Franck Mauvais Jarvis

Summary

Affiliation: Baylor College of Medicine
Country: USA

Publications

  1. ncbi PAX4 gene variations predispose to ketosis-prone diabetes
    Franck Mauvais-Jarvis
    Division of Diabetes, Endocrinology and Metabolism, Department of Medicine, Baylor College of Medicine, Houston, TX 77030, USA
    Hum Mol Genet 13:3151-9. 2004
  2. pmc Estrogens protect pancreatic beta-cells from apoptosis and prevent insulin-deficient diabetes mellitus in mice
    Cedric Le May
    Division of Diabetes, Endocrinology and Metabolism, Department of, Baylor College of Medicine, Houston, TX 77030, USA
    Proc Natl Acad Sci U S A 103:9232-7. 2006
  3. ncbi Antidiabetic actions of estrogen: insight from human and genetic mouse models
    Jean Francois Louet
    Department of Medicine, Division of Diabetes, Endocrinology and Metabolism, Baylor College of Medicine, One Baylor Plaza, 520B, Houston, TX 77030, USA
    Curr Atheroscler Rep 6:180-5. 2004
  4. ncbi Ketosis-prone type 2 diabetes in patients of sub-Saharan African origin: clinical pathophysiology and natural history of beta-cell dysfunction and insulin resistance
    Franck Mauvais-Jarvis
    Department of Endocrinology and Diabetes, Saint Louis Hospital and University of Paris VII School of Medicine, Paris, France
    Diabetes 53:645-53. 2004
  5. ncbi Effect of a diabetic environment in utero on predisposition to type 2 diabetes
    Eugene Sobngwi
    Department of Endocrinology and Diabetes, Saint Louis Hospital, Assistance Publique, Hopitaux de Paris, University of Paris VII Medical School, Paris, France
    Lancet 361:1861-5. 2003
  6. ncbi High prevalence of glucose-6-phosphate dehydrogenase deficiency without gene mutation suggests a novel genetic mechanism predisposing to ketosis-prone diabetes
    Eugene Sobngwi
    Department of Endocrinology and Diabetes, St Louis Hospital, University of Paris VII School of Medicine, France
    J Clin Endocrinol Metab 90:4446-51. 2005
  7. pmc Lack of support for the association between GAD2 polymorphisms and severe human obesity
    Michael M Swarbrick
    Diabetes Center, University of California, San Francisco, California, USA
    PLoS Biol 3:e315. 2005
  8. pmc Reduced expression of the murine p85alpha subunit of phosphoinositide 3-kinase improves insulin signaling and ameliorates diabetes
    Franck Mauvais-Jarvis
    Research Division, Joslin Diabetes Center and Department of Medicine, Harvard Medical School, Boston, Massachusetts 02115, USA
    J Clin Invest 109:141-9. 2002
  9. ncbi Cellular and molecular mechanisms of adipose tissue plasticity in muscle insulin receptor knockout mice
    Bertrand Cariou
    Department of Endocrinology, Institut National de la Sante et de la Recherche Medicale, Unité 567 Centre National de la Recherche Scientifique, Paris, France
    Endocrinology 145:1926-32. 2004
  10. pmc p50alpha/p55alpha phosphoinositide 3-kinase knockout mice exhibit enhanced insulin sensitivity
    Dong Chen
    Research Division, Joslin Diabetes Center and Department of Medicine, Harvard Medical School, Boston, Massachusetts 02215, USA
    Mol Cell Biol 24:320-9. 2004

Collaborators

  • Michael S German
  • J F Gautier
  • M Molokhia
  • C Kahn
  • Pierre Jean Guillausseau
  • LAURIE GOODYEAR
  • J Girard
  • Richard Pratley
  • Guillaume Charpentier
  • Kei Sakamoto
  • Samy Hadjadj
  • LEN ALEXANDER PENNACCHIO
  • D Accili
  • C Postic
  • R McPherson
  • Gilberto Velho
  • Rohit Kulkarni
  • Wen Chi Hsueh
  • Franck Mauvais-Jarvis
  • Eugene Sobngwi
  • Patrick Vexiau
  • Christian Vaisse
  • Cedric Le May
  • Jean Pierre Riveline
  • Jean Philippe Kevorkian
  • Raphael Porcher
  • Na Kyung Lee
  • Michael M Swarbrick
  • Sumei Zhang
  • Suzanne M Leal
  • Bertrand Cariou
  • Jean Francois Louet
  • Dong Chen
  • Philippe Boudou
  • Kohjiro Ueki
  • Eiichi Hinoi
  • Zhiyou Zhang
  • Hideaki Sowa
  • Jong Deok Ahn
  • Jason K Kim
  • Dae Young Jung
  • Romain Dacquin
  • Cyrille Confavreux
  • Patrick J Mee
  • Mathieu Ferron
  • Gerard Karsenty
  • Marc D McKee
  • Patricia Ducy
  • Ming Jer Tsai
  • Min Hu
  • Evan R Simpson
  • Khoi Chu
  • Christina S Ortega
  • Kenneth S Korach
  • Denise L Lind
  • Pui Yan Kwok
  • Clive R Pullinger
  • Anke Hinney
  • Raphael Merriman
  • Anna Ustaszewska
  • Frank Geller
  • Mary Malloy
  • Johannes Hebebrand
  • Björn Waldenmaier
  • Martha M Cavazos
  • Jean Marie Villette
  • Andre Scherag
  • Robert Dent
  • Winfried Rief
  • John P Kane
  • Arun S Rajan
  • Alison Jozsi
  • Simon J Fisher
  • Stuart B Smith
  • Matthias Bluher
  • Remy Burcelin
  • Cedric LeMay
  • Anne Françoise Burnol
  • Fabien Calvo
  • Hervé Leblanc
  • P Antonio Tataranni
  • Alain Duvallet
  • Jean Pierre Garnier
  • Lewis C Cantley
  • David A Fruman
  • Michael F Hirshman
  • Matteo Iannacone

