DONALD ANTHONY

Summary

Affiliation: Case Western Reserve University
Country: USA

Publications

  1. ncbi Baseline levels of soluble CD14 and CD16+56- natural killer cells are negatively associated with response to interferon/ribavirin therapy during HCV-HIV-1 coinfection
    Donald D Anthony
    Department of Medicine, Divisions of Infectious and Rheumatic Diseases, Case Western Reserve University, Center for AIDS Research, University Hospitals of Cleveland and VA Medical Center, Cleveland, OH 44106, USA
    J Infect Dis 206:969-73. 2012
  2. ncbi Lower peripheral blood CD14+ monocyte frequency and higher CD34+ progenitor cell frequency are associated with HBV vaccine induced response in HIV infected individuals
    D D Anthony
    Case Western Reserve University, Cleveland, OH 44106, USA
    Vaccine 29:3558-63. 2011
  3. ncbi Determinants of protection among HIV‐exposed seronegative persons: an overview
    Michael M Lederman
    Center for AIDS Research, Case Western Reserve University School of Medicine, University Hospitals Case Medical Center, Cleveland, OH 44106, USA
    J Infect Dis 202:S333-8. 2010
  4. ncbi Interferon-α is the primary plasma type-I IFN in HIV-1 infection and correlates with immune activation and disease markers
    Gareth A D Hardy
    Department of Pathology, Case Western Reserve University and University Hospitals Case Medical Center, Cleveland, Ohio, USA
    PLoS ONE 8:e56527. 2013

Research Grants

  1. Immunity of neoantigen response in HCV, HIV, and HCV-HIV
    DONALD ANTHONY; Fiscal Year: 2007
  2. DC-NK cell interaction in HCV and HCV-HIV infection
    DONALD ANTHONY; Fiscal Year: 2006
  3. Role of immature DC in host defense against HCV
    DONALD ANTHONY; Fiscal Year: 2007
  4. Role of immature DC in host defense against HCV
    Donald D Anthony; Fiscal Year: 2010

Detail Information

Publications4

  1. ncbi Baseline levels of soluble CD14 and CD16+56- natural killer cells are negatively associated with response to interferon/ribavirin therapy during HCV-HIV-1 coinfection
    Donald D Anthony
    Department of Medicine, Divisions of Infectious and Rheumatic Diseases, Case Western Reserve University, Center for AIDS Research, University Hospitals of Cleveland and VA Medical Center, Cleveland, OH 44106, USA
    J Infect Dis 206:969-73. 2012
    ..IL28B genotype was associated with therapy response but not sCD14 or CD16(+)56(-) NK frequency. Markers of innate immune activation predict poor host response to IFN-α-based HCV therapy during HCV-HIV coinfection...
  2. ncbi Lower peripheral blood CD14+ monocyte frequency and higher CD34+ progenitor cell frequency are associated with HBV vaccine induced response in HIV infected individuals
    D D Anthony
    Case Western Reserve University, Cleveland, OH 44106, USA
    Vaccine 29:3558-63. 2011
    ..Additionally, GM-CSF augmented the preservation of peripheral blood MDSC, which may contribute to the lack of improved vaccine responses...
  3. ncbi Determinants of protection among HIV‐exposed seronegative persons: an overview
    Michael M Lederman
    Center for AIDS Research, Case Western Reserve University School of Medicine, University Hospitals Case Medical Center, Cleveland, OH 44106, USA
    J Infect Dis 202:S333-8. 2010
    ....
  4. ncbi Interferon-α is the primary plasma type-I IFN in HIV-1 infection and correlates with immune activation and disease markers
    Gareth A D Hardy
    Department of Pathology, Case Western Reserve University and University Hospitals Case Medical Center, Cleveland, Ohio, USA
    PLoS ONE 8:e56527. 2013
    ..003). These studies identify IFNα derived from lymph nodes, rather than blood leukocytes, as a possible source of the IFN-I signature that contributes to immune activation in HIV-1 infection...

Research Grants9

  1. Immunity of neoantigen response in HCV, HIV, and HCV-HIV
    DONALD ANTHONY; Fiscal Year: 2007
    ..The goal will be to characterize the formation of protective immunity in the HCV, HIV and HCV-HIV infected host, and provide insight for the development of new immunotherapeutic strategies for these chronic infections. ..
  2. DC-NK cell interaction in HCV and HCV-HIV infection
    DONALD ANTHONY; Fiscal Year: 2006
    ..HCV, HIV, and HCV-HIV infected subjects. ..
  3. Role of immature DC in host defense against HCV
    DONALD ANTHONY; Fiscal Year: 2007
    ....
  4. Role of immature DC in host defense against HCV
    Donald D Anthony; Fiscal Year: 2010
    ..abstract_text> ..