Carlos D Rose

Summary

Affiliation: Alfred I. duPont Hospital for Children
Country: USA

Publications

  1. doi request reprint Prolonged synovitis in pediatric Lyme arthritis cannot be predicted by clinical or laboratory parameters
    Annemarie C Brescia
    Division of Rheumatology, AI duPont Hospital for Children, Wilmington, DE 19803, USA
    Clin Rheumatol 28:591-3. 2009
  2. ncbi request reprint Pediatric granulomatous arthritis: an international registry
    Carlos D Rose
    duPont Children s Hospital, Wilmington, Delaware 19899, USA
    Arthritis Rheum 54:3337-44. 2006
  3. pmc NOD2-associated pediatric granulomatous arthritis, an expanding phenotype: study of an international registry and a national cohort in Spain
    Carlos D Rose
    Thomas Jefferson University, Philadelphia, Pennsylvania, USA
    Arthritis Rheum 60:1797-803. 2009
  4. ncbi request reprint Blau syndrome mutation of CARD15/NOD2 in sporadic early onset granulomatous arthritis
    Carlos D Rose
    Thomas Jefferson University, duPont Children s Hospital, Wilmington, Delaware, USA
    J Rheumatol 32:373-5. 2005
  5. ncbi request reprint Caspase recruitment domain 15 mutations and rheumatic diseases
    Carlos D Rose
    Pediatric Rheumatology, duPont Children s Hospital, Philadelphia, Pennsylvania, USA
    Curr Opin Rheumatol 17:579-85. 2005
  6. ncbi request reprint Epidemiology of juvenile rheumatoid arthritis in the Americas: an update
    Carlos D Rose
    Division of Rheumatology, duPont Hospital for Children, Wilmington, DE 19899, USA
    J Clin Rheumatol 12:129-30. 2006
  7. doi request reprint Blau arteritis resembling Takayasu disease with a novel NOD2 mutation
    Raju P Khubchandani
    Division of Rheumatology, duPont Children s Hospital, 1600 Rockland Road, Wilmington, DE 19899, USA
    J Rheumatol 39:1888-92. 2012
  8. doi request reprint Blau syndrome revisited
    Carlos D Rose
    Thomas Jefferson University and duPont Children s Hospital, Wilmington, Delaware, USA
    Curr Opin Rheumatol 23:411-8. 2011
  9. ncbi request reprint Blau syndrome and related genetic disorders causing childhood arthritis
    Mara L Becker
    Division of Pediatric Rheumatology, duPont Children s Hospital, Wilmington, DE 19899, USA
    Curr Rheumatol Rep 7:427-33. 2005
  10. pmc Long-term effects of immunization with recombinant lipoprotein outer surface protein a on serologic test for lyme disease
    Paul T Fawcett
    Immunology Laboratory, Department of Research, A I duPont Hospital for Children, Wilmington, Delaware 19803, USA
    Clin Diagn Lab Immunol 11:808-10. 2004

