J K Smith

Summary

Affiliation: East Tennessee State University
Country: USA

Publications

  1. ncbi request reprint Long-term exercise and atherogenic activity of blood mononuclear cells in persons at risk of developing ischemic heart disease
    J K Smith
    Department of Internal Medicine, James H Quillen College of Medicine, East Tennessee State University, Johnson City 37614, USA
    JAMA 281:1722-7. 1999
  2. ncbi request reprint Interferon-alpha upregulates gene expression of aquaporin-5 in human parotid glands
    J K Smith
    Department of Internal Medicine, James H Quillen College of Medicine, Johnson City, TN 37614 0622, USA
    J Interferon Cytokine Res 19:929-35. 1999
  3. ncbi request reprint Human endothelium as a source of multifunctional cytokines: molecular regulation and possible role in human disease
    G Krishnaswamy
    Department of Internal Medicine, James H Quillen College of Medicine, East Tennessee State University, Johnson City 37614 0622, USA
    J Interferon Cytokine Res 19:91-104. 1999
  4. ncbi request reprint Exercise and atherogenesis
    J K Smith
    Division of Immunology, Department of Internal Medicine, James H Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee, TN 37614, USA
    Exerc Sport Sci Rev 29:49-53. 2001
  5. ncbi request reprint Oral use of interferon-alpha stimulates ISG-15 transcription and production by human buccal epithelial cells
    J K Smith
    Department of Internal Medicine, James H Quillen College of Medicine, East Tennessee State University, Johnson City 37614 0622, USA
    J Interferon Cytokine Res 19:923-8. 1999

Detail Information

Publications5

  1. ncbi request reprint Long-term exercise and atherogenic activity of blood mononuclear cells in persons at risk of developing ischemic heart disease
    J K Smith
    Department of Internal Medicine, James H Quillen College of Medicine, East Tennessee State University, Johnson City 37614, USA
    JAMA 281:1722-7. 1999
    ..It is not known whether long-term exercise alters this atherogenic and atheroprotective activity directly...
  2. ncbi request reprint Interferon-alpha upregulates gene expression of aquaporin-5 in human parotid glands
    J K Smith
    Department of Internal Medicine, James H Quillen College of Medicine, Johnson City, TN 37614 0622, USA
    J Interferon Cytokine Res 19:929-35. 1999
    ..We conclude that IFN-alpha upregulates AQP5 gene expression in human parotid acinar cells in vitro. To our knowledge, this is the first demonstration that IFN-alpha regulates the gene expression of an aquaporin...
  3. ncbi request reprint Human endothelium as a source of multifunctional cytokines: molecular regulation and possible role in human disease
    G Krishnaswamy
    Department of Internal Medicine, James H Quillen College of Medicine, East Tennessee State University, Johnson City 37614 0622, USA
    J Interferon Cytokine Res 19:91-104. 1999
    ..Genetic and pharmacologic manipulation of endothelial-derived cytokines provides an additional approach to the management of chronic inflammatory diseases...
  4. ncbi request reprint Exercise and atherogenesis
    J K Smith
    Division of Immunology, Department of Internal Medicine, James H Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee, TN 37614, USA
    Exerc Sport Sci Rev 29:49-53. 2001
    ..Exercise training contributes to the arrest and even reversal of atherosclerosis by modifying risk factors and by inducing an atheroprotective phenotype in endothelial cells and T cells...
  5. ncbi request reprint Oral use of interferon-alpha stimulates ISG-15 transcription and production by human buccal epithelial cells
    J K Smith
    Department of Internal Medicine, James H Quillen College of Medicine, East Tennessee State University, Johnson City 37614 0622, USA
    J Interferon Cytokine Res 19:923-8. 1999
    ..We conclude that orally administered IFN-alpha exerts its immunomodulatory effects in humans in part by upregulating the production of ISG-15 by BEC, thereby enhancing the immune reactivity of mucosa-associated lymphocytes...