Detail Information

Publications13

  1. ncbi PAX4 gene variations predispose to ketosis-prone diabetes
    Franck Mauvais-Jarvis
    Division of Diabetes, Endocrinology and Metabolism, Department of Medicine, Baylor College of Medicine, Houston, TX 77030, USA
    Hum Mol Genet 13:3151-9. 2004
    ....
  2. pmc Estrogens protect pancreatic beta-cells from apoptosis and prevent insulin-deficient diabetes mellitus in mice
    Cedric Le May
    Division of Diabetes, Endocrinology and Metabolism, Department of, Baylor College of Medicine, Houston, TX 77030, USA
    Proc Natl Acad Sci U S A 103:9232-7. 2006
    ..This study demonstrates that estradiol, acting, at least in part, through ERalpha, protects beta-cells from oxidative injury and prevents diabetes in mice of both genders...
  3. ncbi Antidiabetic actions of estrogen: insight from human and genetic mouse models
    Jean Francois Louet
    Department of Medicine, Division of Diabetes, Endocrinology and Metabolism, Baylor College of Medicine, One Baylor Plaza, 520B, Houston, TX 77030, USA
    Curr Atheroscler Rep 6:180-5. 2004
    ....
  4. ncbi Ketosis-prone type 2 diabetes in patients of sub-Saharan African origin: clinical pathophysiology and natural history of beta-cell dysfunction and insulin resistance
    Franck Mauvais-Jarvis
    Department of Endocrinology and Diabetes, Saint Louis Hospital and University of Paris VII School of Medicine, Paris, France
    Diabetes 53:645-53. 2004
    ....
  5. ncbi Effect of a diabetic environment in utero on predisposition to type 2 diabetes
    Eugene Sobngwi
    Department of Endocrinology and Diabetes, Saint Louis Hospital, Assistance Publique, Hopitaux de Paris, University of Paris VII Medical School, Paris, France
    Lancet 361:1861-5. 2003
    ..Type 2 diabetes is affected by genetics and environmental factors. We aimed to assess the effect of an in-utero diabetic environment independently of the genetic background for type 2 diabetes...
  6. ncbi High prevalence of glucose-6-phosphate dehydrogenase deficiency without gene mutation suggests a novel genetic mechanism predisposing to ketosis-prone diabetes
    Eugene Sobngwi
    Department of Endocrinology and Diabetes, St Louis Hospital, University of Paris VII School of Medicine, France
    J Clin Endocrinol Metab 90:4446-51. 2005
    ..Ketosis-prone diabetes (KPD) is mostly observed in males of West African descent and is characterized by phasic or permanent insulin dependence without apparent autoimmune process...
  7. pmc Lack of support for the association between GAD2 polymorphisms and severe human obesity
    Michael M Swarbrick
    Diabetes Center, University of California, San Francisco, California, USA
    PLoS Biol 3:e315. 2005
    ....
  8. pmc Reduced expression of the murine p85alpha subunit of phosphoinositide 3-kinase improves insulin signaling and ameliorates diabetes
    Franck Mauvais-Jarvis
    Research Division, Joslin Diabetes Center and Department of Medicine, Harvard Medical School, Boston, Massachusetts 02115, USA
    J Clin Invest 109:141-9. 2002
    ..Furthermore, Pik3r1 heterozygosity protects mice with genetic insulin resistance from developing diabetes. These data suggest that regulation of p85alpha levels may provide a novel therapeutic target for the treatment of type 2 diabetes...
  9. ncbi Cellular and molecular mechanisms of adipose tissue plasticity in muscle insulin receptor knockout mice
    Bertrand Cariou
    Department of Endocrinology, Institut National de la Sante et de la Recherche Medicale, Unité 567 Centre National de la Recherche Scientifique, Paris, France
    Endocrinology 145:1926-32. 2004
    ..The MIRKO mouse confirms the importance of WAT plasticity in the maintenance of whole body insulin sensitivity and represents an interesting model to search for new secreted molecules that positively alter adipose tissue biology...
  10. pmc p50alpha/p55alpha phosphoinositide 3-kinase knockout mice exhibit enhanced insulin sensitivity
    Dong Chen
    Research Division, Joslin Diabetes Center and Department of Medicine, Harvard Medical School, Boston, Massachusetts 02215, USA
    Mol Cell Biol 24:320-9. 2004
    ..Taken together, these data indicate that p50alpha and p55alpha play an important role in insulin signaling and action, especially in lipid and glucose metabolism...
  11. ncbi Glucose response to intense aerobic exercise in type 1 diabetes: maintenance of near euglycemia despite a drastic decrease in insulin dose
    Franck Mauvais-Jarvis
    Diabetes Care 26:1316-7. 2003
  12. ncbi Knockout models are useful tools to dissect the pathophysiology and genetics of insulin resistance
    Franck Mauvais-Jarvis
    Department of Endocrinology and Diabetes, Saint Louis Hospital and University of Paris VII Medical School, France
    Clin Endocrinol (Oxf) 57:1-9. 2002
    ..The development of type 2 diabetes is linked to insulin resistance coupled with a failure of pancreatic beta-cells to compensate by adequate insulin secretion...
  13. pmc Endocrine regulation of energy metabolism by the skeleton
    Na Kyung Lee
    Department of Genetics and Development, College of Physicians and Surgeons, Columbia University, New York, NY 10032, USA
    Cell 130:456-69. 2007
    ..By revealing that the skeleton exerts an endocrine regulation of sugar homeostasis this study expands the biological importance of this organ and our understanding of energy metabolism...