Rhonda L Feinbaum

Summary

Affiliation: Harvard University
Country: USA

Publications

  1. pmc Genome-wide identification of Pseudomonas aeruginosa virulence-related genes using a Caenorhabditis elegans infection model
    Rhonda L Feinbaum
    Department of Genetics, Harvard Medical School, and Department of Molecular Biology, Massachusetts General Hospital, Boston, Massachusetts, United States of America
    PLoS Pathog 8:e1002813. 2012
  2. pmc Distinct pathogenesis and host responses during infection of C. elegans by P. aeruginosa and S. aureus
    Javier E Irazoqui
    Program of Developmental Immunology, Department of Pediatrics, Massachusetts General Hospital, Department of Pediatrics, Harvard Medical School, Boston, Massachusetts, United States of America
    PLoS Pathog 6:e1000982. 2010
  3. pmc The evolutionarily conserved mediator subunit MDT-15/MED15 links protective innate immune responses and xenobiotic detoxification
    Read Pukkila-Worley
    Division of Infectious Diseases Massachusetts General Hospital Harvard Medical School Boston, Massachusetts, United States of America Department of Molecular Biology Massachusetts General Hospital Harvard Medical School Boston, Massachusetts, United States of America Department of Genetics Harvard Medical School Boston, Massachusetts, United States of America
    PLoS Pathog 10:e1004143. 2014
  4. pmc Genomic analysis reveals that Pseudomonas aeruginosa virulence is combinatorial
    Daniel G Lee
    Department of Molecular Biology, Massachusetts General Hospital, Cambridge Street, Boston, Massachusetts 02114, USA
    Genome Biol 7:R90. 2006

Collaborators

Detail Information

Publications4

  1. pmc Genome-wide identification of Pseudomonas aeruginosa virulence-related genes using a Caenorhabditis elegans infection model
    Rhonda L Feinbaum
    Department of Genetics, Harvard Medical School, and Department of Molecular Biology, Massachusetts General Hospital, Boston, Massachusetts, United States of America
    PLoS Pathog 8:e1002813. 2012
    ..elegans are biased towards evolutionarily conserved genes...
  2. pmc Distinct pathogenesis and host responses during infection of C. elegans by P. aeruginosa and S. aureus
    Javier E Irazoqui
    Program of Developmental Immunology, Department of Pediatrics, Massachusetts General Hospital, Department of Pediatrics, Harvard Medical School, Boston, Massachusetts, United States of America
    PLoS Pathog 6:e1000982. 2010
    ..aeruginosa nor the S. aureus-triggered response requires canonical TLR signaling, they imply the existence of unidentified mechanisms for pathogen detection in C. elegans, with potentially conserved roles also in mammals...
  3. pmc The evolutionarily conserved mediator subunit MDT-15/MED15 links protective innate immune responses and xenobiotic detoxification
    Read Pukkila-Worley
    Division of Infectious Diseases Massachusetts General Hospital Harvard Medical School Boston, Massachusetts, United States of America Department of Molecular Biology Massachusetts General Hospital Harvard Medical School Boston, Massachusetts, United States of America Department of Genetics Harvard Medical School Boston, Massachusetts, United States of America
    PLoS Pathog 10:e1004143. 2014
    ..These data define a central role for MDT-15/MED15 in the coordination of xenobiotic detoxification and innate immune responses. ..
  4. pmc Genomic analysis reveals that Pseudomonas aeruginosa virulence is combinatorial
    Daniel G Lee
    Department of Molecular Biology, Massachusetts General Hospital, Cambridge Street, Boston, Massachusetts 02114, USA
    Genome Biol 7:R90. 2006
    ..We therefore sequenced a highly virulent strain of P. aeruginosa, PA14, and compared it with a previously sequenced (and less pathogenic) strain, PAO1, to identify novel virulence genes...