Research Topics
| T KitazawaSummaryAffiliation: Georgetown University Country: USA Publications
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Detail Information
Publications
Reconstitution of protein kinase C-induced contractile Ca2+ sensitization in triton X-100-demembranated rabbit arterial smooth muscleT Kitazawa
Department of Physiology and Biophysics, Georgetown University Medical Center, Washington, DC 20007, USA
J Physiol 520:139-52. 1999..7. Our results indicate that PKC requires CPI-17 phosphorylation at Thr-38 but not calponin for reconstitution of the contractile Ca2+ sensitization in the demembranated arterial smooth muscle...
Agonists trigger G protein-mediated activation of the CPI-17 inhibitor phosphoprotein of myosin light chain phosphatase to enhance vascular smooth muscle contractilityT Kitazawa
Department of Physiology and Biophysics, Georgetown University Medical Center, Washington, DC 20007, USA
J Biol Chem 275:9897-900. 2000..The results show that agonists such as histamine signal through phosphorylation of CPI-17 to produce Ca(2+) sensitization of smooth muscle contraction...
Expression of CPI-17 and myosin phosphatase correlates with Ca(2+) sensitivity of protein kinase C-induced contraction in rabbit smooth muscleT P Woodsome
Department of Physiology and Biophysics, Georgetown University Medical Center, Washington, DC 20007, USA
J Physiol 535:553-64. 2001..This implicates CPI-17 inhibition of MLCP as an important component in modulating vascular muscle tone...
Non-genomic mechanism of 17 beta-oestradiol-induced inhibition of contraction in mammalian vascular smooth muscleT Kitazawa
Department of Physiology and Biophysics, Georgetown University School of Medicine, Washington DC 20007, USA
J Physiol 499:497-511. 1997....
