Craig Bolte

Summary

Affiliation: Cincinnati Children's Hospital Medical Center
Country: USA

Publications

  1. ncbi Postnatal ablation of Foxm1 from cardiomyocytes causes late onset cardiac hypertrophy and fibrosis without exacerbating pressure overload-induced cardiac remodeling
    Craig Bolte
    Division of Pulmonary Biology, Cincinnati Children s Hospital Research Foundation, Cincinnati, Ohio, United States of America
    PLoS ONE 7:e48713. 2012
  2. ncbi Expression of Foxm1 transcription factor in cardiomyocytes is required for myocardial development
    Craig Bolte
    Division of Pulmonary Biology, Cincinnati Children s Hospital Research Foundation, Cincinnati, Ohio, United States of America
    PLoS ONE 6:e22217. 2011

Detail Information

Publications2

  1. ncbi Postnatal ablation of Foxm1 from cardiomyocytes causes late onset cardiac hypertrophy and fibrosis without exacerbating pressure overload-induced cardiac remodeling
    Craig Bolte
    Division of Pulmonary Biology, Cincinnati Children s Hospital Research Foundation, Cincinnati, Ohio, United States of America
    PLoS ONE 7:e48713. 2012
    ..However, Foxm1 deletion did not exacerbate cardiac hypertrophy or fibrosis following chronic pressure overload. Our results demonstrate that Foxm1 regulates genes critical for age-induced cardiomyocyte hypertrophy and cardiac fibrosis...
  2. ncbi Expression of Foxm1 transcription factor in cardiomyocytes is required for myocardial development
    Craig Bolte
    Division of Pulmonary Biology, Cincinnati Children s Hospital Research Foundation, Cincinnati, Ohio, United States of America
    PLoS ONE 6:e22217. 2011
    ..Our results indicate that Foxm1 expression in cardiomyocytes is critical for proper heart development and required for cardiomyocyte proliferation and myocardial growth...