Research Topics
| Deborah R TaylorSummaryAffiliation: Food and Drug Administration Country: USA Publications
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Detail Information
Publications
Severe acute respiratory syndrome coronavirus infection in vaccinated ferretsMiriam E R Darnell
Laboratory of Hepatitis and Related Emerging Agents, Division of Emerging and Transfusion Transmitted Diseases, Office of Blood Research and Review, CBER FDA, Bethesda, MD 20892, USA
J Infect Dis 196:1329-38. 2007..Previous vaccine reports have noted robust neutralizing antibody and inflammatory responses in ferrets, resulting in enhanced hepatitis...
Obstacles and advances in SARS vaccine developmentDeborah R Taylor
Division of Emerging and Transfusion Transmitted Diseases, Office of Blood Research and Review, Center for Biologics Evaluation and Research CBER, US Food and Drug Administration, Bethesda, MD 20892, USA
Vaccine 24:863-71. 2006..Described here are issues related to progress in vaccine development and the obstacles that lie ahead for both researchers and regulatory agencies...
New antiviral pathway that mediates hepatitis C virus replicon interferon sensitivity through ADAR1Deborah R Taylor
CBER FDA, HFM 448, 8800 Rockville Pike, Bethesda, MD 20892, USA
J Virol 79:6291-8. 2005..These results may explain successful HCV replicon clearance by IFN-alpha in vitro and may provide a promising new therapeutic strategy for HCV as well as other viral infections...
Persistent growth of a human plasma-derived hepatitis C virus genotype 1b isolate in cell cultureErica Silberstein
Division of Emerging and Transfusion Transmitted Diseases, Center for Biologics Evaluation and Research, Food and Drug Administration, Bethesda, Maryland, USA
PLoS Pathog 6:e1000910. 2010..This virus constitutes a novel tool for the study of one of the most relevant strains of HCV, genotype 1b, which will now be available for HCV life cycle research and useful for the development of new therapeutics...
Evaluation of inactivation methods for severe acute respiratory syndrome coronavirus in noncellular blood productsMiriam E R Darnell
Center for Biologics Evaluation and Research, U.S. Food and Drug Administration, Bethesda, Maryland, USA
Transfusion 46:1770-7. 2006..CONCLUSION: Heat, UVC irradiation, and S/D treatments effectively inactivate SARS-CoV, whereas octanoic acid treatment is insufficient for inactivation of the virus...
Achieving a golden mean: mechanisms by which coronaviruses ensure synthesis of the correct stoichiometric ratios of viral proteinsEwan P Plant
Laboratory of Hepatitis and Related Emerging Agents, Division of Emerging and Transfusion Transmitted Diseases, Office of Blood Research and Review, CBER, FDA, 8800 Rockville Pike, HFM310, Bethesda, Maryland 20892, USA
J Virol 84:4330-40. 2010..The findings of these analyses all support a "golden mean" model in which viruses use both programmed ribosomal frameshifting and translational attenuation to control the relative ratios of their encoded proteins...
Inactivation of the coronavirus that induces severe acute respiratory syndrome, SARS-CoVMiriam E R Darnell
Center for Biologics Evaluation and Research, US Food and Drug Administration, 8800 Rockville Pike, HFM448, Bethesda, MD 20892, USA
J Virol Methods 121:85-91. 2004..We describe the kinetics of these efficient viral inactivation methods, which will allow research with SARS-CoV containing materials, that are rendered non-infectious, to be conducted at reduced safety levels...
Innate immunity and hepatitis C virus: eluding the host cell defenseDeborah R Taylor
Laboratory of Hepatitis and Related Emerging Agents, Division of Emerging and Transfusion Transmitted Diseases, Office of Blood Research and Review, CBER FDA, Bethesda, MD 20892, USA
Front Biosci 14:4950-61. 2009..Here we review the different ways that HCV evades the immune response elicited by IFN...
Protein synthesis and endoplasmic reticulum stress can be modulated by the hepatitis C virus envelope protein E2 through the eukaryotic initiation factor 2alpha kinase PERKNicole Pavio
Howard Hughes Medical Institute, Department of Molecular Microbiology and Immunology, School of Medicine, University of Southern California, Los Angeles, California 90033, USA
J Virol 77:3578-85. 2003..These findings may also explain how the virus promotes persistent infection by overcoming the cellular ER stress response...
Hepatitis C virus NS5A colocalizes with the core protein on lipid droplets and interacts with apolipoproteinsStephanie T Shi
Department of Molecular Microbiology and Immunology, University of Southern California, Keck School of Medicine, Los Angeles, California 90033, USA
Virology 292:198-210. 2002....
Detection of a novel unglycosylated form of hepatitis C virus E2 envelope protein that is located in the cytosol and interacts with PKRNicole Pavio
Howard Hughes Medical Institute, Keck School of Medicine, University of Southern California, Los Angeles, California 90033, USA
J Virol 76:1265-72. 2002..Thus, an ER protein can exist in the cytosol as an unglycosylated species and impair cellular functions...
