Research Topics
Species | Wendy S GarrettSummaryAffiliation: Harvard University Country: USA Publications
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Publications
Enterobacteriaceae act in concert with the gut microbiota to induce spontaneous and maternally transmitted colitisWendy S Garrett
Department of Immunology and Infectious Diseases, Harvard School of Public Health, Boston, MA 02115, USA
Cell Host Microbe 8:292-300. 2010..Cross-fostering experiments indicated a role for these organisms in maternal transmission of disease. Our findings illustrate how gut microbial communities work in concert with specific culturable colitogenic agents to cause IBD...
Homeostasis and inflammation in the intestineWendy S Garrett
Department of Immunology and Infectious Diseases, Harvard School of Public Health, Boston, MA 02115, USA
Cell 140:859-70. 2010..In this Review, we consider the many cellular and molecular methods by which inflammatory responses are regulated to maintain intestinal homeostasis and the disease states that can ensue when this balance is lost...
Colitis-associated colorectal cancer driven by T-bet deficiency in dendritic cellsWendy S Garrett
Department of Immunology and Infectious Diseases, Harvard School of Public Health, Boston, MA 02115, USA
Cancer Cell 16:208-19. 2009..TRUC colitis-associated colorectal cancer resembles the human disease and provides ample opportunity to probe how inflammation drives colorectal cancer development and to test preventative and therapeutic strategies preclinically...
Bifidobacterium animalis subsp. lactis fermented milk product reduces inflammation by altering a niche for colitogenic microbesPatrick Veiga
Harvard School of Public Health, Boston, MA 02115, USA
Proc Natl Acad Sci U S A 107:18132-7. 2010..lactis-containing fermented milk product, and therefore this model provides a framework for evaluating and optimizing probiotic-based functional foods...
T-bet-/- RAG2-/- ulcerative colitis: the role of T-bet as a peacekeeper of host-commensal relationshipsWendy S Garrett
Department of Immunology and Infectious Diseases, Harvard School of Public Health, Boston, MA 02115, USA
Cytokine 48:144-7. 2009..1]. The TRUC (T-bet-/- RAG2-/- ulcerative colitis) model is discussed in the broader context of the adaptive and innate immune mechanisms that regulate host-commensal relationships within the intestine...
Severity of innate immune-mediated colitis is controlled by the cytokine deficiency-induced colitis susceptibility-1 (Cdcs1) locusJoerg Ermann
Department of Immunology and Infectious Diseases, Harvard School of Public Health, Harvard Medical School, Boston, MA 02115, USA
Proc Natl Acad Sci U S A 108:7137-41. 2011..We conclude that the Cdcs1 locus controls colitis severity in T-bet(-/-).Rag2(-/-) mice through innate immune cells...
Current concepts of the intestinal microbiota and the pathogenesis of infectionLeslie H Wardwell
Department of Immunology and Infectious Diseases, Harvard School of Public Health, 665 Huntington Avenue, SPH 1 909, Boston, MA, 02115, USA
Curr Infect Dis Rep 13:28-34. 2011..Key concepts of colonization resistance, host-commensal microbe interaction in immunity, antibiotics and gut bacterial communities, viral-gut bacterial interactions, and evolving methods for studying commensal microbes are explored...
Metagenomic biomarker discovery and explanationNicola Segata
Department of Biostatistics, 677 Huntington Avenue, Harvard School of Public Health, Boston, MA 02115, USA
Genome Biol 12:R60. 2011..We extensively validate our method on several microbiomes and a convenient online interface for the method is provided at http://huttenhower.sph.harvard.edu/lefse/...
Host and gut microbiota symbiotic factors: lessons from inflammatory bowel disease and successful symbiontsSonia A Ballal
Harvard School of Public Health, Boston, MA, USA
Cell Microbiol 13:508-17. 2011..While model symbionts have provided perspective into host-microbial homeostasis, high-throughput approaches are becoming increasingly practical for functionally characterizing the gut microbiota as a community...
Communicable ulcerative colitis induced by T-bet deficiency in the innate immune systemWendy S Garrett
Department of Immunology and Infectious Diseases, Harvard School of Public Health, Boston, MA 02115, USA
Cell 131:33-45. 2007..These findings reveal a novel function for T-bet as a peacekeeper of host-commensal relationships and provide new perspectives on the pathophysiology of IBD...
