Christine A Richardson

Summary

Affiliation: Columbia University
Country: USA

Publications

  1. ncbi Rad51 overexpression promotes alternative double-strand break repair pathways and genome instability
    Christine Richardson
    Department of Pathology, Institute of Cancer Genetics, Columbia University College of Physicians and Surgeons, 1150 St Nicholas Avenue, New York, NY 10032, USA
    Oncogene 23:546-53. 2004
  2. ncbi RAD51, genomic stability, and tumorigenesis
    Christine Richardson
    Department of Pathology, Institute for Cancer Genetics, College of Physicians and Surgeons, Columbia University, 1150 St Nicholas Ave, New York, NY 10032, USA
    Cancer Lett 218:127-39. 2005
  3. ncbi Modeling oncogenic translocations: distinct roles for double-strand break repair pathways in translocation formation in mammalian cells
    David M Weinstock
    Department of Medicine, Memorial Sloan Kettering Cancer Center, 1275 York Avenue, New York, NY 10021, USA
    DNA Repair (Amst) 5:1065-74. 2006
  4. ncbi Up-regulation of AMP-activated kinase by dysfunctional cystic fibrosis transmembrane conductance regulator in cystic fibrosis airway epithelial cells mitigates excessive inflammation
    Kenneth R Hallows
    Department of Medicine, University of Pittsburgh School of Medicine, Pennsylvania 15261, USA
    J Biol Chem 281:4231-41. 2006

Collaborators

Detail Information

Publications4

  1. ncbi Rad51 overexpression promotes alternative double-strand break repair pathways and genome instability
    Christine Richardson
    Department of Pathology, Institute of Cancer Genetics, Columbia University College of Physicians and Surgeons, 1150 St Nicholas Avenue, New York, NY 10032, USA
    Oncogene 23:546-53. 2004
    ..Increased Rad51 also promoted aneuploidy and multiple chromosomal rearrangements. These data provide a link between elevated Rad51 protein levels, genome instability, and tumor progression...
  2. ncbi RAD51, genomic stability, and tumorigenesis
    Christine Richardson
    Department of Pathology, Institute for Cancer Genetics, College of Physicians and Surgeons, Columbia University, 1150 St Nicholas Ave, New York, NY 10032, USA
    Cancer Lett 218:127-39. 2005
    ..Insights into the connection between disregulated Rad51 and malignant phenotype indicate that Rad51 is a potential target for new anti-cancer regimens including those that use siRNA technology...
  3. ncbi Modeling oncogenic translocations: distinct roles for double-strand break repair pathways in translocation formation in mammalian cells
    David M Weinstock
    Department of Medicine, Memorial Sloan Kettering Cancer Center, 1275 York Avenue, New York, NY 10021, USA
    DNA Repair (Amst) 5:1065-74. 2006
    ..Although SSA can efficiently mediate translocations between identical repeats, its contribution to translocation formation is likely very limited because of sequence divergence between repetitive elements in the genome...
  4. ncbi Up-regulation of AMP-activated kinase by dysfunctional cystic fibrosis transmembrane conductance regulator in cystic fibrosis airway epithelial cells mitigates excessive inflammation
    Kenneth R Hallows
    Department of Medicine, University of Pittsburgh School of Medicine, Pennsylvania 15261, USA
    J Biol Chem 281:4231-41. 2006
    ..We propose that therapies to activate AMPK in the CF airway may be beneficial in reducing excessive airway inflammation, a major cause of CF morbidity...