Department of Biochemistry and Cancer Biology, University of Toledo College of Medicine, Toledo, OH 43614, USA
    Mutat Res 706:36-45. 2011
    ..We hypothesize that both phosphorylation of hMSH6 and total concentration of bound hMutSα are involved in cellular signaling of either DNA mismatch repair or MMR-dependent damage recognition activities...
  2. ncbi Prolonged cell cycle response of HeLa cells to low-level alkylation exposure
    Allen G Schroering
    Department of Biochemistry and Cancer Biology, University of Toledo College of Medicine, Toledo, Ohio 43614, USA
    Cancer Res 69:6307-14. 2009
    ..This information should prove clinically relevant for the future design of enhanced cancer chemotherapeutics...
  3. ncbi The ATPase motif in RAD51D is required for resistance to DNA interstrand crosslinking agents and interaction with RAD51C
    Aaron M Gruver
    Department of Physiology and Cardiovascular Genomics, Medical University of Ohio, Toledo, OH 43614 5804, USA
    Mutagenesis 20:433-40. 2005
    ..These cell-based studies are the first evidence that ATP binding and hydrolysis by RAD51D are required for efficient HR repair of DNA interstrand crosslinks...