Research Topics
Species | James SuliburkSummaryAffiliation: Baylor College of Medicine Country: USA Publications
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Detail Information
Publications
Ketamine-induced gastroprotection during endotoxemia: role of heme-oxygenase-1Kenneth S Helmer
Department of Surgery, University of Texas Medical School, Houston, Texas 77026, USA
Dig Dis Sci 51:1571-81. 2006..Ketamine diminishes the susceptibility of gastric mucosa to damage from luminal irritants during endotoxemia, which is mediated in part by down-regulation of iNOS and up-regulation of HO-1...- Which handheld should I buy?James Suliburk
Department of Surgery, University of Texas at Houston, Houston, Texas 77030, USA
Curr Surg 60:75-6. 2003..Try remembering the office number of a referring physician in the middle of the night or suffering through the Muzak-ridden delays of waiting for a hospital operator to give you a specific extension and you will carry your PDA too!.. - Ketamine attenuates early lipopolysaccharide-induced gastric dysfunction: role of stress-inducible phosphoproteinsJames W Suliburk
Department of General Surgery, The University of Texas Medical School at Houston, Houston, Texas, USA
J Trauma 62:316-9. 2007.... - Ketamine attenuates liver injury attributed to endotoxemia: role of cyclooxygenase-2James W Suliburk
Department of Surgery, The University of Texas Medical School at Houston, 77030, USA
Surgery 138:134-40. 2005..Because ketamine anesthesia has anti-inflammatory effects in some tissues, we hypothesized that it would attenuate lipopolysaccharide (LPS)-induced liver injury... - Ketamine inhibits lipopolysacharide (LPS) induced gastric luminal fluid accumulationJames W Suliburk
Department of General Surgery, The University of Texas Medical School at Houston, Houston, Texas 77026, USA
J Surg Res 127:203-7. 2005..Moreover, while iNOS may play a role in LPS induced gastric luminal fluid accumulation, it does not appear to be a major mediator of the gastrointestinal ileus caused by LPS... 
Ketamine-induced hepatoprotection: the role of heme oxygenase-1James W Suliburk
UT Houston Medical School, Dept of Surgery, 6431 Fannin, MSB Suite 4 264, Houston, TX 77030, USA
Am J Physiol Gastrointest Liver Physiol 296:G1360-9. 2009..Although CO abolished the ability of LPS to diminish PPAR-gamma activity, it enhanced NF-kappaB activity. These data suggest that the hepatoprotective effects of ketamine are mediated primarily by HO-1 and its end product CO...- Conventional dose hypertonic saline provides optimal gut protection and limits remote organ injury after gut ischemia reperfusionErnest A Gonzalez
Department of Surgery, The University of Texas Medical School at Houston, Houston, Texas 77030, USA
J Trauma 61:66-73; discussion 73-4. 2006..We hypothesized that a dose dependent effect exists related to increasing tonicity and that the optimal gut protective dose would provide better protection against remote organ injury than large volume isotonic crystalloids... - Effect of NOS inhibition on rat gastric matrix metalloproteinase production during endotoxemiaEmily K Robinson
Department of Surgery, The University of Texas Health Science Center at Houston, Houston, TX 77030, USA
Shock 25:507-14. 2006..Therefore, the purpose of these studies was to compare the effects of selective and nonselective NOS inhibition on LPS-induced gastric MMP production... - Hypertonic saline resuscitation after mesenteric ischemia/reperfusion induces ileal apoptosisErnest A Gonzalez
Department of Surgery, The University of Texas Medical School at Houston, Houston, Texas 77030, USA
J Trauma 59:1092-8. 2005..In contrast to I/R cell necrosis, apoptosis provides controlled cell death that minimizes inflammation. We therefore hypothesized that HS resuscitation after mesenteric I/R would induce apoptosis and decrease mucosal injury... - Differential effects of anesthetics on endotoxin-induced liver injuryJames W Suliburk
Department of Surgery, The University of Texas Medical School at Houston, Houston, Texas, USA
J Trauma 58:711-6; discussion 716-7. 2005..Thus, ketamine may be the anesthetic agent of choice for septic patients requiring anesthesia... - Rat gastric injury after lipopolysaccharide: role of inducible nitric oxide synthaseEmily K Robinson
Department of Surgery, University of Texas Health Science Center, Houston, TX 77026, USA
Surgery 138:523-9. 2005..The effects of long-term treatment with LPS on gastric injury are unknown as is the role of iNOS. We hypothesized that LPS would cause macroscopic gastric injury at later time points through an iNOS-dependent pathway... - Time-dependent aggravation or attenuation of lipopolysaccharide-induced gastric injury by nitric oxide synthase inhibitionJames W Suliburk
Department of Surgery, University of Texas Medical School at Houston, Houston, Texas 77026, USA
J Surg Res 129:265-71. 2005..In this setting, iNOS has a pathologic role while the constitutive NOS isoforms play gastroprotective roles... - Effects of lipopolysaccharide on gastric stasis: role of cyclooxygenaseSonlee D West
Department of Surgery, University of Texas Medical School, Houston, Texas, 77030, USA
Dig Dis Sci 51:754-65. 2006..However, the ability of LPS to cause gastric stasis and increase gastric luminal fluid accumulation involves COX-2... - Handheld medicine--handheld software to ease the day and improve patient careJames Suliburk
Department of General Surgery, University of Texas at Houston, Houston, Texas 77030, USA
Curr Surg 60:176-7; discussion 177. 2003 - Cyclooxygenase-1 suppresses lipopolysaccharide-induced changes in rat gastric inducible nitric oxide synthaseSonlee D West
Department of Surgery at the University of New Mexico Health Sciences Center, Albuquerque, NM, USA
Crit Care Med 36:572-9. 2008..However, the interaction between these enzymes is not clearly understood. The objective was to clarify the interactions between NOS and COX in the rat gastric mucosa in the presence and absence of lipopolysaccharide...