Buckminster Farrow

Summary

Affiliation: Baylor College of Medicine
Country: USA

Publications

  1. ncbi Characterization of tumor-derived pancreatic stellate cells
    Buckminster Farrow
    Michael E DeBakey Department of Surgery, Houston, Texas 77030, USA
    J Surg Res 157:96-102. 2009
  2. doi Tumor-derived pancreatic stellate cells promote pancreatic cancer cell invasion through release of thrombospondin-2
    Buckminster Farrow
    Michael E DeBakey Department of Surgery, Baylor College of Medicine, TX 77030, USA
    J Surg Res 156:155-60. 2009
  3. doi More than 150 consecutive open umbilical hernia repairs in a major Veterans Administration Medical Center
    Buckminster Farrow
    Department of Surgery, Michael E DeBakey VA Medical Center, Baylor College of Medicine Houston, Houston, TX, USA
    Am J Surg 196:647-51. 2008
  4. doi The role of the tumor microenvironment in the progression of pancreatic cancer
    Buckminster Farrow
    Department of Surgery, Baylor College of Medicine, Michael E DeBakey Veterans Affairs Medical Center, Houston, Texas 77030, USA
    J Surg Res 149:319-28. 2008
  5. ncbi Selective activation of PPARgamma inhibits pancreatic cancer invasion and decreases expression of tissue plasminogen activator
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, Galveston, TX 77555, USA
    Surgery 134:206-12. 2003
  6. ncbi Butyrate inhibits pancreatic cancer invasion
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, Galveston, Texas 77555, USA
    J Gastrointest Surg 7:864-70. 2003
  7. ncbi Analysis of differential gene expression patterns in colon cancer and cancer stroma using microdissected tissues
    Yuko Sugiyama
    Department of Surgery, University of Texas Medical Branch, Galveston, TX 77555, USA
    Gastroenterology 128:480-6. 2005
  8. ncbi Inhibition of pancreatic cancer cell growth and induction of apoptosis with novel therapies directed against protein kinase A
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, Galveston, TX, USA
    Surgery 134:197-205. 2003
  9. ncbi Rectal cancer patients benefit from implementation of a dedicated colorectal cancer center in a Veterans Affairs Medical Center
    Jonathan A Wilks
    Department of Surgery, Michael E DeBakey Veterans Affairs Medical Center, Houston, TX, USA
    Am J Surg 198:100-4. 2009
  10. ncbi Outcomes of Veterans Affairs patients older than age 80 after surgical procedures for colon malignancies
    Liz Lee
    Department of Surgery, Michael E DeBakey VA Medical Center, 2002 Holcombe Blvd, OCL 5th Floor, Room 112, Houston, TX 77030, USA
    Am J Surg 194:646-51. 2007

