A A Grom

Summary

Affiliation: Children's Hospital Medical Center
Country: USA

Publications

  1. ncbi request reprint Macrophage activation syndrome and reactive hemophagocytic lymphohistiocytosis: the same entities?
    Alexei A Grom
    Cincinnati Children s Hospital Medical Center, OH 45215, USA
    Curr Opin Rheumatol 15:587-90. 2003
  2. ncbi request reprint T-cell and T-cell receptor abnormalities in the immunopathogenesis of juvenile rheumatoid arthritis
    A A Grom
    William S Rowe Division of Rheumatology, Children s Hospital Medical Center, Cincinnati, Ohio 45229, USA
    Curr Opin Rheumatol 12:420-4. 2000
  3. ncbi request reprint Natural killer cell dysfunction in patients with systemic-onset juvenile rheumatoid arthritis and macrophage activation syndrome
    Alexei A Grom
    William S Rowe Division of Rheumatology, and the Division of Hematology Oncology, Children s Hospital Medical Center, Cincinnati, Ohio 45229, USA
    J Pediatr 142:292-6. 2003
  4. ncbi request reprint Expression of angiogenic factors in juvenile rheumatoid arthritis: correlation with revascularization of human synovium engrafted into SCID mice
    M P Scola
    Children's Hospital Medical Center, Cincinnati, Ohio 45229, USA
    Arthritis Rheum 44:794-801. 2001
  5. ncbi request reprint Antibodies to the 45 kDa DEK nuclear antigen in pauciarticular onset juvenile rheumatoid arthritis and iridocyclitis: selective association with MHC gene
    K J Murray
    Department of Pediatrics, Children s Hospital Medical Center, University of Cincinnati College of Medicine, OH 45229, USA
    J Rheumatol 24:560-7. 1997
  6. ncbi request reprint Systemic features and early prognostic factors in Hispanic and non-Hispanic children from the United States of America and Mexico with systemic juvenile idiopathic arthritis
    M Shishov
    Cincinnati Children s Hospital Medical Center, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA
    Clin Exp Rheumatol 25:907-14. 2007
  7. ncbi request reprint Natural killer cell dysfunction: A common pathway in systemic-onset juvenile rheumatoid arthritis, macrophage activation syndrome, and hemophagocytic lymphohistiocytosis?
    Alexei A Grom
    Division of Rheumatology, Cincinnati Children s Hospital Medical Center, Cincinnati, Ohio 45215, USA
    Arthritis Rheum 50:689-98. 2004
  8. pmc Natural killer cell dysfunction is a distinguishing feature of systemic onset juvenile rheumatoid arthritis and macrophage activation syndrome
    Joyce Villanueva
    1Division of Hematology Oncology, Children s Hospital Medical Center, Cincinnati, Ohio, USA
    Arthritis Res Ther 7:R30-7. 2005
  9. ncbi request reprint The diagnostic significance of soluble CD163 and soluble interleukin-2 receptor alpha-chain in macrophage activation syndrome and untreated new-onset systemic juvenile idiopathic arthritis
    Jack Bleesing
    Children s Hospital Medical Center, Cincinnati, Ohio 45229, USA
    Arthritis Rheum 56:965-71. 2007
  10. ncbi request reprint Interleukin-15 inhibits sodium nitroprusside-induced apoptosis of synovial fibroblasts and vascular endothelial cells
    Lin Yang
    Children s Hospital Medical Center, Cincinnati, Ohio 45229, USA
    Arthritis Rheum 46:3010-4. 2002

Collaborators

  • Hermine Brunner
  • Daniel J Lovell
  • T Brent Graham
  • Murray H Passo
  • Joyce Villanueva
  • Jack Bleesing
  • Thomas Griffin
  • Judyann Olson
  • David D Sherry
  • Ndate Fall
  • M Shishov
  • Norman T Ilowite
  • Alexandra Filipovich
  • Sherry Thornton
  • David N Glass
  • Andrea C Barron
  • Lin Yang
  • Michael P Scola
  • M P Scola
  • D N Glass
  • E H Giannini
  • Lorie Luyrink
  • E Solis-Vallejo
  • Anne Prada
  • Susan Thompson
  • B Huang
  • R Burgos-Vargas
  • David M Siegel
  • N Rubio-PĂ©rez
  • Athimalaipet V Ramanan
  • R Romero-Feregrino
  • M Henrickson
  • Robert A Colbert
  • Michael Barnes
  • V Baca
  • Beth S Gottlieb
  • Edward H Giannini
  • Susan Lee
  • Thomas A Griffin
  • K J Murray
  • Terry Moore
  • Janalee Taylor
  • Tsz Leung Lee
  • Susan D Thompson
  • David Witte
  • Marc A van Dijk
  • Monica K Tsoras
  • R Hirsch
  • T Imagawa
  • G P Boivin
  • I S Szer
  • W Szer
  • J E Levinson
  • P Donnelly

