Amilcar A Castellano Sanchez

Summary

Affiliation: Emory University
Country: USA

Publications

  1. ncbi Granular cell astrocytomas show a high frequency of allelic loss but are not a genetically defined subset
    Amilcar A Castellano-Sanchez
    Department of Pathology, Laboratory Medicine, Emory University School of Medicine, Atlanta, GA 30322, USA
    Brain Pathol 13:185-94. 2003
  2. ncbi Evaluation of hematoxylin and eosin and special stains for the detection of acanthamoeba keratitis in penetrating keratoplasties
    Hans E Grossniklaus
    Department of Ophthalmology, Emory University School of Medicine, Atlanta, Georgia, USA
    Am J Ophthalmol 136:520-6. 2003
  3. ncbi Pseudopalisades in glioblastoma are hypoxic, express extracellular matrix proteases, and are formed by an actively migrating cell population
    Daniel J Brat
    Department of Pathology, Emory University School of Medicine, Atlanta, Georgia, USA
    Cancer Res 64:920-7. 2004
  4. ncbi Congenital glioblastoma: a clinicopathologic and genetic analysis
    Daniel J Brat
    Departments of Pathology and Laboratory Medicine and Winship Cancer Institute, Emory University School of Medicine, 1364 Clifton Road NE, Atlanta, GA 30322, USA
    Brain Pathol 17:276-81. 2007
  5. doi Von Hippel-Lindau (VHL) disease: an update on the clinico-pathologic and genetic aspects
    Bahig M Shehata
    Department of Pathology, Children s Healthcare of Atlanta, Egleston, Atlanta, GA 30322, USA
    Adv Anat Pathol 15:165-71. 2008
  6. ncbi Castleman's disease in a child presenting with a partly mineralized solitary meningeal mass
    Ana Sotrel
    Department of Pathology, Miami Children s Hospital, Miami, Florida 33135, USA
    Pediatr Neurosurg 38:232-7. 2003

Collaborators

Detail Information

Publications6

  1. ncbi Granular cell astrocytomas show a high frequency of allelic loss but are not a genetically defined subset
    Amilcar A Castellano-Sanchez
    Department of Pathology, Laboratory Medicine, Emory University School of Medicine, Atlanta, GA 30322, USA
    Brain Pathol 13:185-94. 2003
    ..Instead, it appears that granular cell change occurs across genetic subsets. The high frequency of allelic loss, especially on 9p and 10q, may confer aggressive growth potential and be related to their rapid clinical progression...
  2. ncbi Evaluation of hematoxylin and eosin and special stains for the detection of acanthamoeba keratitis in penetrating keratoplasties
    Hans E Grossniklaus
    Department of Ophthalmology, Emory University School of Medicine, Atlanta, Georgia, USA
    Am J Ophthalmol 136:520-6. 2003
    ..To compare the efficacy of a battery of routine and special histologic stains for the detection of acanthamoeba keratitis...
  3. ncbi Pseudopalisades in glioblastoma are hypoxic, express extracellular matrix proteases, and are formed by an actively migrating cell population
    Daniel J Brat
    Department of Pathology, Emory University School of Medicine, Atlanta, Georgia, USA
    Cancer Res 64:920-7. 2004
    ....
  4. ncbi Congenital glioblastoma: a clinicopathologic and genetic analysis
    Daniel J Brat
    Departments of Pathology and Laboratory Medicine and Winship Cancer Institute, Emory University School of Medicine, 1364 Clifton Road NE, Atlanta, GA 30322, USA
    Brain Pathol 17:276-81. 2007
    ..Nonetheless, the strong nuclear expression of p53 in these and other pediatric GBMs could indicate that p53 dysregulation is important to tumorigenesis...
  5. doi Von Hippel-Lindau (VHL) disease: an update on the clinico-pathologic and genetic aspects
    Bahig M Shehata
    Department of Pathology, Children s Healthcare of Atlanta, Egleston, Atlanta, GA 30322, USA
    Adv Anat Pathol 15:165-71. 2008
    ..This paper will detail the variety of tumors associated with VHL disease and discuss the genetic mechanisms that lead to the predisposition for neoplasia...
  6. ncbi Castleman's disease in a child presenting with a partly mineralized solitary meningeal mass
    Ana Sotrel
    Department of Pathology, Miami Children s Hospital, Miami, Florida 33135, USA
    Pediatr Neurosurg 38:232-7. 2003
    ..At the 6-month follow-up, the patient was seizure free on antiepileptics and had returned to normal daily activities. MRI showed no residual lesion, and a workup for systemic disease was negative...