Research Topics
| Baochong GaoSummaryCountry: USA Publications
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Detail Information
Publications
Endogenous ligands of Toll-like receptorsMin Fu Tsan
Office of Research Oversight, Department of Veterans Affairs, 50 Irving Street, NW, Washington, DC 20422, USA
J Leukoc Biol 76:514-9. 2004....
The heat sensitivity of cytokine-inducing effect of lipopolysaccharideBaochong Gao
Research Service, VA Medical Center, Washington, DC, USA
J Leukoc Biol 80:359-66. 2006....
Induction of cytokines by heat shock proteins and endotoxin in murine macrophagesBaochong Gao
Institute for Clinical Research, Washington, DC 20422, USA
Biochem Biophys Res Commun 317:1149-54. 2004..These data suggest that rhHsp60 and rhHsp70 do not activate cytokine genes in murine macrophages...
Recombinant human heat shock protein 60 does not induce the release of tumor necrosis factor alpha from murine macrophagesBaochong Gao
Institute for Clinical Research, Washington, D C 20422, USA
J Biol Chem 278:22523-9. 2003..Approximately 50% of the observed TNF-alpha-inducing activity in the rhHSP60-2 preparation is due to LPS contamination, whereas the rest of the activity was due to the contamination of LPS-associated molecule(s)...
Endotoxin contamination in recombinant human heat shock protein 70 (Hsp70) preparation is responsible for the induction of tumor necrosis factor alpha release by murine macrophagesBaochong Gao
Institute for Clinical Research and Veterans Affairs Medical Center, Washington, DC 20422, USA
J Biol Chem 278:174-9. 2003..These results suggest that rhHsp-70 does not induce TNFalpha release from murine macrophages and that the observed TNFalpha-inducing activity in the rhHsp70-2 preparation is entirely due to the contaminating LPS...
Role of alpha(v)beta(3)-integrin in TNF-alpha-induced endothelial cell migrationBaochong Gao
Department of Physiology, Albany Medical College, Albany, NY 12208, USA
Am J Physiol Cell Physiol 283:C1196-205. 2002....
TNF-alpha disruption of lung endothelial integrity: reduced integrin mediated adhesion to fibronectinRobert F Rotundo
Department of Physiology and Cell Biology, Neil Hellman Medical Research Building, Albany Medical College, Albany, New York 12208, USA
Am J Physiol Lung Cell Mol Physiol 282:L316-29. 2002....
Pathogen-associated molecular pattern contamination as putative endogenous ligands of Toll-like receptorsMin Fu Tsan
Department of Veterans Affairs, Research Service, VA Medical Center, Washington, DC 20422, USA
J Endotoxin Res 13:6-14. 2007..Strategies are proposed to avoid future designation of PAMP contamination as putative endogenous ligands of TLRs...
Heat shock protein and innate immunityMin Fu Tsan
Research Service, VA Medical Center, Washington DC 20422, USA
Cell Mol Immunol 1:274-9. 2004..This concise review summarizes the current controversy over the role of HSPs in innate immunity...
Induction of cytokines by heat shock proteins and concanavalin A in murine splenocytesYun Wang
Institute for Clinical Research, Washington, DC 20422, USA
Cytokine 32:149-54. 2005..In contrast, concanavalin A induced the expression of a number of cytokine genes and the release of IFN-gamma and IL-2. These results suggest that Hsp60 and Hsp70 do not induce cytokine production by murine splenocytes...
Heat shock proteins and immune systemMin Fu Tsan
Office of Research Oversight, Department of Veterans Affairs, 50 Irving Street, N W, Washington, DC 20422, USA
J Leukoc Biol 85:905-10. 2009..The reported antigen presentation and cross-presentation and in vitro HSP cytokine functions are a result of molecules bound to or chaperoned by HSPs but not a result of HSPs themselves...
Cytokine function of heat shock proteinsMin Fu Tsan
Research Service, Veterans Affairs Medical Center, Washington, DC 20422, USA
Am J Physiol Cell Physiol 286:C739-44. 2004....
Exogenous procalcitonin evokes a pro-inflammatory cytokine responseAngelike P Liappis
Medical Service and Section of Infectious Diseases, Veterans Affairs Medical Center, 50 Irving Street, NW 4A155, Washington, DC, USA
Inflamm Res 60:203-7. 2011..Endogenous levels of ProCT might influence the response of polymorphonuclear leukocytes (PMNs), independently of endotoxin, in clinical disease...