Detail Information

Publications13

  1. ncbi request reprint Macrophage activation syndrome and reactive hemophagocytic lymphohistiocytosis: the same entities?
    Alexei A Grom
    Cincinnati Children s Hospital Medical Center, OH 45215, USA
    Curr Opin Rheumatol 15:587-90. 2003
    ..The main purpose of the review is to summarize current understanding of the relation between macrophage activation syndrome and other clinically similar hemophagocytic disorders...
  2. ncbi request reprint T-cell and T-cell receptor abnormalities in the immunopathogenesis of juvenile rheumatoid arthritis
    A A Grom
    William S Rowe Division of Rheumatology, Children s Hospital Medical Center, Cincinnati, Ohio 45229, USA
    Curr Opin Rheumatol 12:420-4. 2000
    ....
  3. ncbi request reprint Natural killer cell dysfunction in patients with systemic-onset juvenile rheumatoid arthritis and macrophage activation syndrome
    Alexei A Grom
    William S Rowe Division of Rheumatology, and the Division of Hematology Oncology, Children s Hospital Medical Center, Cincinnati, Ohio 45229, USA
    J Pediatr 142:292-6. 2003
    ..To assess natural killer (NK) and cytotoxic functions in patients with systemic-onset juvenile rheumatoid arthrithis (soJRA) complicated by macrophage activation syndrome (MAS)...
  4. ncbi request reprint Expression of angiogenic factors in juvenile rheumatoid arthritis: correlation with revascularization of human synovium engrafted into SCID mice
    M P Scola
    Children's Hospital Medical Center, Cincinnati, Ohio 45229, USA
    Arthritis Rheum 44:794-801. 2001
    ..CONCLUSION: JRA synovium is characterized by high angiogenic activity. SCID mouse-human JRA synovium chimeras may provide a good approach to study the in vivo regulation of angiogenesis in JRA...
  5. ncbi request reprint Antibodies to the 45 kDa DEK nuclear antigen in pauciarticular onset juvenile rheumatoid arthritis and iridocyclitis: selective association with MHC gene
    K J Murray
    Department of Pediatrics, Children s Hospital Medical Center, University of Cincinnati College of Medicine, OH 45229, USA
    J Rheumatol 24:560-7. 1997
    ..Class I and Class II HLA associations with reactivity to the antigen were also sought...
  6. ncbi request reprint Systemic features and early prognostic factors in Hispanic and non-Hispanic children from the United States of America and Mexico with systemic juvenile idiopathic arthritis
    M Shishov
    Cincinnati Children s Hospital Medical Center, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA
    Clin Exp Rheumatol 25:907-14. 2007
    ....
  7. ncbi request reprint Natural killer cell dysfunction: A common pathway in systemic-onset juvenile rheumatoid arthritis, macrophage activation syndrome, and hemophagocytic lymphohistiocytosis?
    Alexei A Grom
    Division of Rheumatology, Cincinnati Children s Hospital Medical Center, Cincinnati, Ohio 45215, USA
    Arthritis Rheum 50:689-98. 2004
  8. pmc Natural killer cell dysfunction is a distinguishing feature of systemic onset juvenile rheumatoid arthritis and macrophage activation syndrome
    Joyce Villanueva
    1Division of Hematology Oncology, Children s Hospital Medical Center, Cincinnati, Ohio, USA
    Arthritis Res Ther 7:R30-7. 2005
    ..This phenomenon was particularly common in the systemic form of JRA, a clinical entity strongly associated with MAS...
  9. ncbi request reprint The diagnostic significance of soluble CD163 and soluble interleukin-2 receptor alpha-chain in macrophage activation syndrome and untreated new-onset systemic juvenile idiopathic arthritis
    Jack Bleesing
    Children s Hospital Medical Center, Cincinnati, Ohio 45229, USA
    Arthritis Rheum 56:965-71. 2007
    ..This study was undertaken to assess the value of serum sIL-2Ralpha and sCD163 in diagnosing acute macrophage activation syndrome complicating systemic juvenile idiopathic arthritis (JIA)...
  10. ncbi request reprint Interleukin-15 inhibits sodium nitroprusside-induced apoptosis of synovial fibroblasts and vascular endothelial cells
    Lin Yang
    Children s Hospital Medical Center, Cincinnati, Ohio 45229, USA
    Arthritis Rheum 46:3010-4. 2002
    ..Since synovial vascular endothelial cells (VECs) expressed IL-15 receptors, the present study was undertaken to investigate the hypothesis that IL-15 might play a role in neovascularization of the pannus...
  11. ncbi request reprint Interferon-gamma:interleukin 4 ratios and associated type 1 cytokine expression in juvenile rheumatoid arthritis synovial tissue
    Michael P Scola
    Department of Pediatrics, Children s Hospital Medical Center, Cincinnati, Ohio 45229 3039, USA
    J Rheumatol 29:369-78. 2002
    ..To compare synovial tissue cytokine mRNA expression between patients with juvenile rheumatoid arthritis (JRA) and a heterogeneous group of non-autoimmune arthropathies (controls) with respect to type 1/type 2 balance...
  12. ncbi request reprint Feasibility and construct validity of the parent willingness-to-pay technique for children with juvenile idiopathic arthritis
    Andrea C Barron
    Cincinnati Children s Hospital Medical Center, University of Cincinnati, Cincinnati, Ohio 45229 3039, USA
    Arthritis Rheum 51:899-908. 2004
    ..To assess the feasibility and construct validity of the willingness-to-pay (WTP) technique for measuring health care preferences in families of children with juvenile idiopathic arthritis (JIA)...
  13. ncbi request reprint Gene expression profiling of peripheral blood from patients with untreated new-onset systemic juvenile idiopathic arthritis reveals molecular heterogeneity that may predict macrophage activation syndrome
    Ndate Fall
    Children s Hospital Medical Center, Cincinnati, Ohio 45229, USA
    Arthritis Rheum 56:3793-804. 2007
    ..This study was undertaken to better understand the relationship between systemic JIA and macrophage activation syndrome...