Research Topics
Genomes and Genes | Bridget Shafit-ZagardoSummaryAffiliation: Albert Einstein College of Medicine Country: USA Publications
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Publications
Loss of the receptor tyrosine kinase Axl leads to enhanced inflammation in the CNS and delayed removal of myelin debris during experimental autoimmune encephalomyelitisJason G Weinger
Department of Pathology, Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, New York 10461, USA
J Neuroinflammation 8:49. 2011..To test for this, we studied the susceptibility of Axl-/- mice to experimental autoimmune encephalomyelitis (EAE), an animal model for multiple sclerosis...
GAS6 enhances repair following cuprizone-induced demyelinationVladislav Tsiperson
Department of Pathology, Albert Einstein College of Medicine, Bronx, New York, United States of America
PLoS ONE 5:e15748. 2010..Relative to PBS-treated mice, rhGas6-treated mice had fewer activated microglia in the corpus callosum by Iba1 immunostaining. The data show that rhGas6 treatment resulted in more efficient repair following cuprizone-induced injury...
Fyn phosphorylates human MAP-2c on tyrosine 67S Pilar Zamora-Leon
Department of Pathology, Albert Einstein College of Medicine, Bronx, New York 10461, USA
J Biol Chem 280:1962-70. 2005..Thus, MAP-2c can directly recruit multiple signaling proteins important for central nervous system development...
In brain, Axl recruits Grb2 and the p85 regulatory subunit of PI3 kinase; in vitro mutagenesis defines the requisite binding sites for downstream Akt activationJason G Weinger
Department of Pathology, Albert Einstein College of Medicine, Bronx, New York 10461, USA
J Neurochem 106:134-46. 2008....
Up-regulation of soluble Axl and Mer receptor tyrosine kinases negatively correlates with Gas6 in established multiple sclerosis lesionsJason G Weinger
Department of Pathology, Albert Einstein College of Medicine, Bronx, New York 10461, USA
Am J Pathol 175:283-93. 2009..These data suggest that in multiple sclerosis lesions, dysregulation of protective Gas6 receptor signaling may prolong lesion activity...
The growth arrest-specific gene product Gas6 promotes the survival of human oligodendrocytes via a phosphatidylinositol 3-kinase-dependent pathwaySai Latha Shankar
Department of Pathology, Albert Einstein College of Medicine, Bronx, New York 10461, USA
J Neurosci 23:4208-18. 2003..Thus Gas6 sustains human fetal oligodendrocyte viability by receptor activation and downstream signaling via the PI3-kinase/Akt pathway...
Multiple sclerosis: re-expression of a developmental pathway that restricts oligodendrocyte maturationGareth R John
Department of Pathology Neuropathology, Albert Einstein College of Medicine, Bronx, New York, USA
Nat Med 8:1115-21. 2002..These data are the first to implicate the Notch pathway in the limited remyelination in MS. Thus, Notch may represent a potential target for therapeutic intervention in this disease...
Interferon regulatory factor 3 inhibits astrocyte inflammatory gene expression through suppression of the proinflammatory miR-155 and miR-155*Leonid Tarassishin
Department of Pathology, Albert Einstein College of Medicine, Bronx, NY, USA
Glia 59:1911-22. 2011..Our results demonstrate a novel proinflammatory role for miR-155/miR-155* in human astrocytes and suggest that IRF3 can suppress neuroinflammation through regulating immunomodulatory miRNA expression. © 2011 Wiley-Liss, Inc...
Disruption of the actin network enhances MAP-2c and Fyn-induced process outgrowthS Pilar Zamora-Leon
Department of Pathology, Albert Einstein College of Medicine, Bronx, New York 10461, USA
Cell Motil Cytoskeleton 62:110-23. 2005..These studies suggest that independent of one another MAP-2c and Fyn are able to induce process outgrowth and in concert can initiate and enhance process outgrowth in an additive manner...
Axl-/- mice have delayed recovery and prolonged axonal damage following cuprizone toxicityHannah J Hoehn
Department of Pathology, Albert Einstein College of Medicine, Bronx, NY 10461, USA
Brain Res 1240:1-11. 2008....
UCN-01 alters phosphorylation of Akt and GSK3beta and induces apoptosis in six independent human neuroblastoma cell linesSai Latha Shankar
Department of Pathology, Albert Einstein College of Medicine, Bronx, New York 10461, USA
J Neurochem 90:702-11. 2004..Thus, UCN-01 was effective at inducing apoptosis in neuroblastoma cell lines...
Up-regulation of MAP2e-expressing oligodendrocytes in the white matter of patients with HIV-1 encephalitisMelissa A Cosenza
Department of Pathology, Albert Einstein College of Medicine, Bronx NY 10461, USA
Neuropathol Appl Neurobiol 28:480-8. 2002..Our results demonstrate that MAP2e can serve as a marker of white matter damage in HIVE and support the notion that oligodendrocyte damage/repair occurs during HIV-1 infection...
Gas6/Axl signaling activates the phosphatidylinositol 3-kinase/Akt1 survival pathway to protect oligodendrocytes from tumor necrosis factor alpha-induced apoptosisSai Latha Shankar
Department of Pathology, Albert Einstein College of Medicine, Bronx, New York 10461, USA
J Neurosci 26:5638-48. 2006..We conclude that gas6 signaling through the Axl receptor and the PI3 kinase/Akt1 survival pathway protects oligodendrocytes from growth factor withdrawal and TNFalpha-mediated cell death...
Inhibition of human neuroblastoma cell growth by CAY10404, a highly selective Cox-2 inhibitorBhupesh Parashar
Department of Radiation Oncology, Montefiore Medical Center, Bronx, NY 10461, USA
J Neurooncol 71:141-8. 2005....
