Ning Zhang

Summary

Affiliation: Caliper Life Sciences
Country: USA

Publications

  1. ncbi A spontaneous acinar cell carcinoma model for monitoring progression of pancreatic lesions and response to treatment through noninvasive bioluminescence imaging
    Ning Zhang
    Caliper Life Sciences, Alameda, CA 94501, USA
    Clin Cancer Res 15:4915-24. 2009
  2. ncbi Imaging pulmonary NF-kappaB activation and therapeutic effects of MLN120B and TDZD-8
    Dan Ansaldi
    Caliper Life Sciences, Alameda, California, United States of America
    PLoS ONE 6:e25093. 2011
  3. ncbi Serum amyloid A-luciferase transgenic mice: response to sepsis, acute arthritis, and contact hypersensitivity and the effects of proteasome inhibition
    Ning Zhang
    Xenogen, Alameda, CA 94501, USA
    J Immunol 174:8125-34. 2005

Collaborators

  • Jae Beom Kim
  • Dan Ansaldi
  • Rajendra Singh
  • Fabio Stellari
  • Mark Roskey
  • Kevin P Francis
  • Eldad A Hod
  • Ed Lim

Detail Information

Publications3

  1. ncbi A spontaneous acinar cell carcinoma model for monitoring progression of pancreatic lesions and response to treatment through noninvasive bioluminescence imaging
    Ning Zhang
    Caliper Life Sciences, Alameda, CA 94501, USA
    Clin Cancer Res 15:4915-24. 2009
    ..We have generated an EL1-luc/TAg transgenic mouse model that develops spontaneous and bioluminescent acinar cell carcinomas. We applied this model to noninvasively monitor tumor development and drug response...
  2. ncbi Imaging pulmonary NF-kappaB activation and therapeutic effects of MLN120B and TDZD-8
    Dan Ansaldi
    Caliper Life Sciences, Alameda, California, United States of America
    PLoS ONE 6:e25093. 2011
    ..This approach will potentiate further studies on NF-κB regulation under various inflammatory conditions...
  3. ncbi Serum amyloid A-luciferase transgenic mice: response to sepsis, acute arthritis, and contact hypersensitivity and the effects of proteasome inhibition
    Ning Zhang
    Xenogen, Alameda, CA 94501, USA
    J Immunol 174:8125-34. 2005
    ..These results suggest that both local and systemic induction of A-SAA occur during inflammation and may contribute to the pathogenesis of chronic inflammatory diseases associated with amyloid deposition...