Andrew F Teich

Summary

Affiliation: Columbia University
Country: USA

Publications

  1. ncbi request reprint Comparison among some models of orientation selectivity
    Andrew F Teich
    Center for Neurobiology and Behavior, Mahoney Center for Brain and Behaviour Research, and Department of Physiology and Cellular Biophysics, Columbia University, New York, NY 10032, USA
    J Neurophysiol 96:404-19. 2006
  2. pmc A reliable way to detect endogenous murine β-amyloid
    Andrew F Teich
    Department of Pathology and Cell Biology, Taub Institute for Research on Alzheimer s Disease and the Aging Brain, Columbia University, New York, New York, United States of America
    PLoS ONE 8:e55647. 2013
  3. pmc Is the amyloid hypothesis of Alzheimer's disease therapeutically relevant?
    Andrew F Teich
    Department of Pathology and Cell Biology, Columbia University Medical Center, 630 West 168th Street, PH15 124, New York, NY 10032, USA
    Biochem J 446:165-77. 2012
  4. pmc V1 orientation plasticity is explained by broadly tuned feedforward inputs and intracortical sharpening
    Andrew F Teich
    Department of Pathology, Columbia University, New York, New York 10032, USA
    Vis Neurosci 27:57-73. 2010
  5. ncbi request reprint Learning and adaptation in a recurrent model of V1 orientation selectivity
    Andrew F Teich
    Center for Neurobiology and Behavior and Department of Physiology and Cellular Biophysics, Columbia University, New York, New York 10032, USA
    J Neurophysiol 89:2086-100. 2003
  6. doi request reprint 5-HT₄ receptor stimulation leads to soluble AβPPα production through MMP-9 upregulation
    Gakuji Hashimoto
    Taub Institute for Research on Alzheimer s Disease and the Aging Brain, Columbia University Medical Center, New York, NY, USA
    J Alzheimers Dis 32:437-45. 2012

Research Grants

Collaborators

Detail Information

Publications6

  1. ncbi request reprint Comparison among some models of orientation selectivity
    Andrew F Teich
    Center for Neurobiology and Behavior, Mahoney Center for Brain and Behaviour Research, and Department of Physiology and Cellular Biophysics, Columbia University, New York, NY 10032, USA
    J Neurophysiol 96:404-19. 2006
    ..Because orientation-tuned V1 cells show a continuum of simple- to complex-cell behavior, the MRM provides the best description of V1 data...
  2. pmc A reliable way to detect endogenous murine β-amyloid
    Andrew F Teich
    Department of Pathology and Cell Biology, Taub Institute for Research on Alzheimer s Disease and the Aging Brain, Columbia University, New York, New York, United States of America
    PLoS ONE 8:e55647. 2013
    ..Here, we examine the background signal of several murine β-amyloid ELISAs, and conclude that the majority of the background is from non-APP derived proteins. Most importantly, we identify ELISAs that eliminate this background signal...
  3. pmc Is the amyloid hypothesis of Alzheimer's disease therapeutically relevant?
    Andrew F Teich
    Department of Pathology and Cell Biology, Columbia University Medical Center, 630 West 168th Street, PH15 124, New York, NY 10032, USA
    Biochem J 446:165-77. 2012
    ....
  4. pmc V1 orientation plasticity is explained by broadly tuned feedforward inputs and intracortical sharpening
    Andrew F Teich
    Department of Pathology, Columbia University, New York, New York 10032, USA
    Vis Neurosci 27:57-73. 2010
    ..We predict that the plastic properties must be absent for cells whose orientation tuning arises from a feedforward mechanism...
  5. ncbi request reprint Learning and adaptation in a recurrent model of V1 orientation selectivity
    Andrew F Teich
    Center for Neurobiology and Behavior and Department of Physiology and Cellular Biophysics, Columbia University, New York, New York 10032, USA
    J Neurophysiol 89:2086-100. 2003
    ..Instead, human adaptation studies can be better accounted for by the learning data from behaving animals. Our work suggests that adaptation in behaving subjects may be viewed as a short-term form of learning...
  6. doi request reprint 5-HT₄ receptor stimulation leads to soluble AβPPα production through MMP-9 upregulation
    Gakuji Hashimoto
    Taub Institute for Research on Alzheimer s Disease and the Aging Brain, Columbia University Medical Center, New York, NY, USA
    J Alzheimers Dis 32:437-45. 2012
    ..Taken all together, these experiments demonstrate that 5-HT4 receptor stimulation induces expression of MMP-9 which cleaves AβPP through α-secretase-like activity, leading to an increase of sAβPPα levels and a reduction of Aβ load...

Research Grants1

  1. Learning and Adaptation in Primary Visual Cortex
    ANDREW TEICH; Fiscal Year: 2005
    ..The goal of Specific Aim 2 is to experimentally test the prediction that adaptation in alert, non-anesthetized subjects causes physiological changes that are a transient version of the changes seen in learning. ..