Mary B Terry

Summary

Affiliation: Columbia University
Country: USA

Publications

  1. request reprint
    Terry M, Knight J, Zablotska L, Wang Q, John E, Andrulis I, et al. Alcohol metabolism, alcohol intake, and breast cancer risk: a sister-set analysis using the Breast Cancer Family Registry. Breast Cancer Res Treat. 2007;106:281-8 pubmed
    ..However, only 19% and 42% of sisters were discordant by ADH1B and ADH1C, respectively, and even fewer were discordant by both genotype and alcohol intake, making it difficult to detect differences if they existed. ..
  2. Quante A, Whittemore A, Shriver T, Hopper J, Strauch K, Terry M. Practical problems with clinical guidelines for breast cancer prevention based on remaining lifetime risk. J Natl Cancer Inst. 2015;107: pubmed publisher
    ..Guidelines based on short-term risks would be more useful, as models are generally developed and validated under a short fixed time horizon (?10 years). ..
  3. Quante A, Herz J, Whittemore A, Fischer C, Strauch K, Terry M. Assessing absolute changes in breast cancer risk due to modifiable risk factors. Breast Cancer Res Treat. 2015;152:193-197 pubmed publisher
    ..This contrast illustrates the value of using information on modifiable risk factors in risk assessment and in sharing information with patients of their absolute risks with and without modifiable risk factors. ..
  4. Terry M, McDonald J, Wu H, Eng S, Santella R. Epigenetic Biomarkers of Breast Cancer Risk: Across the Breast Cancer Prevention Continuum. Adv Exp Med Biol. 2016;882:33-68 pubmed publisher
    ..Large, prospective epidemiological studies will provide essential evidence of the overall utility of adding these markers to primary prevention efforts, screening, and clinical care. ..
  5. Akinyemiju T, Tehranifar P, Flom J, Liao Y, Wei Y, Terry M. Early life growth, socioeconomic status, and mammographic breast density in an urban US birth cohort. Ann Epidemiol. 2016;26:540-545.e2 pubmed publisher
    ..24, 95% CI: 0.04-0.43). Similar associations were observed for dense area, but those estimates were not statistically significant. These results suggest opposite and independent effects of early life SES and growth on MD. ..
  6. request reprint
    Terry M, Gammon M, Zhang F, Eng S, Sagiv S, Paykin A, et al. Polymorphism in the DNA repair gene XPD, polycyclic aromatic hydrocarbon-DNA adducts, cigarette smoking, and breast cancer risk. Cancer Epidemiol Biomarkers Prev. 2004;13:2053-8 pubmed
    ..Overall, this study suggests that those individuals with this polymorphism in the XPD gene may face an increased risk of breast cancer from PAH-DNA adducts and cigarette smoking. ..
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    Terry M, Gammon M, Zhang F, Knight J, Wang Q, Britton J, et al. ADH3 genotype, alcohol intake and breast cancer risk. Carcinogenesis. 2006;27:840-7 pubmed
    ..9, 95 % CI=1.2-7.1; postmenopausal women OR=1.8, 95% CI=0.9-3.8). These population-based data support the hypothesis that fast metabolizers of alcohol have a higher risk of breast cancer risk, from alcohol intake than slow metabolizers. ..
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    Terry M, Perrin M, Salafia C, Zhang F, Neugut A, Teitelbaum S, et al. Preeclampsia, pregnancy-related hypertension, and breast cancer risk. Am J Epidemiol. 2007;165:1007-14 pubmed
    ..These data suggest that pregnancy conditions related to hypertension, particularly preeclampsia, play a role in reducing breast cancer risk. Possible biologic mechanisms underpinning these associations should be further explored...
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    Terry M, Gammon M, Zhang F, Vaughan T, Chow W, Risch H, et al. Alcohol dehydrogenase 3 and risk of esophageal and gastric adenocarcinomas. Cancer Causes Control. 2007;18:1039-46 pubmed
    ..These data suggest ADH3 genotype may be associated with risk of esophageal and gastric cardia adenocarcinomas. ..

More Information

Publications15

  1. Terry M, Ferris J, Tehranifar P, Wei Y, Flom J. Birth weight, postnatal growth, and age at menarche. Am J Epidemiol. 2009;170:72-9 pubmed publisher
    ..These New York Women's Birth Cohort Adult Follow-up data (2001-2006) suggest that infant weight gain, in addition to childhood weight gain, may be associated with earlier age at menarche. ..
  2. Terry M, Flom J, Tehranifar P, Susser E. The role of birth cohorts in studies of adult health: the New York women's birth cohort. Paediatr Perinat Epidemiol. 2009;23:431-45 pubmed publisher
    ..Understanding the factors that are associated with tracing and participation in these existing cohorts will help in interpreting the validity and generalisability of the findings from these invaluable cohorts. ..
  3. Terry M, Delgado Cruzata L, Vin Raviv N, Wu H, Santella R. DNA methylation in white blood cells: association with risk factors in epidemiologic studies. Epigenetics. 2011;6:828-37 pubmed
  4. Strizich G, Gammon M, Jacobson J, Wall M, Abrahamson P, Bradshaw P, et al. Latent class analysis suggests four distinct classes of complementary medicine users among women with breast cancer. BMC Complement Altern Med. 2015;15:411 pubmed publisher
    ..Further research should explore the reproducibility of these classes and investigate the association between latent class membership and breast cancer outcomes. ..
  5. Terry M, Goldberg M, Schechter S, Houghton L, White M, O Toole K, et al. Comparison of Clinical, Maternal, and Self Pubertal Assessments: Implications for Health Studies. Pediatrics. 2016;138: pubmed publisher
    ..These differences in validity should be considered in studies measuring pubertal changes longitudinally when they do not have access to clinical assessments. ..
  6. Flom J, Cohn B, Tehranifar P, Houghton L, Wei Y, Protacio A, et al. Earlier age at menarche in girls with rapid early life growth: cohort and within sibling analyses. Ann Epidemiol. 2017;27:187-193.e2 pubmed publisher
    ..6 months earlier than girls whose growth was stable. Faster postnatal weight gains in infancy and early childhood before the age of 4 years are associated with earlier age at menarche. ..