Collaborators

Detail Information

Publications14

  1. doi request reprint Prolonged synovitis in pediatric Lyme arthritis cannot be predicted by clinical or laboratory parameters
    Annemarie C Brescia
    Division of Rheumatology, AI duPont Hospital for Children, Wilmington, DE 19803, USA
    Clin Rheumatol 28:591-3. 2009
    ..Linear and logistic regression showed no predictors of disease duration. One third of pediatric LA patients require >6 months to resolve synovitis. Duration is not associated with delay in treatment, age, or seroreactivity...
  2. ncbi request reprint Pediatric granulomatous arthritis: an international registry
    Carlos D Rose
    duPont Children s Hospital, Wilmington, Delaware 19899, USA
    Arthritis Rheum 54:3337-44. 2006
    ..We chose the term "pediatric granulomatous arthritis" to refer to both. An international registry was established in the spring of 2005 to define the phenotype spectrum and establish the mutation frequency and variants...
  3. pmc NOD2-associated pediatric granulomatous arthritis, an expanding phenotype: study of an international registry and a national cohort in Spain
    Carlos D Rose
    Thomas Jefferson University, Philadelphia, Pennsylvania, USA
    Arthritis Rheum 60:1797-803. 2009
    ..To study the phenotype characteristics of the largest to date cohort of patients with pediatric granulomatous arthritis (PGA) and documented mutations in the NOD2 gene...
  4. ncbi request reprint Blau syndrome mutation of CARD15/NOD2 in sporadic early onset granulomatous arthritis
    Carlos D Rose
    Thomas Jefferson University, duPont Children s Hospital, Wilmington, Delaware, USA
    J Rheumatol 32:373-5. 2005
    ..This would explain the profound clinical identity and the lack of disease history in the parents...
  5. ncbi request reprint Caspase recruitment domain 15 mutations and rheumatic diseases
    Carlos D Rose
    Pediatric Rheumatology, duPont Children s Hospital, Philadelphia, Pennsylvania, USA
    Curr Opin Rheumatol 17:579-85. 2005
    ..The purpose of this article is to review the foundational work and current developments on a group of rheumatic disorders associated with mutations in the caspase recruitment domain 15/nucleotide oligomerization domain 2 gene...
  6. ncbi request reprint Epidemiology of juvenile rheumatoid arthritis in the Americas: an update
    Carlos D Rose
    Division of Rheumatology, duPont Hospital for Children, Wilmington, DE 19899, USA
    J Clin Rheumatol 12:129-30. 2006
  7. doi request reprint Blau arteritis resembling Takayasu disease with a novel NOD2 mutation
    Raju P Khubchandani
    Division of Rheumatology, duPont Children s Hospital, 1600 Rockland Road, Wilmington, DE 19899, USA
    J Rheumatol 39:1888-92. 2012
    ..To put forward a new concept--Blau arteritis, a form of large-vessel vasculitis phenotypically related to Takayasu disease but genetically and clinically part of an expanded phenotype of Blau syndrome...
  8. doi request reprint Blau syndrome revisited
    Carlos D Rose
    Thomas Jefferson University and duPont Children s Hospital, Wilmington, Delaware, USA
    Curr Opin Rheumatol 23:411-8. 2011
    ..Not only there has been significant progress in disease characterization but also the biological pathways associated with NOD2 and related proteins of the innate immunity are better understood...
  9. ncbi request reprint Blau syndrome and related genetic disorders causing childhood arthritis
    Mara L Becker
    Division of Pediatric Rheumatology, duPont Children s Hospital, Wilmington, DE 19899, USA
    Curr Rheumatol Rep 7:427-33. 2005
    ..This paper will review the clinical characteristics of these three disorders and their association with mutations in the CARD 15 gene...
  10. pmc Long-term effects of immunization with recombinant lipoprotein outer surface protein a on serologic test for lyme disease
    Paul T Fawcett
    Immunology Laboratory, Department of Research, A I duPont Hospital for Children, Wilmington, Delaware 19803, USA
    Clin Diagn Lab Immunol 11:808-10. 2004
    ..We tested sera from 152 vaccine recipients by using in-house and commercial Western blot assays and found that vaccination caused interference in up to 25% of recipients and can persist for over 6 years...
  11. pmc In vitro assessment of antiborrelial activity of OspA vaccine sera
    Paul T Fawcett
    Immunology Laboratory, Department of Research, A I duPont Hospital for Children, Wilmington, Delaware 19803, USA
    Clin Diagn Lab Immunol 9:919-20. 2002
    ..We report on an in vitro microculture assay, which can be used to provide an indicator of the need for booster doses of vaccine...
  12. ncbi request reprint Myocarditis and sacroiliitis: 2 previously unrecognized manifestations of tumor necrosis factor receptor associated periodic syndrome
    Stephanie Trost
    Department of Rheumatology, Alfred I duPont Hospital for Children, Wilmington, Delaware 19899, USA
    J Rheumatol 32:175-7. 2005
    ..We describe a 9-year-old African American boy with the P46L mutation of the TNF receptor who presented with 2 previously unrecognized manifestations: sacroiliitis and myocardiopathy, both showing a reversible course...
  13. ncbi request reprint Granulomatous nephritis associated with R334Q mutation in NOD2
    Silvia Mónica Meiorin
    J Rheumatol 34:1945-7. 2007
  14. pmc Infantile onset panniculitis with uveitis and systemic granulomatosis: a new clinicopathologic entity
    Carine H Wouters
    University Hospital Gasthuisberg, Leuven, Belgium
    J Pediatr 151:707-9. 2007
    ..Neither CARD15 nor CIAS1 mutations were found. Despite immunosuppressive therapy, disease course was progressive. Response to anti-tumor necrosis factor monoclonal antibody in 3 patients is of note...