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Genomes and GenesSpecies | Darrick T BaluSummaryAffiliation: Harvard University Country: USA Publications
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Publications
Neuronal D-serine regulates dendritic architecture in the somatosensory cortexDarrick T Balu
Department of Psychiatry, Harvard Medical School, and Laboratory for Psychiatric and Molecular Neuroscience, McLean Hospital, Belmont, MA 02478, USA
Neurosci Lett 517:77-81. 2012..Moreover, they highlight the glycine modulatory site (GMS) of the NMDAR as a potential target for therapeutic intervention in diseases characterized by synaptic deficits, like schizophrenia...
Enhanced sensitivity of the MRL/MpJ mouse to the neuroplastic and behavioral effects of acute and chronic antidepressant treatmentsDarrick T Balu
Harvard Medical School, Department of Psychiatry, McLean Hospital, Belmont, MA 02478, USA
Exp Clin Psychopharmacol 18:71-7. 2010....
Neuroplasticity signaling pathways linked to the pathophysiology of schizophreniaDarrick T Balu
Department of Psychiatry, Harvard Medical School, Belmont, MA, USA
Neurosci Biobehav Rev 35:848-70. 2011....
The NMDA receptor co-agonists, D-serine and glycine, regulate neuronal dendritic architecture in the somatosensory cortexDarrick T Balu
Department of Psychiatry, Harvard Medical School, Boston, MA 02478, USA
Neurobiol Dis 45:671-82. 2012..Moreover, they suggest that NMDAR hypofunction might, in part, be contributing to the dendritic and synaptic changes observed in schizophrenia and highlight this signaling pathway as a potential target for therapeutic intervention...
Glutamate receptor composition of the post-synaptic density is altered in genetic mouse models of NMDA receptor hypo- and hyperfunctionDarrick T Balu
Department of Psychiatry, Harvard Medical School, Belmont, MA, USA
Brain Res 1392:1-7. 2011..Similar to SR-/- mice, there were no protein changes observed in the PFC. These findings illustrate the complexity of synaptic adaptation to altered NMDAR function...
