Hugh Randolph Byers

Summary

Affiliation: Boston University
Country: USA

Publications

  1. ncbi Role of cytoplasmic dynein in melanosome transport in human melanocytes
    H R Byers
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02118, USA
    J Invest Dermatol 114:990-7. 2000
  2. ncbi Role of cytoplasmic dynein in perinuclear aggregation of phagocytosed melanosomes and supranuclear melanin cap formation in human keratinocytes
    H Randolph Byers
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02114, USA
    J Invest Dermatol 121:813-20. 2003
  3. ncbi RNAi-mediated knockdown of protein kinase C-alpha inhibits cell migration in MM-RU human metastatic melanoma cell line
    Hugh Randolph Byers
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02118, USA
    Melanoma Res 20:171-8. 2010
  4. ncbi Requirement of dynactin p150(Glued) subunit for the functional integrity of the keratinocyte microparasol
    H Randolph Byers
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02118, USA
    J Invest Dermatol 127:1736-44. 2007
  5. ncbi Role of zyxin in differential cell spreading and proliferation of melanoma cells and melanocytes
    Ellen J van der Gaag
    Department of Dermatology, Boston University Medical School, Boston, Massachusetts 02118, USA
    J Invest Dermatol 118:246-54. 2002
  6. ncbi Telomere-based DNA damage responses: a new approach to melanoma
    Neelu Puri
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02118-2394, USA
    FASEB J 18:1373-81. 2004

Collaborators

  • Barbara Gilchrest
  • Neelu Puri
  • Ellen J van der Gaag
  • John Kubera
  • Mark S Eller
  • Sarah Dykstra
  • Dana M Amodeo
  • Nicole L Jalbert
  • Marie Therese Leccia
  • Sybren K Dekker

Detail Information

Publications6

  1. ncbi Role of cytoplasmic dynein in melanosome transport in human melanocytes
    H R Byers
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02118, USA
    J Invest Dermatol 114:990-7. 2000
    ....
  2. ncbi Role of cytoplasmic dynein in perinuclear aggregation of phagocytosed melanosomes and supranuclear melanin cap formation in human keratinocytes
    H Randolph Byers
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02114, USA
    J Invest Dermatol 121:813-20. 2003
    ....
  3. ncbi RNAi-mediated knockdown of protein kinase C-alpha inhibits cell migration in MM-RU human metastatic melanoma cell line
    Hugh Randolph Byers
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02118, USA
    Melanoma Res 20:171-8. 2010
    ..These findings provide further evidence that PKC-alpha plays an important role in melanoma cell migration and may have implications in therapies designed to disrupt melanoma cell motility by alteration of PKC-alpha signaling...
  4. ncbi Requirement of dynactin p150(Glued) subunit for the functional integrity of the keratinocyte microparasol
    H Randolph Byers
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02118, USA
    J Invest Dermatol 127:1736-44. 2007
    ..The findings provide evidence that dynactin p150(Glued) plays an important role in the functional integrity of the keratinocyte microparasol...
  5. ncbi Role of zyxin in differential cell spreading and proliferation of melanoma cells and melanocytes
    Ellen J van der Gaag
    Department of Dermatology, Boston University Medical School, Boston, Massachusetts 02118, USA
    J Invest Dermatol 118:246-54. 2002
    ..Therapies directed at the downregulation of this focal adhesion phosphoprotein in melanoma cells implicate a new approach for controlling melanoma cell growth...
  6. ncbi Telomere-based DNA damage responses: a new approach to melanoma
    Neelu Puri
    Department of Dermatology, Boston University School of Medicine, Boston, Massachusetts 02118-2394, USA
    FASEB J 18:1373-81. 2004
    ..We suggest that T-oligo mimics a physiologic DNA damage signal that is frequently masked in malignant cells and thereby activates innate cancer prevention responses. T-oligos may provide a novel therapeutic approach to melanoma...