Research Topics
| Elizabeth DialSummaryAffiliation: Baylor College of Medicine Country: USA Publications
Research Grants
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Detail Information
Publications
Importance of biliary excretion of indomethacin in gastrointestinal and hepatic injuryElizabeth J Dial
Department of Integrative Biology and Pharmacology, The University of Texas Health Science Center at Houston, Medical School, Houston, Texas 77030, USA
J Gastroenterol Hepatol 23:e384-9. 2008..We investigated the role of bile acids and PC in the mechanism of indomethacin-induced epithelial injury...
Oral phosphatidylcholine preserves the gastrointestinal mucosal barrier during LPS-induced inflammationElizabeth J Dial
Department of Integrative Biology and Pharmacology, The University of Texas Health Science Center at Houston Medical School, Houston, TX 77030, USA
Shock 30:729-33. 2008..Under the conditions studied, oral PC does not block systemic effects of LPS. However, enteral formulations containing PC may be useful adjuncts in the prevention of GI injury from endotoxemia...
A report on associations among gastric pH, bleeding, duodenogastric reflux, and outcomes after traumaElizabeth Dial
Department of Integrative Biology and Pharmacology, The University of Texas Health Science Center at Houston Medical School, Houston, Texas, USA
J Trauma 64:105-10. 2008..In a prospective study, the gastric fluid of major torso trauma patients was examined for evidence of duodenogastric reflux and potential gastric injury, and was compared with patient outcomes regarding MOF...
Role of phosphatidylcholine saturation in preventing bile salt toxicity to gastrointestinal epithelia and membranesElizabeth J Dial
Department of Integrative Biology and Pharmacology, Medical School, The University of Texas Health Science Center at Houston, Houston, Texas 77030, USA
J Gastroenterol Hepatol 23:430-6. 2008....
Phosphatidylcholine-associated nonsteroidal anti-inflammatory drugs (NSAIDs) inhibit DNA synthesis and the growth of colon cancer cells in vitroElizabeth J Dial
Department of Integrative Biology and Pharmacology, The University of Texas Medical School, Houston, 77030, USA
Cancer Chemother Pharmacol 57:295-300. 2006..Due to its greater efficacy in this model system, IBU-PC should be further evaluated as a chemopreventive agent that is safer for the GI tract than unmodified NSAID...
Recombinant human lactoferrin prevents NSAID-induced intestinal bleeding in rodentsElizabeth J Dial
Department of Integrative Biology and Pharmacology, Medical School, University of Texas Health Science Center at Houston, 6431 Fannin, Houston, TX 77030 1503, USA
J Pharm Pharmacol 57:93-9. 2005..We conclude that orally administered RHLF is effective at preventing NSAID-induced intestinal injury in rodents and should be investigated for this potential therapeutic use in man...
Effect of lactoferrin on Helicobacter felis induced gastritisElizabeth J Dial
Department of Integrative Biology and Pharmacology, University of Texas Health Science Center, Houston Medical School, 77225, USA
Biochem Cell Biol 80:113-7. 2002....
Effect of indomethacin on bile acid-phospholipid interactions: implication for small intestinal injury induced by nonsteroidal anti-inflammatory drugsYong Zhou
Department of Pediatrics Gastroenterology, Baylor College of Medicine, Houston, TX, USA
Am J Physiol Gastrointest Liver Physiol 298:G722-31. 2010..This mechanism potentially contributes to the NSAID-induced injury in the small bowel...
The effects of aspirin on gastric mucosal integrity, surface hydrophobicity, and prostaglandin metabolism in cyclooxygenase knockout miceRebecca L Darling
Department of Integrative Biology and Pharmacology, The University of Texas Medical School, Houston, Texas 77030, USA
Gastroenterology 127:94-104. 2004..Aspirin seems to paradoxically increase the gastric mucosal prostaglandin E(2) concentration in cyclooxygenase-1 knockout mice, possibly by the induction of cyclooxygenase-2...
NSAID injury to the gastrointestinal tract: evidence that NSAIDs interact with phospholipids to weaken the hydrophobic surface barrier and induce the formation of unstable pores in membranesLenard M Lichtenberger
The Department of Integrative Biology and Pharmacology, The University of Texas Medical School, Houston, TX 77030, USA
J Pharm Pharmacol 58:1421-8. 2006..This understanding of the interaction of NSAIDs with membrane phospholipids may prove valuable in the design of novel NSAID formulations with reduced gastrointestinal side-effects...
Pathophysiology of LPS-induced gastrointestinal injury in the rat: role of secretory phospholipase A2Mayssa Zayat
Department of Pediatric Gastroenterology, Baylor College of Medicine, Houston, TX, USA
Shock 30:206-11. 2008....
A direct role for secretory phospholipase A2 and lysophosphatidylcholine in the mediation of LPS-induced gastric injuryElizabeth J Dial
Department of Integrative Biology and Pharmacology, University of Texas Medical School at Houston, Texas, USA
Shock 33:634-8. 2010..An LPS-induced increase in sPLA2 activity in the gastric lumen and its product, lyso-PC, are capable of directly disrupting the gastric hydrophobic layer and may contribute to gastric barrier disruption and subsequent inflammation...
Lipopolysaccharide-induced gastrointestinal injury in rats: role of surface hydrophobicity and bile saltsElizabeth J Dial
Department of Integrative Biology and Pharmacology, The University of Texas Health Science Center, Houston Medical School, 77225, USA
Shock 17:77-80. 2002..We conclude that LPS disrupts gastrointestinal barrier integrity, in part by mechanisms involving bile constituents and an attenuation in the mucosa's hydrophobic characteristics...
Bile acids improve the antimicrobial effect of rifaximinCharles Darkoh
University of Texas Graduate School of Biomedical Sciences, Houston, TX, USA
Antimicrob Agents Chemother 54:3618-24. 2010..The water insolubility of rifaximin is the likely explanation for the drug's minimal effects on colonic flora and fecal pathogens, despite in vitro susceptibility...
Research Grants
- THERAPY FOR GASTROINTESTINAL DISORDERSElizabeth Dial; Fiscal Year: 2002..Development of this therapeutic potential for rhLF will fulfill a crucial need for a way to safely administer NSAIDs. PROPOSED COMMERCIAL APPLICATION: NOT AVAILABLE ..