Collaborators

Detail Information

Publications16

  1. ncbi Characterization of tumor-derived pancreatic stellate cells
    Buckminster Farrow
    Michael E DeBakey Department of Surgery, Houston, Texas 77030, USA
    J Surg Res 157:96-102. 2009
    ..We sought to isolate and characterize tumor-derived pancreatic stellate (TDPS) cells to further understand how these stromal cells influence pancreatic cancer behavior...
  2. doi Tumor-derived pancreatic stellate cells promote pancreatic cancer cell invasion through release of thrombospondin-2
    Buckminster Farrow
    Michael E DeBakey Department of Surgery, Baylor College of Medicine, TX 77030, USA
    J Surg Res 156:155-60. 2009
    ..We sought to determine whether TDPS cells promote the local invasion of pancreatic cancer cells and mechanisms involved...
  3. doi More than 150 consecutive open umbilical hernia repairs in a major Veterans Administration Medical Center
    Buckminster Farrow
    Department of Surgery, Michael E DeBakey VA Medical Center, Baylor College of Medicine Houston, Houston, TX, USA
    Am J Surg 196:647-51. 2008
    ..The purpose of this study was to determine the rate of surgical site infection for open elective umbilical hernia repairs and to identify the factors related to an increased risk of infection and/or recurrence...
  4. doi The role of the tumor microenvironment in the progression of pancreatic cancer
    Buckminster Farrow
    Department of Surgery, Baylor College of Medicine, Michael E DeBakey Veterans Affairs Medical Center, Houston, Texas 77030, USA
    J Surg Res 149:319-28. 2008
    ..Finally we suggest ideas for new avenues of research into the pancreatic tumor microenvironment which may permit the development of novel, more effective treatments for pancreatic cancer...
  5. ncbi Selective activation of PPARgamma inhibits pancreatic cancer invasion and decreases expression of tissue plasminogen activator
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, Galveston, TX 77555, USA
    Surgery 134:206-12. 2003
    ..The purpose of this study was to define the inhibitory effect of PPARgamma ligands on pancreatic cancer invasion and the expression of invasion-related genes...
  6. ncbi Butyrate inhibits pancreatic cancer invasion
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, Galveston, Texas 77555, USA
    J Gastrointest Surg 7:864-70. 2003
    ..NaBT may represent a novel strategy to inhibit pancreatic cancer invasion and improve the prognosis of this deadly disease...
  7. ncbi Analysis of differential gene expression patterns in colon cancer and cancer stroma using microdissected tissues
    Yuko Sugiyama
    Department of Surgery, University of Texas Medical Branch, Galveston, TX 77555, USA
    Gastroenterology 128:480-6. 2005
    ..Here, we analyzed patterns of gene expression in colon cancer cells, surrounding stroma, and normal mucosa and normal stroma using laser capture microdissection...
  8. ncbi Inhibition of pancreatic cancer cell growth and induction of apoptosis with novel therapies directed against protein kinase A
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, Galveston, TX, USA
    Surgery 134:197-205. 2003
    ..In pancreatic cancer cells, we sought to determine (1) whether inhibition of PKA can inhibit growth or induce apoptosis, and (2) whether growth can be inhibited by silencing of RIalpha expression...
  9. ncbi Rectal cancer patients benefit from implementation of a dedicated colorectal cancer center in a Veterans Affairs Medical Center
    Jonathan A Wilks
    Department of Surgery, Michael E DeBakey Veterans Affairs Medical Center, Houston, TX, USA
    Am J Surg 198:100-4. 2009
    ..A dedicated colorectal cancer (CRC) center was created in a Veterans Affairs Medical Center with the intent of improving quality of patient care and multidisciplinary cooperation...
  10. ncbi Outcomes of Veterans Affairs patients older than age 80 after surgical procedures for colon malignancies
    Liz Lee
    Department of Surgery, Michael E DeBakey VA Medical Center, 2002 Holcombe Blvd, OCL 5th Floor, Room 112, Houston, TX 77030, USA
    Am J Surg 194:646-51. 2007
    ..There is a lack of information concerning surgical outcomes in this patient population regarding colon cancer. Herein we report a 10-year experience of surgical outcomes...
  11. doi Translation of recent advances and discoveries in molecular biology and immunology in the diagnosis and treatment of pancreatic cancer
    Daniel Albo
    Michael E DeBakey Department of Surgery, Baylor College of Medicine, Michael E DeBakey Veterans Affairs Medical Center, 2002 Holcombe Boulevard, OCL 112A, Houston, TX 77030, USA
    Surg Oncol Clin N Am 17:357-76, ix. 2008
    ..Although a clear "magic bullet" has yet to emerge, this type of targeted approach offers hope in the management of this dreadful disease. This article offers an update on these promising diagnostic and treatment modalities...
  12. ncbi Activation of conventional PKC isoforms increases expression of the pro-apoptotic protein Bad and TRAIL receptors
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, Galveston, TX 77555, USA
    Int J Gastrointest Cancer 32:63-72. 2002
    ..The purpose of our study was to assess the effect of PKC stimulation on apoptotic gene expression in pancreatic cancer cells...
  13. pmc Inflammatory mechanisms contributing to pancreatic cancer development
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, Galveston, TX 77555 0536, USA
    Ann Surg 239:763-9; discussion 769-71. 2004
    ..Pancreatic cancer is the most deadly of all gastrointestinal (GI) malignancies, yet relatively little is known regarding mechanisms of tumor development including the role of inflammation...
  14. ncbi Stem cells: what is their future in surgery?
    Buckminster Farrow
    Department of Surgery, University of Texas Medical Branch, Galveston, Texas, USA
    Adv Surg 37:51-69. 2003
  15. ncbi Activation of PPARgamma increases PTEN expression in pancreatic cancer cells
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, 301 University Boulevard, Galveston, TX 77555, USA
    Biochem Biophys Res Commun 301:50-3. 2003
    ..Taken together, our results suggest that activation of PPARgamma may represent a novel approach for the treatment of pancreatic cancer by increasing PTEN levels and inhibiting PI3K activity...
  16. ncbi Inflammation and the development of pancreatic cancer
    Buckminster Farrow
    Department of Surgery, The University of Texas Medical Branch, 301 University Boulevard, Galveston 77555, USA
    Surg Oncol 10:153-69. 2002
    ....