Sara Cherry

Summary

Affiliation: Harvard University
Country: USA

Publications

  1. ncbi Entry is a rate-limiting step for viral infection in a Drosophila melanogaster model of pathogenesis
    Sara Cherry
    Department of Genetics, Harvard Medical School, 200 Longwood Avenue, Boston, Massachusetts 02115, USA
    Nat Immunol 5:81-7. 2004
  2. ncbi Genome-wide RNAi screen reveals a specific sensitivity of IRES-containing RNA viruses to host translation inhibition
    Sara Cherry
    Department of Genetics, Harvard Medical School and Howard Hughes Medical Instutite, Boston, Massachusetts 02115, USA
    Genes Dev 19:445-52. 2005
  3. ncbi Temperature-sensitive control of protein activity by conditionally splicing inteins
    Martin P Zeidler
    Department of Genetics, Howard Hughes Medical Institute, Harvard Medical School, 77 Avenue Louis Pasteur, Boston, Massachusetts 02115, USA
    Nat Biotechnol 22:871-6. 2004
  4. ncbi COPI activity coupled with fatty acid biosynthesis is required for viral replication
    Sara Cherry
    University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania, United States of America
    PLoS Pathog 2:e102. 2006
  5. ncbi Host-pathogen interactions in drosophila: new tricks from an old friend
    Sara Cherry
    Department of Microbiology, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania 19104, USA
    Nat Immunol 7:911-7. 2006
  6. ncbi Genomic RNAi screening in Drosophila S2 cells: what have we learned about host-pathogen interactions?
    Sara Cherry
    Department of Microbiology, Penn Genomics Frontiers Institute, University of Pennsylvania, Philadelphia, PA 19104, United States
    Curr Opin Microbiol 11:262-70. 2008

Collaborators

Detail Information

Publications6

  1. ncbi Entry is a rate-limiting step for viral infection in a Drosophila melanogaster model of pathogenesis
    Sara Cherry
    Department of Genetics, Harvard Medical School, 200 Longwood Avenue, Boston, Massachusetts 02115, USA
    Nat Immunol 5:81-7. 2004
    ..Thus, this virus model provides a sensitive and efficient approach for identifying components required for pathogenesis...
  2. ncbi Genome-wide RNAi screen reveals a specific sensitivity of IRES-containing RNA viruses to host translation inhibition
    Sara Cherry
    Department of Genetics, Harvard Medical School and Howard Hughes Medical Instutite, Boston, Massachusetts 02115, USA
    Genes Dev 19:445-52. 2005
    ..Finally, this increased sensitivity to ribosome levels also holds true for poliovirus infection of human cells, demonstrating the generality of these findings...
  3. ncbi Temperature-sensitive control of protein activity by conditionally splicing inteins
    Martin P Zeidler
    Department of Genetics, Howard Hughes Medical Institute, Harvard Medical School, 77 Avenue Louis Pasteur, Boston, Massachusetts 02115, USA
    Nat Biotechnol 22:871-6. 2004
    ....
  4. ncbi COPI activity coupled with fatty acid biosynthesis is required for viral replication
    Sara Cherry
    University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania, United States of America
    PLoS Pathog 2:e102. 2006
    ..Additionally, because these pathways are also limiting in flies and in human cells infected with the related RNA virus poliovirus, they may represent novel targets for antiviral therapies...
  5. ncbi Host-pathogen interactions in drosophila: new tricks from an old friend
    Sara Cherry
    Department of Microbiology, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania 19104, USA
    Nat Immunol 7:911-7. 2006
    ..This review discusses advances in these exciting new areas of drosophila immunity...
  6. ncbi Genomic RNAi screening in Drosophila S2 cells: what have we learned about host-pathogen interactions?
    Sara Cherry
    Department of Microbiology, Penn Genomics Frontiers Institute, University of Pennsylvania, Philadelphia, PA 19104, United States
    Curr Opin Microbiol 11:262-70. 2008
    ..This review will highlight the application of RNAi screening to pathogen-host interactions in Drosophila cells and will reveal some of the lessons learned from this approach...